CD8+ T cells promote inflammation and apoptosis in the liver after sepsis: role of Fas-FasL.
about
The role of the liver in sepsisFas ligand-expressing lymphocytes enhance alveolar macrophage apoptosis in the resolution of acute pulmonary inflammation.Receptor-interacting protein kinase 3 deficiency inhibits immune cell infiltration and attenuates organ injury in sepsisFull activation of CD4+ T cells early during sepsis requires specific antigen.Regulation of lymphocyte trafficking by CXC chemokine receptor 3 during septic shockMechanisms of methicillin-resistant Staphylococcus aureus pneumonia-induced intestinal epithelial apoptosisMitochondrial ROS Induces Cardiac Inflammation via a Pathway through mtDNA Damage in a Pneumonia-Related Sepsis Model.Differential alterations of tissue T-cell subsets after sepsis.The dual functions of receptor interacting protein 1 in fas-induced hepatocyte death during sepsis.Viral inhibitor of apoptosis vFLIP/K13 protects endothelial cells against superoxide-induced cell death.Association between apoptotis and CD4(+)/CD8(+) T-lymphocyte ratio in aseptic loosening after total hip replacementThe role of hepatic invariant NKT cells in systemic/local inflammation and mortality during polymicrobial septic shockKupffer cells potentiate liver sinusoidal endothelial cell injury in sepsis by ligating programmed cell death ligand-1.MitoQ administration prevents endotoxin-induced cardiac dysfunction.Caspase and calpain activation both contribute to sepsis-induced diaphragmatic weakness.IL-10 helps control pathogen load during high-level bacteremia.Neutrophil-endothelial interactions mediate angiopoietin-2-associated pulmonary endothelial cell dysfunction in indirect acute lung injury in miceClinical and Experimental Sepsis Impairs CD8 T-Cell-Mediated Immunity.Effect of PD-1: PD-L1 in Invariant Natural Killer T-Cell Emigration and Chemotaxis Following Sepsis.Role of Programmed Cell Death in the Immunopathogenesis of Sepsis.Activation of the peroxisome proliferator-activated receptor γ counteracts sepsis-induced T cell cytotoxicity toward alloantigenic target cells.Kupffer cells protect liver sinusoidal endothelial cells from Fas-dependent apoptosis in sepsis by down-regulating gp130.Decreases in activated CD8+ T cells in patients with severe hepatitis B are related to outcomes.Dexamethasone attenuates lipopolysaccharide-induced liver injury by downregulating glucocorticoid-induced tumor necrosis factor receptor ligand in Kupffer cells.IL-10 distinguishes a unique population of activated, effector-like CD8+ T cells in murine acute liver inflammation.
P2860
Q26864861-DAE077F8-795D-4E52-95FC-C1AE609CA1AEQ33840384-A59493A3-4CA2-40F9-8EAA-806D163DD478Q34485020-CBDFA365-0AFB-4544-A567-A647607FF4F3Q35013590-32879730-8DC0-4255-978E-B2A95A4DB43EQ35813689-36F3CD00-FB43-421C-96F3-C5D90961BA39Q36082831-019B69B1-8DE4-4F9B-A78F-C2082329F298Q36137078-A9D8427E-77DD-4680-A046-EE8A1ECF6200Q36259440-458169FC-40E1-427D-B78C-AFAEE904684EQ36394948-6EF2182D-267E-490D-AF98-81A076C51183Q37033342-B959F94A-22A7-45D8-B31E-807AB8D2993AQ37093201-A26B0EF7-E68D-4708-9920-6DCAE1A2FF41Q37122052-23F91A54-EB24-4796-AF4A-68441CDAEF03Q37238322-508CEC78-9E04-4536-83DF-352C209F9C9AQ37391879-67640261-04F4-414B-B82D-AE8F1F0F700EQ37422187-9662294B-ADE5-4AB0-9809-25B2240DE478Q37476110-B31E2EBB-2026-47B7-9029-A7750FDE7072Q37596104-D226C329-FC49-44B2-9F7C-89D6501EAF1AQ37649004-2128A88A-BA36-48AE-8E4D-1DD0D2BA2486Q40241359-40C1F319-F65E-428A-8283-8CE2E39BC2E0Q41114371-FCC8505E-13A4-4C98-97D4-92F536D6726AQ41563646-48EECF2B-57C0-4B48-9A0E-57859ECA9372Q41663817-E341E6A1-9B42-4990-B29B-3C25C1F901F7Q42202592-49BCDCBC-7863-4426-8253-D77F86328329Q42726554-BBCDBA95-89E9-4116-8032-AAA4D7A1A096Q51227665-4874349D-59BF-4FEF-9920-177387FB9DCC
P2860
CD8+ T cells promote inflammation and apoptosis in the liver after sepsis: role of Fas-FasL.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
CD8+ T cells promote inflammat ...... fter sepsis: role of Fas-FasL.
@ast
CD8+ T cells promote inflammat ...... fter sepsis: role of Fas-FasL.
@en
type
label
CD8+ T cells promote inflammat ...... fter sepsis: role of Fas-FasL.
@ast
CD8+ T cells promote inflammat ...... fter sepsis: role of Fas-FasL.
@en
prefLabel
CD8+ T cells promote inflammat ...... fter sepsis: role of Fas-FasL.
@ast
CD8+ T cells promote inflammat ...... fter sepsis: role of Fas-FasL.
@en
P2093
P2860
P1476
CD8+ T cells promote inflammat ...... fter sepsis: role of Fas-FasL.
@en
P2093
Carol A Ayala
Chun-Shiang Chung
Doreen E Wesche-Soldato
Stephen H Gregory
Thais P Salazar-Mather
P2860
P356
10.2353/AJPATH.2007.061099
P407
P50
P577
2007-07-01T00:00:00Z