Bidirectional crosstalk between PD-L1 expression and epithelial to mesenchymal transition: significance in claudin-low breast cancer cells - Wikidata - wikimedia - TriplyDB

Bidirectional crosstalk between PD-L1 expression and epithelial to mesenchymal transition: significance in claudin-low breast cancer cells

Bidirectional crosstalk between PD-L1 expression and epithelial to mesenchymal transition: significance in claudin-low breast cancer cells

http://www.wikidata.org/entity/Q35922960

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Homeostatic Signaling by Cell-Cell Junctions and Its Dysregulation during Cancer Progression Aryl hydrocarbon receptor/cytochrome P450 1A1 pathway mediates breast cancer stem cells expansion through PTEN inhibition and β-Catenin and Akt activation Initiation and Maintenance of Gastric Cancer: A Focus on CD44 Variant Isoforms and Cancer Stem Cells.Transcription Factor Networks derived from Breast Cancer Stem Cells control the immune response in the Basal subtype.Programmed death-ligand 1 expression at tumor invasive front is associated with epithelial-mesenchymal transition and poor prognosis in esophageal squamous cell carcinoma.New insights into the role of EMT in tumor immune escape.The PD-1, PD-L1 expression and CD3+ T cell infiltration in relation to outcome in advanced gastric signet-ring cell carcinoma, representing a potential biomarker for immunotherapy.Cellular Expression of PD-L1 in the Peripheral Blood of Lung Cancer Patients is Associated with Worse Survival.Drug resistance originating from a TGF-β/FGF-2-driven epithelial-to-mesenchymal transition and its reversion in human lung adenocarcinoma cell lines harboring an EGFR mutation.Intra-tumoral heterogeneity in the expression of programmed-death (PD) ligands in isogeneic primary and metastatic lung cancer: Implications for immunotherapy.Knockdown of PD-L1 in Human Gastric Cancer Cells Inhibits Tumor Progression and Improves the Cytotoxic Sensitivity to CIK Therapy.STAT3-targeted treatment with silibinin overcomes the acquired resistance to crizotinib in ALK-rearranged lung cancer.Epithelial-mesenchymal plasticity and circulating tumor cells: Travel companions to metastases.Circulating and disseminated tumour cells - mechanisms of immune surveillance and escape.PD-L1 promotes OCT4 and Nanog expression in breast cancer stem cells by sustaining PI3K/AKT pathway activation.Silencing PRDM14 expression by an innovative RNAi therapy inhibits stemness, tumorigenicity, and metastasis of breast cancer.Epithelial-Mesenchymal Transition and Immune Evasion during Lung Cancer Progression: The Chicken or the Egg?Type Iγ phosphatidylinositol phosphate kinase regulates PD-L1 expression by activating NF-κB.Dramatic response of metaplastic breast cancer to chemo-immunotherapy.Characterisation of PD-L1-positive subsets of microsatellite-unstable colorectal cancers.A novel bifunctional anti-PD-L1/TGF-β Trap fusion protein (M7824) efficiently reverts mesenchymalization of human lung cancer cells.Crk adaptor protein promotes PD-L1 expression, EMT and immune evasion in a murine model of triple-negative breast cancer.Manganese-12 acetate suppresses the migration, invasion, and epithelial-mesenchymal transition by inhibiting Wnt/β-catenin and PI3K/AKT signaling pathways in breast cancer cells.Clinical significance of programmed cell death-ligand 1 expression and the immune microenvironment at the invasive front of colorectal cancers with high microsatellite instability.MUC1-C activates polycomb repressive complexes and downregulates tumor suppressor genes in human cancer cells.PD-L1 and epithelial-mesenchymal transition in circulating tumor cells from non-small cell lung cancer patients: A molecular shield to evade immune system?Prognostic significance of PD-L1 expression on tumor cells and tumor-infiltrating mononuclear cells in upper tract urothelial carcinoma.ATM‑JAK‑PD‑L1 signaling pathway inhibition decreases EMT and metastasis of androgen‑independent prostate cancer.MUC1-C Induces PD-L1 and Immune Evasion in Triple-Negative Breast Cancer.Prognostic impact of programmed cell death ligand 1 (PD-L1) expression and its association with epithelial-mesenchymal transition in extrahepatic cholangiocarcinoma.Monitoring Immune Checkpoint Regulators as Predictive Biomarkers in Hepatocellular Carcinoma Tumor-Intrinsic PD-L1 Signaling in Cancer Initiation, Development and Treatment: Beyond Immune Evasion Relationship between epithelial-to-mesenchymal transition and the inflammatory microenvironment of hepatocellular carcinoma Cellular Phenotype Plasticity in Cancer Dormancy and Metastasis

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Bidirectional crosstalk between PD-L1 expression and epithelial to mesenchymal transition: significance in claudin-low breast cancer cells

http://www.wikidata.org/entity/Q35922960

description

2015 nî lūn-bûn

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Asma Tulbah

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Hanaa Fitwi

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P2860

Costimulatory B7-H1 in renal cell carcinoma patients: Indicator of tumor aggressiveness and potential therapeutic target

Comprehensive molecular portraits of human breast tumours

The epithelial-mesenchymal transition generates cells with properties of stem cells

Engagement of the PD-1 immunoinhibitory receptor by a novel B7 family member leads to negative regulation of lymphocyte activation

The immunoinhibitory B7-H1 molecule as a potential target in cancer: killing many birds with one stone

Use of chemotherapy plus a monoclonal antibody against HER2 for metastatic breast cancer that overexpresses HER2.

Safety and Activity of Anti–PD-L1 Antibody in Patients with Advanced Cancer

Identification of human triple-negative breast cancer subtypes and preclinical models for selection of targeted therapies

Phenotypic and molecular characterization of the claudin-low intrinsic subtype of breast cancer

Loss of tumor suppressor PTEN function increases B7-H1 expression and immunoresistance in glioma

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