Suppression of PGC-1α Is Critical for Reprogramming Oxidative Metabolism in Renal Cell Carcinoma
about
Novel drugs that target the metabolic reprogramming in renal cell cancerThe ever-expanding role of HIF in tumour and stromal biologyMuscle Arnt/Hif1β Is Dispensable in Myofiber Type Determination, Vascularization and Insulin Sensitivity.The metabolic co-regulator PGC1α suppresses prostate cancer metastasis.Role of Mitochondrial DNA Copy Number Alteration in Human Renal Cell Carcinoma.Skeletal muscle hypoxia-inducible factor-1 and exercise.PGC1α drives a metabolic block on prostate cancer progression.The Anti-Warburg Effect Elicited by the cAMP-PGC1α Pathway Drives Differentiation of Glioblastoma Cells into Astrocytes.LINC00152: A pivotal oncogenic long non-coding RNA in human cancers.PGC-1 Coactivators: Shepherding the Mitochondrial Biogenesis of Tumors.Non-electron transfer chain mitochondrial defects differently regulate HIF-1α degradation and transcription.Reactive oxygen species (ROS) are a key determinant of cancer's metabolic phenotype.HIF drives lipid deposition and cancer in ccRCC via repression of fatty acid metabolism.PGC1α: Friend or Foe in Cancer?Hypoxia inducible factor (HIF) in the tumor microenvironment: friend or foe?Lead promotes abnormal angiogenesis induced by CCM3 gene defects via mitochondrial pathway.Teaching the basics of reactive oxygen species and their relevance to cancer biology: Mitochondrial reactive oxygen species detection, redox signaling, and targeted therapies.Suppression of homology-dependent DNA double-strand break repair induces PARP inhibitor sensitivity in VHL-deficient human renal cell carcinoma.PGC1α promotes cholangiocarcinoma metastasis by upregulating PDHA1 and MPC1 expression to reverse the Warburg effect.Peroxisome Proliferator-Activated Receptor γ and PGC-1α in Cancer: Dual Actions as Tumor Promoter and Suppressor.Mitochondria and Cancer.Deciphering the Dichotomous Effects of PGC-1α on Tumorigenesis and Metastasis.MicroRNA-21-5p induces the metastatic phenotype of human cervical carcinoma cells in vitro by targeting the von Hippel-Lindau tumor suppressor.Traditional Chinese medicine Qiliqiangxin attenuates phenylephrine-induced cardiac hypertrophy via upregulating PPARγ and PGC-1α.Hypoxia-Inducible Factor 2-Dependent Pathways Driving Von Hippel-Lindau-Deficient Renal Cancer.PPARγ is dispensable for clear cell renal cell carcinoma progression.Metabolic Plasticity of Tumor Cell MitochondriaFatty acid oxidation is associated with proliferation and prognosis in breast and other cancers
P2860
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P2860
Suppression of PGC-1α Is Critical for Reprogramming Oxidative Metabolism in Renal Cell Carcinoma
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
Suppression of PGC-1α Is Criti ...... bolism in Renal Cell Carcinoma
@ast
Suppression of PGC-1α Is Criti ...... bolism in Renal Cell Carcinoma
@en
type
label
Suppression of PGC-1α Is Criti ...... bolism in Renal Cell Carcinoma
@ast
Suppression of PGC-1α Is Criti ...... bolism in Renal Cell Carcinoma
@en
prefLabel
Suppression of PGC-1α Is Criti ...... bolism in Renal Cell Carcinoma
@ast
Suppression of PGC-1α Is Criti ...... bolism in Renal Cell Carcinoma
@en
P2093
P2860
P1433
P1476
Suppression of PGC-1α Is Criti ...... bolism in Renal Cell Carcinoma
@en
P2093
Adam D Richardson
Amato J Giaccia
Chien-Kuang Cornelia Ding
Colleen Wu
Cullen M Taniguchi
David A Scott
Edward L LaGory
Rie von Eyben
P2860
P304
P356
10.1016/J.CELREP.2015.06.006
P577
2015-06-25T00:00:00Z