IL-17A produced by αβ T cells drives airway hyper-responsiveness in mice and enhances mouse and human airway smooth muscle contraction. - Wikidata - wikimedia - TriplyDB

IL-17A produced by αβ T cells drives airway hyper-responsiveness in mice and enhances mouse and human airway smooth muscle contraction.

IL-17A produced by αβ T cells drives airway hyper-responsiveness in mice and enhances mouse and human airway smooth muscle contraction.

http://www.wikidata.org/entity/Q35874754

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Herbal Medicines for Asthmatic Inflammation: From Basic Researches to Clinical Applications Dendritic cells in lung immunopathology The roles and functional mechanisms of interleukin-17 family cytokines in mucosal immunity Does IL-17 Respond to the Disordered Lung Microbiome and Contribute to the Neutrophilic Phenotype in Asthma?IL-4 and IL-13 signaling in allergic airway disease Th17-mediated inflammation in asthma The cell biology of asthma Innate lymphocyte cells in asthma phenotypes Microbial Ligand Costimulation Drives Neutrophilic Steroid-Refractory Asthma A critical role for dendritic cells in the evolution of IL-1β-mediated murine airway disease Mechanisms underlying helper T-cell plasticity: implications for immune-mediated disease.Breathing Better Through Bugs: Asthma and the Microbiome Effect of IL-17 receptor A blockade with brodalumab in inflammatory diseases Integrin α9β1 in airway smooth muscle suppresses exaggerated airway narrowing.The phosphatase CD148 promotes airway hyperresponsiveness through SRC family kinases.TGF-β activation and function in immunity.Involvement of interleukin-17A-induced hypercontractility of intestinal smooth muscle cells in persistent gut motor dysfunction Cytokine inhibition in the treatment of COPD.New insights into the role of the aryl hydrocarbon receptor in the function of CD11c⁺ cells during respiratory viral infection Intraepithelial neutrophils in pediatric severe asthma are associated with better lung function.Directing traffic: IL-17 and IL-22 coordinate pulmonary immune defense.Airway contractility in the precision-cut lung slice after cryopreservation.Loss of regulator of G protein signaling 5 promotes airway hyperresponsiveness in the absence of allergic inflammation.Decreased percentage of CD4(+)Foxp3(+)TGF-β(+) and increased percentage of CD4(+)IL-17(+) cells in bronchoalveolar lavage of asthmatics The complex relationship between inflammation and lung function in severe asthma The roles of innate lymphoid cells in the development of asthma.Inflammation-induced interstitial migration of effector CD4⁺ T cells is dependent on integrin αV.Induction of IL-17A Precedes Development of Airway Hyperresponsiveness during Diet-Induced Obesity and Correlates with Complement Factor D Effect of antigen sensitization and challenge on oscillatory mechanics of the lung and pulmonary inflammation in obese carboxypeptidase E-deficient mice Human airway musculature on a chip: an in vitro model of allergic asthmatic bronchoconstriction and bronchodilation.Epicutaneous exposure to staphylococcal superantigen enterotoxin B enhances allergic lung inflammation via an IL-17A dependent mechanism.Randomized, double-blind, placebo-controlled study of brodalumab, a human anti-IL-17 receptor monoclonal antibody, in moderate to severe asthma.The transcription factor Etv5 controls TH17 cell development and allergic airway inflammation.IL-17 in severe asthma. Where do we stand?IL-17A impairs host tolerance during airway chronic infection by Pseudomonas aeruginosa Negative regulation of pulmonary Th17 responses by C3a anaphylatoxin during allergic inflammation in mice.Increased frequency of dual-positive TH2/TH17 cells in bronchoalveolar lavage fluid characterizes a population of patients with severe asthma.Experimental human pneumococcal carriage augments IL-17A-dependent T-cell defence of the lung.Trif-dependent induction of Th17 immunity by lung dendritic cells All-trans retinoic acid attenuates airway inflammation by inhibiting Th2 and Th17 response in experimental allergic asthma

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IL-17A produced by αβ T cells drives airway hyper-responsiveness in mice and enhances mouse and human airway smooth muscle contraction.

http://www.wikidata.org/entity/Q35874754

description

2012 nî lūn-bûn

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IL-17A produced by αβ T cells ...... way smooth muscle contraction.

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IL-17A produced by αβ T cells ...... way smooth muscle contraction.

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IL-17A produced by αβ T cells ...... way smooth muscle contraction.

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Andrew C Melton

http://www.w3.org/2001/XMLSchema#string

Chun Chen

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Dean Sheppard

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John T Li

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Kamran Atabai

http://www.w3.org/2001/XMLSchema#string

Katherine E Huang

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Makoto Kudo

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Mary B Engler

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Xiaozhu Huang

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Xin Bernstein

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P2860

The differentiation of human T(H)-17 cells requires transforming growth factor-beta and induction of the nuclear receptor RORgammat

Structural basis of receptor sharing by interleukin 17 cytokines

T cell interleukin-17 induces stromal cells to produce proinflammatory and hematopoietic cytokines

Th17 and regulatory T cells in mediating and restraining inflammation.

Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells

The orphan nuclear receptor RORgammat directs the differentiation program of proinflammatory IL-17+ T helper cells

Induction of intestinal Th17 cells by segmented filamentous bacteria

Making sense of latent TGFbeta activation

Transforming growth factor-beta induces development of the T(H)17 lineage

STAT3 regulates cytokine-mediated generation of inflammatory helper T cells

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547-554

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10.1038/NM.2684

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4

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18

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221886180

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22388091

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3321096

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