IL-22 Protects Against Liver Pathology and Lethality of an Experimental Blood-Stage Malaria Infection.
about
CD4 T-cell subsets in malaria: TH1/TH2 revisitedHepatoprotective and anti-fibrotic functions of interleukin-22: therapeutic potential for the treatment of alcoholic liver diseaseCytokine profiles amongst Sudanese patients with visceral leishmaniasis and malaria co-infections.Liver-inherent immune system: its role in blood-stage malaria.Hemozoin induces hepatic inflammation in mice and is differentially associated with liver pathology depending on the Plasmodium strain.Pathogenesis of alcoholic liver disease: role of oxidative metabolism.Interleukin-22 ameliorates liver fibrogenesis by attenuating hepatic stellate cell activation and downregulating the levels of inflammatory cytokines.Disruption of IL-21 signaling affects T cell-B cell interactions and abrogates protective humoral immunity to malaria.Interleukin-22: immunobiology and pathology.IFNγ and IL-12 Restrict Th2 Responses during Helminth/Plasmodium Co-Infection and Promote IFNγ from Th2 CellsA Novel Model of Asymptomatic Plasmodium Parasitemia That Recapitulates Elements of the Human Immune Response to Chronic Infection.Environmental Correlation Analysis for Genes Associated with Protection against MalariaIL-22 dampens the T cell response in experimental malaria.Behavioural and neurological symptoms accompanied by cellular neuroinflammation in IL-10-deficient mice infected with Plasmodium chabaudi.A new hypothesis on the manifestation of cerebral malaria: the secret is in the liverA Functional IL22 Polymorphism (rs2227473) Is Associated with Predisposition to Childhood Cerebral Malaria.The cytokine IL-22 promotes pathogen colonization by suppressing related commensal bacteria.Pro- and anti-inflammatory cytokines in children with malaria in Franceville, Gabon.The aryl hydrocarbon receptor in innate T cell immunity.Therapeutic opportunities of the IL-22-IL-22R1 system.How Does Interleukin-22 Mediate Liver Regeneration and Prevent Injury and Fibrosis?Parasite-Specific CD4+ IFN-γ+ IL-10+ T Cells Distribute within Both Lymphoid and Nonlymphoid Compartments and Are Controlled Systemically by Interleukin-27 and ICOS during Blood-Stage Malaria InfectionIL-23 protection against Plasmodium berghei infection in mice is partially dependent on IL-17 from macrophages.Testosterone persistently dysregulates hepatic expression of Tlr6 and Tlr8 induced by Plasmodium chabaudi malaria.Early Inhibition of Fatty Acid Synthesis Reduces Generation of Memory Precursor Effector T Cells in Chronic Infection.Tissue-specific immunopathology during malaria infection.The role of IL-22 in the resolution of sterile and nonsterile inflammation.
P2860
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P2860
IL-22 Protects Against Liver Pathology and Lethality of an Experimental Blood-Stage Malaria Infection.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
IL-22 Protects Against Liver P ...... Blood-Stage Malaria Infection.
@ast
IL-22 Protects Against Liver P ...... Blood-Stage Malaria Infection.
@en
type
label
IL-22 Protects Against Liver P ...... Blood-Stage Malaria Infection.
@ast
IL-22 Protects Against Liver P ...... Blood-Stage Malaria Infection.
@en
prefLabel
IL-22 Protects Against Liver P ...... Blood-Stage Malaria Infection.
@ast
IL-22 Protects Against Liver P ...... Blood-Stage Malaria Infection.
@en
P2093
P2860
P50
P356
P1476
IL-22 Protects Against Liver P ...... Blood-Stage Malaria Infection.
@en
P2093
Ana Paula Freitas do Rosario
Anne-Marit Sponaas
Béatris Mastelic
Jean Christophe Renauld
Sophie Roetynck
William Jarra
P2860
P356
10.3389/FIMMU.2012.00085
P577
2012-04-25T00:00:00Z