Sequence-specific recruitment of heterochromatin protein 1 via interaction with Krüppel-like factor 11, a human transcription factor involved in tumor suppression and metabolic diseases
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Functional characterization of EZH2β reveals the increased complexity of EZH2 isoforms involved in the regulation of mammalian gene expressionNew role for Kruppel-like factor 14 as a transcriptional activator involved in the generation of signaling lipidsThe Triple-Code Model for Pancreatic Cancer: Cross Talk Among Genetics, Epigenetics, and Nuclear StructureUbiquitination of Lysine 867 of the Human SETDB1 Protein Upregulates Its Histone H3 Lysine 9 (H3K9) Methyltransferase ActivityKrüppel-like factor 11 regulates the expression of metabolic genes via an evolutionarily conserved protein interaction domain functionally disrupted in maturity onset diabetes of the young.Single and combinatorial chromatin coupling events underlies the function of transcript factor Krüppel-like factor 11 in the regulation of gene networks.A role for SUV39H1-mediated H3K9 trimethylation in the control of genome stability and senescence in WI38 human diploid lung fibroblastsDifferential coupling of KLF10 to Sin3-HDAC and PCAF regulates the inducibility of the FOXP3 geneA novel role of the Sp/KLF transcription factor KLF11 in arresting progression of endometriosis.Role for Krüppel-like transcription factor 11 in mesenchymal cell function and fibrosisBrowning of human adipocytes requires KLF11 and reprogramming of PPARγ superenhancers.Krüppel-like factors are effectors of nuclear receptor signaling.Targeting protein-protein interfaces using macrocyclic peptides.Krüppel-like factor 11 differentially couples to histone acetyltransferase and histone methyltransferase chromatin remodeling pathways to transcriptionally regulate dopamine D2 receptor in neuronal cells.Phenotypic Characterization of Mice Carrying Homozygous Deletion of KLF11, a Gene in Which Mutations Cause Human Neonatal and MODY VII Diabetes.Evidence supporting a critical contribution of intrinsically disordered regions to the biochemical behavior of full-length human HP1γ.Chronic Social Stress and Ethanol Increase Expression of KLF11, a Cell Death Mediator, in Rat BrainKrüppel-like factor-11, a transcription factor involved in diabetes mellitus, suppresses endothelial cell activation via the nuclear factor-κB signaling pathway.The expression of KLF11 (TIEG2), a monoamine oxidase B transcriptional activator in the prefrontal cortex of human alcohol dependence.Insights into the epigenetic mechanisms controlling pancreatic carcinogenesis.KLF11 is an Epigenetic Mediator of DRD2/Dopaminergic Signaling in Endometriosis.Binge ethanol exposure increases the Krüppel-like factor 11-monoamine oxidase (MAO) pathway in rats: Examining the use of MAO inhibitors to prevent ethanol-induced brain injurySUV39H1 mediated SIRT1 trans-repression contributes to cardiac ischemia-reperfusion injury.KLF11 epigenetically regulates glycodelin-A, a marker of endometrial biology via histone-modifying chromatin mechanisms.Unraveling transcription factor interactions with heterochromatin protein 1 using fluorescence lifetime imaging microscopy and fluorescence correlation spectroscopyThe promise of epigenomic therapeutics in pancreatic cancerEpigenetic Regulation of Cellular Senescence and Aging.Krüppel-like factor 5 as potential molecular marker in cervical cancer and the KLF family profile expression.Krüppel-like factors and vascular wall homeostasis.
P2860
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P2860
Sequence-specific recruitment of heterochromatin protein 1 via interaction with Krüppel-like factor 11, a human transcription factor involved in tumor suppression and metabolic diseases
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Sequence-specific recruitment ...... ression and metabolic diseases
@ast
Sequence-specific recruitment ...... ression and metabolic diseases
@en
type
label
Sequence-specific recruitment ...... ression and metabolic diseases
@ast
Sequence-specific recruitment ...... ression and metabolic diseases
@en
prefLabel
Sequence-specific recruitment ...... ression and metabolic diseases
@ast
Sequence-specific recruitment ...... ression and metabolic diseases
@en
P2093
P2860
P356
P1476
Sequence-specific recruitment ...... ression and metabolic diseases
@en
P2093
Adrienne Grzenda
Angela J Mathison
Cathrine J DeMars
Ezequiel Calvo
Gwen Lomberk
Juan Iovanna
Juliana Bonilla-Velez
Martin E Fernandez-Zapico
Navtej S Buttar
Raul Urrutia
P2860
P304
13026-13039
P356
10.1074/JBC.M112.342634
P407
P577
2012-02-08T00:00:00Z