Prostaglandin E2 suppresses antifungal immunity by inhibiting interferon regulatory factor 4 function and interleukin-17 expression in T cells. - Wikidata - wikimedia - TriplyDB

Prostaglandin E2 suppresses antifungal immunity by inhibiting interferon regulatory factor 4 function and interleukin-17 expression in T cells.

Prostaglandin E2 suppresses antifungal immunity by inhibiting interferon regulatory factor 4 function and interleukin-17 expression in T cells.

http://www.wikidata.org/entity/Q35907174

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Production of cross-kingdom oxylipins by pathogenic fungi: An update on their role in development and pathogenicity Endemic mycoses in patients with STAT3-mutated hyper-IgE (Job) syndrome Mesenchymal stem cells repress Th17 molecular program through the PD-1 pathway.Scavenger receptor A modulates the immune response to pulmonary Cryptococcus neoformans infection.Decreased RORC-dependent silencing of prostaglandin receptor EP2 induces autoimmune Th17 cells.Fatty acids, lipid mediators, and T-cell function.Immune modulation mediated by cryptococcal laccase promotes pulmonary growth and brain dissemination of virulent Cryptococcus neoformans in mice.Negative regulation of pulmonary Th17 responses by C3a anaphylatoxin during allergic inflammation in mice.Immune suppressive effects of tonsil-derived mesenchymal stem cells on mouse bone-marrow-derived dendritic cells.Exosome-like nanoparticles from intestinal mucosal cells carry prostaglandin E2 and suppress activation of liver NKT cells.Interleukin-22: immunobiology and pathology.Type I IFN Induction via Poly-ICLC Protects Mice against Cryptococcosis.Blockage of Eosinopoiesis by IL-17A Is Prevented by Cytokine and Lipid Mediators of Allergic Inflammation.Gut Microbiota Regulates K/BxN Autoimmune Arthritis through Follicular Helper T but Not Th17 Cells.Cryptococcus neoformans growth and protection from innate immunity are dependent on expression of a virulence-associated DEAD-box protein, Vad1.HVEM: An unusual TNF receptor family member important for mucosal innate immune responses to microbes.Cigarette smoke inhibits ROCK2 activation in T cells and modulates IL-22 production Susceptibility of intact germinating Arabidopsis thaliana to human fungal pathogens Cryptococcus neoformans and C. gattii.Signaling via the IL-20 receptor inhibits cutaneous production of IL-1β and IL-17A to promote infection with methicillin-resistant Staphylococcus aureus.Role of laccase in the virulence of Talaromyces marneffei: A common link between AIDS-related fungal pathogens?Parental dietary fat intake alters offspring microbiome and immunity.Inhibition of protein translation as a novel mechanism for prostaglandin E2 regulation of cell functions.Interleukin-17A enhances host defense against cryptococcal lung infection through effects mediated by leukocyte recruitment, activation, and gamma interferon production Mechanisms of infection by the human fungal pathogen Cryptococcus neoformans.IL-22, not simply a Th17 cytokine.Lipid mediators in immune dysfunction after severe inflammation.IRF4 and IRF8 Act in CD11c+ Cells To Regulate Terminal Differentiation of Lung Tissue Dendritic Cells.Distinctive role of efferocytosis in dendritic cell maturation and migration in sterile or infectious conditions.IL-33 Signaling Regulates Innate IL-17A and IL-22 Production via Suppression of Prostaglandin E2 during Lung Fungal Infection.CD8+ T cells produce a dialyzable antigen-specific activator of dendritic cells.Dermatophytes activate skin keratinocytes via mitogen-activated protein kinase signaling and induce immune responses Exploitation of Scavenger Receptor, Macrophage Receptor with Collagenous Structure, by Cryptococcus neoformans Promotes Alternative Activation of Pulmonary Lymph Node CD11b+ Conventional Dendritic Cells and Non-Protective Th2 Bias.IL-17 in neonatal health and disease.Prostaglandin E2 as a Regulator of Immunity to Pathogens.Masking the Pathogen: Evolutionary Strategies of Fungi and Their Bacterial Counterparts.Contribution of IL-1RI Signaling to Protection against Cryptococcus neoformans 52D in a Mouse Model of Infection.Intracellular cAMP Sensor EPAC: Physiology, Pathophysiology, and Therapeutics Development.Immunology of Cryptococcal Infections: Developing a Rational Approach to Patient Therapy.

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P2860

Prostaglandin E2 suppresses antifungal immunity by inhibiting interferon regulatory factor 4 function and interleukin-17 expression in T cells.

http://www.wikidata.org/entity/Q35907174

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2012 nî lūn-bûn

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Arthur L Shaffer

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Brian M Janelsins

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Patricia A Valdez

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Paul J Vithayathil

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Peter R Williamson

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P2860

Selective regulatory function of Socs3 in the formation of IL-17-secreting T cells

The biological functions of T helper 17 cell effector cytokines in inflammation

Lineage-specific modulation of interleukin 4 signaling by interferon regulatory factor 4

The orphan nuclear receptor RORgammat directs the differentiation program of proinflammatory IL-17+ T helper cells

STAT3 mutations in the hyper-IgE syndrome

Bcl6 mediates the development of T follicular helper cells

Control of T(reg) and T(H)17 cell differentiation by the aryl hydrocarbon receptor

STAT3 regulates cytokine-mediated generation of inflammatory helper T cells

T helper 17 lineage differentiation is programmed by orphan nuclear receptors ROR alpha and ROR gamma

Prostaglandin E2-EP4 signaling promotes immune inflammation through Th1 cell differentiation and Th17 cell expansion

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