Late virus replication events in microglia are required for neurovirulent retrovirus-induced spongiform neurodegeneration: evidence from neural progenitor-derived chimeric mouse brains
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Endoplasmic Reticulum Stress Is a Determinant of Retrovirus-Induced Spongiform NeurodegenerationPostinhibitory rebound neurons and networks are disrupted in retrovirus-induced spongiform neurodegeneration.Brain infection by neuroinvasive but avirulent murine oncornaviruses.Differential glycosylation of the Cas-Br-E env protein is associated with retrovirus-induced spongiform neurodegenerationIncreased expression of MIP-1 alpha and MIP-1 beta mRNAs in the brain correlates spatially and temporally with the spongiform neurodegeneration induced by a murine oncornavirus.Protection against murine leukemia virus-induced spongiform myeloencephalopathy in mice overexpressing Bcl-2 but not in mice deficient for interleukin-6, inducible nitric oxide synthetase, ICE, Fas, Fas ligand, or TNF-R1 genesImplications and limitations of cellular reprogramming for psychiatric drug development.Gene expression profiling of microglia infected by a highly neurovirulent murine leukemia virus: implications for neuropathogenesisRetrovirus-induced spongiform neurodegeneration is mediated by unique central nervous system viral targeting and expression of env aloneDisparate regions of envelope protein regulate syncytium formation versus spongiform encephalopathy in neurological disease induced by murine leukemia virus TR.Neurologic disease induced by polytropic murine retroviruses: neurovirulence determined by efficiency of spread to microglial cellsEcotropic Murine Leukemia Virus Infection of Glial Progenitors Interferes with Oligodendrocyte Differentiation: Implications for Neurovirulence.Unique N-linked glycosylation of CasBrE Env influences its stability, processing, and viral infectivity but not its neurotoxicity.Abundant defective viral particles budding from microglia in the course of retroviral spongiform encephalopathy.Microglial activation varies in different models of Creutzfeldt-Jakob disease.Neural stem cells as engraftable packaging lines can mediate gene delivery to microglia: evidence from studying retroviral env-related neurodegeneration.Misfolding of CasBrE SU is reversed by interactions with 4070A Env: implications for gammaretroviral neuropathogenesis.Rebound from Inhibition: Self-Correction against Neurodegeneration?
P2860
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P2860
Late virus replication events in microglia are required for neurovirulent retrovirus-induced spongiform neurodegeneration: evidence from neural progenitor-derived chimeric mouse brains
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年論文
@yue
1996年論文
@zh-hant
1996年論文
@zh-hk
1996年論文
@zh-mo
1996年論文
@zh-tw
1996年论文
@wuu
1996年论文
@zh
1996年论文
@zh-cn
name
Late virus replication events ...... -derived chimeric mouse brains
@ast
Late virus replication events ...... -derived chimeric mouse brains
@en
type
label
Late virus replication events ...... -derived chimeric mouse brains
@ast
Late virus replication events ...... -derived chimeric mouse brains
@en
prefLabel
Late virus replication events ...... -derived chimeric mouse brains
@ast
Late virus replication events ...... -derived chimeric mouse brains
@en
P2093
P2860
P1433
P1476
Late virus replication events ...... -derived chimeric mouse brains
@en
P2093
A H Sharpe
E Y Snyder
J L Portis
L Qualtiere
P2860
P304
P407
P577
1996-12-01T00:00:00Z