TWEAK/Fn14 Signaling Involvement in the Pathogenesis of Cutaneous Disease in the MRL/lpr Model of Spontaneous Lupus. - Wikidata - wikimedia - TriplyDB

TWEAK/Fn14 Signaling Involvement in the Pathogenesis of Cutaneous Disease in the MRL/lpr Model of Spontaneous Lupus.

TWEAK/Fn14 Signaling Involvement in the Pathogenesis of Cutaneous Disease in the MRL/lpr Model of Spontaneous Lupus.

http://www.wikidata.org/entity/Q35858608

about

Chemokines and Chemokine Receptors in the Development of Lupus Nephritis TWEAK/Fn14 Activation Participates in Ro52-Mediated Photosensitization in Cutaneous Lupus Erythematosus TWEAK mediates inflammation in experimental atopic dermatitis and psoriasis.Fn14, a Downstream Target of the TGF-β Signaling Pathway, Regulates Fibroblast Activation.Neuropsychiatric systemic lupus erythematosus persists despite attenuation of systemic disease in MRL/lpr mice.Fn14-Fc suppresses germinal center formation and pathogenic B cells in a lupus mouse model via inhibition of the TWEAK/Fn14 Pathway.Role of the TWEAK/Fn14 pathway in autoimmune diseases.Beyond TNF: TNF superfamily cytokines as targets for the treatment of rheumatic diseases.Therapeutic options for cutaneous lupus erythematosus: recent advances and future prospects.Cutaneous lupus erythematosus: updates on pathogenesis and associations with systemic lupus.Roles of tumour necrosis factor-related weak inducer of apoptosis/fibroblast growth factor-inducible 14 pathway in lupus nephritis.The blood brain barrier and neuropsychiatric lupus: new perspectives in light of advances in understanding the neuroimmune interface.Fn14 deficiency protects lupus-prone mice from histological lupus erythematosus-like skin inflammation induced by ultraviolet light.Fn14 is regulated via the RhoA pathway and mediates nuclear factor-kappaB activation by Angiotensin II.TWEAK/Fn14 Activation Participates in Skin Inflammation.TWEAK is not elevated in patients with newly diagnosed inflammatory bowel disease.Mechanisms of neuropsychiatric lupus: The relative roles of the blood-cerebrospinal fluid barrier versus blood-brain barrier.TWEAK/Fn14 activation induces keratinocyte proliferation under psoriatic inflammation.Tnfrsf12a-Mediated Atherosclerosis Signaling and Inflammatory Response as a Common Protection Mechanism of Shuxuening Injection Against Both Myocardial and Cerebral Ischemia-Reperfusion Injuries.Topical TWEAK Accelerates Healing of Experimental Burn Wounds in Mice.Human and Murine Evidence for Mechanisms Driving Autoimmune Photosensitivity

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TWEAK/Fn14 Signaling Involvement in the Pathogenesis of Cutaneous Disease in the MRL/lpr Model of Spontaneous Lupus.

http://www.wikidata.org/entity/Q35858608

description

2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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2015年论文

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2015年论文

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name

TWEAK/Fn14 Signaling Involveme ...... pr Model of Spontaneous Lupus.

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TWEAK/Fn14 Signaling Involveme ...... pr Model of Spontaneous Lupus.

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Journal of Investigative Dermatology

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TWEAK/Fn14 Signaling Involveme ...... lpr Model of Spontaneous Lupus

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Chaim Putterman

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David Schairer

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Jennifer S Michaelson

http://www.w3.org/2001/XMLSchema#string

Jing Wen

http://www.w3.org/2001/XMLSchema#string

Karin Blecher Paz

http://www.w3.org/2001/XMLSchema#string

Lan Wu

http://www.w3.org/2001/XMLSchema#string

Linda C Burkly

http://www.w3.org/2001/XMLSchema#string

Samantha A Chalmers

http://www.w3.org/2001/XMLSchema#string

Yumin Xia

http://www.w3.org/2001/XMLSchema#string

P2860

CD154: an immunoinflammatory mediator in systemic lupus erythematosus and rheumatoid arthritis

Proinflammatory activity of TWEAK on human dermal fibroblasts and synoviocytes: blocking and enhancing effects of anti-TWEAK monoclonal antibodies

Inflammatory cytokines in systemic lupus erythematosus

TWEAK, a member of the TNF superfamily, is a multifunctional cytokine that binds the TweakR/Fn14 receptor

TWEAK, a new secreted ligand in the tumor necrosis factor family that weakly induces apoptosis

Induction of RANTES by TWEAK/Fn14 interaction in human keratinocytes

TNF-like weak inducer of apoptosis (TWEAK) promotes beta cell neogenesis from pancreatic ductal epithelium in adult mice.

TWEAK/Fn14 pathway: an immunological switch for shaping tissue responses.

Prevention of UVB radiation-induced epidermal damage by expression of heat shock protein 70.

Isolation and characterization of cutaneous epithelial stem cells.

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1986-1995

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10.1038/JID.2015.124

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8

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135

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Adam J. Friedman

Jessica Doerner

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2015-03-31T00:00:00Z

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274317903

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25826425

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4504782

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