The role of SHIP1 in macrophage programming and activation.
about
Identification of mitogen-activated protein kinase docking sites in enzymes that metabolize phosphatidylinositols and inositol phosphates.The SH2-domain-containing inositol 5-phosphatase (SHIP) limits the motility of neutrophils and their recruitment to wounds in zebrafishGlobal protein phosphorylation dynamics during deoxynivalenol-induced ribotoxic stress response in the macrophage.Bu Fei Decoction attenuates the tumor associated macrophage stimulated proliferation, migration, invasion and immunosuppression of non-small cell lung cancer, partially via IL-10 and PD-L1 regulation.Elevated miR-155 promotes inflammation in cystic fibrosis by driving hyperexpression of interleukin-8MyD88-dependent SHIP1 regulates proinflammatory signaling pathways in dendritic cells after monophosphoryl lipid A stimulation of TLR4.Macrophages in tumor microenvironments and the progression of tumors.Bench-to-bedside review: endotoxin tolerance as a model of leukocyte reprogramming in sepsisThe inositol phosphatase SHIP controls Salmonella enterica serovar Typhimurium infection in vivoTargeting tumour-associated macrophages.MiR-155-regulated molecular network orchestrates cell fate in the innate and adaptive immune response to Mycobacterium tuberculosis.Arginase: an emerging key player in the mammalian immune system.Tumor-associated macrophages (TAM) as major players of the cancer-related inflammation.Non-cell-autonomous tumor suppression: oncogene-provoked apoptosis promotes tumor cell senescence via stromal crosstalk.NOS-2 signaling and cancer therapy.Signaling mechanisms regulating myelination in the central nervous system.MKP-1 switches arginine metabolism from nitric oxide synthase to arginase following endotoxin challenge.The siRNA cocktail targeting interleukin 10 receptor and transforming growth factor-β receptor on dendritic cells potentiates tumour antigen-specific CD8(+) T cell immunity.SHIP1 intrinsically regulates NK cell signaling and education, resulting in tolerance of an MHC class I-mismatched bone marrow graft in miceA systemic granulomatous response to Schistosoma mansoni eggs alters responsiveness of bone-marrow-derived macrophages to Toll-like receptor agonists.Arginase 1 is a negative regulator of osteoclast differentiation.
P2860
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P2860
The role of SHIP1 in macrophage programming and activation.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
The role of SHIP1 in macrophage programming and activation.
@ast
The role of SHIP1 in macrophage programming and activation.
@en
type
label
The role of SHIP1 in macrophage programming and activation.
@ast
The role of SHIP1 in macrophage programming and activation.
@en
prefLabel
The role of SHIP1 in macrophage programming and activation.
@ast
The role of SHIP1 in macrophage programming and activation.
@en
P2093
P356
P1476
The role of SHIP1 in macrophage programming and activation
@en
P2093
P304
P356
10.1042/BST0320785
P577
2004-11-01T00:00:00Z