about
The Role of Oxidative Stress in Myocardial Ischemia and Reperfusion Injury and Remodeling: RevisitedMetabolic, autophagic, and mitophagic activities in cancer initiation and progressionQuality control systems in cardiac agingMitochondrial dysfunction in cardiac agingMitochondrial health, the epigenome and healthspanProtective mechanisms of mitochondria and heart function in diabetesMyopathic lamin mutations cause reductive stress and activate the nrf2/keap-1 pathwayOverexpression of Ubiquinol-Cytochrome c Reductase Core Protein 1 May Protect H9c2 Cardiac Cells by Binding with Zinc.Mitochondrial respiration and ROS emission during β-oxidation in the heart: An experimental-computational study.Mitochondrial and cellular mechanisms for managing lipid excess.Impact of glucose-6-phosphate dehydrogenase deficiency on the pathophysiology of cardiovascular disease.Increased reactive oxygen species production during reductive stress: The roles of mitochondrial glutathione and thioredoxin reductases.Small heat shock proteins in redox metabolism: implications for cardiovascular diseases.Reductive stress linked to small HSPs, G6PD, and Nrf2 pathways in heart diseaseDeclining signal dependence of Nrf2-MafS-regulated gene expression correlates with aging phenotypes.Human heart failure is accompanied by altered protein kinase A subunit expression and post-translational state.Assessing Cardiac Metabolism: A Scientific Statement From the American Heart Association.Roles for the sarco-/endoplasmic reticulum in cardiac myocyte contraction, protein synthesis, and protein quality control.Mitochondrial dysfunction, alternans, and arrhythmiasReductive Stress in Inflammation-Associated Diseases and the Pro-Oxidant Effect of Antioxidant Agents.Update on innovative initiatives for the American Journal of Physiology-Heart and Circulatory Physiology.Stable Isotope Labeling Reveals Novel Insights Into Ubiquitin-Mediated Protein Aggregation With Age, Calorie Restriction, and Rapamycin Treatment.Cardiac-specific ablation of the E3 ubiquitin ligase Mdm2 leads to oxidative stress, broad mitochondrial deficiency and early death.Thoughts modulate the expression of inflammatory genes and may improve the coronary blood flow in patients after a myocardial infarction.Development of a Photoactivatable Phosphine Probe for Induction of Intracellular Reductive Stress with Single-Cell Precision.Protein Turnover in Aging and Longevity.
P2860
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P2860
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年学术文章
@wuu
2011年学术文章
@zh-cn
2011年学术文章
@zh-hans
2011年学术文章
@zh-my
2011年学术文章
@zh-sg
2011年學術文章
@yue
2011年學術文章
@zh
2011年學術文章
@zh-hant
name
Proteostasis and REDOX state in the heart.
@ast
Proteostasis and REDOX state in the heart.
@en
type
label
Proteostasis and REDOX state in the heart.
@ast
Proteostasis and REDOX state in the heart.
@en
prefLabel
Proteostasis and REDOX state in the heart.
@ast
Proteostasis and REDOX state in the heart.
@en
P2860
P1476
Proteostasis and REDOX state in the heart.
@en
P2093
Elisabeth S Christians
Ivor J Benjamin
P2860
P304
P356
10.1152/AJPHEART.00903.2011
P577
2011-10-14T00:00:00Z