TP53 Silencing Bypasses Growth Arrest of BRAFV600E-Induced Lung Tumor Cells in a Two-Switch Model of Lung Tumorigenesis.
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Context-Dependent Effects of Amplified MAPK Signaling during Lung Adenocarcinoma Initiation and Progression.The generation and characterization of novel Col1a1FRT-Cre-ER-T2-FRT and Col1a1FRT-STOP-FRT-Cre-ER-T2 mice for sequential mutagenesis.TFAP2C promotes lung tumorigenesis and aggressiveness through miR-183- and miR-33a-mediated cell cycle regulation.The Regulation of Cellular Functions by the p53 Protein: Cellular Senescence.p53 loss does not permit escape from BrafV600E-induced senescence in a mouse model of lung cancer.
P2860
TP53 Silencing Bypasses Growth Arrest of BRAFV600E-Induced Lung Tumor Cells in a Two-Switch Model of Lung Tumorigenesis.
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2015 nî lūn-bûn
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2015年の論文
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2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
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2015年论文
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name
TP53 Silencing Bypasses Growth ...... h Model of Lung Tumorigenesis.
@ast
TP53 Silencing Bypasses Growth ...... h Model of Lung Tumorigenesis.
@en
type
label
TP53 Silencing Bypasses Growth ...... h Model of Lung Tumorigenesis.
@ast
TP53 Silencing Bypasses Growth ...... h Model of Lung Tumorigenesis.
@en
prefLabel
TP53 Silencing Bypasses Growth ...... h Model of Lung Tumorigenesis.
@ast
TP53 Silencing Bypasses Growth ...... h Model of Lung Tumorigenesis.
@en
P2093
P2860
P1433
P1476
TP53 Silencing Bypasses Growth ...... h Model of Lung Tumorigenesis.
@en
P2093
David Dankort
Joseph Juan
Martin McMahon
Shon Green
P2860
P304
P356
10.1158/0008-5472.CAN-14-3701
P407
P577
2015-05-22T00:00:00Z