Replication blocking lesions present a unique substrate for homologous recombination.
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A role for the malignant brain tumour (MBT) domain protein LIN-61 in DNA double-strand break repair by homologous recombinationJoint molecule resolution requires the redundant activities of MUS-81 and XPF-1 during Caenorhabditis elegans meiosisCaenorhabditis elegans HIM-18/SLX-4 interacts with SLX-1 and XPF-1 and maintains genomic integrity in the germline by processing recombination intermediatesRad51 Paralogs Remodel Pre-synaptic Rad51 Filaments to Stimulate Homologous Recombination.Homologous recombinational repair factors are recruited and loaded onto the viral DNA genome in Epstein-Barr virus replication compartments.Synthetic cytotoxicity: digenic interactions with TEL1/ATM mutations reveal sensitivity to low doses of camptothecinTax impairs DNA replication forks and increases DNA breaks in specific oncogenic genome regionsC. elegans ring finger protein RNF-113 is involved in interstrand DNA crosslink repair and interacts with a RAD51C homolog.DNA double-strand break repair in Caenorhabditis elegansBioinformatics analysis identify novel OB fold protein coding genes in C. elegans.Interactions among Trypanosoma brucei RAD51 paralogues in DNA repair and antigenic variation.Homologous recombination repair is essential for repair of vosaroxin-induced DNA double-strand breaks.Chromosomal Integrity after UV Irradiation Requires FANCD2-Mediated Repair of Double Strand Breaks.JMJD-5/KDM8 regulates H3K36me2 and is required for late steps of homologous recombination and genome integrity.DOG-1 is the Caenorhabditis elegans BRIP1/FANCJ homologue and functions in interstrand cross-link repairDNA Damage Sensitivity Assays in Caenorhabditis elegans.Promotion of Homologous Recombination by SWS-1 in Complex with RAD-51 Paralogs in Caenorhabditis elegans.ERCC1 is required for FANCD2 focus formation.Homologous recombination rescues ssDNA gaps generated by nucleotide excision repair and reduced translesion DNA synthesis in yeast G2 cells.DNA damage leads to progressive replicative decline but extends the life span of long-lived mutant animals.Repair of gaps opposite lesions by homologous recombination in mammalian cells.A Polar and Nucleotide-Dependent Mechanism of Action for RAD51 Paralogs in RAD51 Filament Remodeling.DNA interstrand crosslink repair in mammalian cells.LINC complexes promote homologous recombination in part through inhibition of nonhomologous end joining.CX-5461 is a DNA G-quadruplex stabilizer with selective lethality in BRCA1/2 deficient tumoursLoss of Caenorhabditis elegans BRCA1 promotes genome stability during replication in smc-5 mutantsMetabolism of postsynaptic recombination intermediates.Homologous recombination as a replication fork escort: fork-protection and recovery.Genome integrity is regulated by the Caenorhabditis elegans Rad51D homolog rfs-1Stalled replication forks generate a distinct mutational signature in yeast.The Shu complex is a conserved regulator of homologous recombination.FancJ/Brip1 helicase protects against genomic losses and gains in vertebrate cells.Methodological considerations for mutagen exposure in C. elegans.FANCM-associated proteins MHF1 and MHF2, but not the other Fanconi anemia factors, limit meiotic crossovers.Genomic stability: FANCJ-dependent G4 DNA repair.
P2860
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P2860
Replication blocking lesions present a unique substrate for homologous recombination.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Replication blocking lesions present a unique substrate for homologous recombination.
@ast
Replication blocking lesions present a unique substrate for homologous recombination.
@en
type
label
Replication blocking lesions present a unique substrate for homologous recombination.
@ast
Replication blocking lesions present a unique substrate for homologous recombination.
@en
prefLabel
Replication blocking lesions present a unique substrate for homologous recombination.
@ast
Replication blocking lesions present a unique substrate for homologous recombination.
@en
P2093
P2860
P356
P1433
P1476
Replication blocking lesions present a unique substrate for homologous recombination.
@en
P2093
Jordan D Ward
Judith Yanowitz
Louise J Barber
Mark Ir Petalcorin
Simon J Boulton
P2860
P304
P356
10.1038/SJ.EMBOJ.7601766
P407
P577
2007-07-05T00:00:00Z