Advanced glycation end products induce glomerular sclerosis and albuminuria in normal rats - Wikidata - wikimedia - TriplyDB

Advanced glycation end products induce glomerular sclerosis and albuminuria in normal rats

Advanced glycation end products induce glomerular sclerosis and albuminuria in normal rats

http://www.wikidata.org/entity/Q35931294

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Immunohistochemical detection of imidazolone, a novel advanced glycation end product, in kidneys and aortas of diabetic patients Advanced glycation endproduct (AGE) receptor 1 is a negative regulator of the inflammatory response to AGE in mesangial cells Orally absorbed reactive glycation products (glycotoxins): an environmental risk factor in diabetic nephropathy Current perspectives on the health risks associated with the consumption of advanced glycation end products: recommendations for dietary management Type 2 diabetes and cognitive impairment: linking mechanisms Diabetic Kidney Disease: Pathophysiology and Therapeutic Targets Development and prevention of advanced diabetic nephropathy in RAGE-overexpressing mice Advanced glycation: an important pathological event in diabetic and age related ocular disease Advanced glycation end products cause epithelial-myofibroblast transdifferentiation via the receptor for advanced glycation end products (RAGE)Advanced glycation endproducts trigger autophagy in cadiomyocyte via RAGE/PI3K/AKT/mTOR pathway Pimagedine: a novel therapy for diabetic nephropathy.[Protein glycation as a pathological mechanism in diabetes].Type IV collagen is transcriptionally regulated by Smad1 under advanced glycation end product (AGE) stimulation.Protective effect of resveratrol on oxidative damage in male and female stroke-prone spontaneously hypertensive rats.Receptors for advanced glycosylation endproducts in human brain: role in brain homeostasis Human glomerular endothelium: interplay among glucose, free fatty acids, angiotensin II, and oxidative stress.Dietary Metabolites and Chronic Kidney Disease.Mechanisms of metabolic memory and renal hypoxia as a therapeutic target in diabetic kidney disease.Serum carboxymethyl-lysine, a dominant advanced glycation end product, is associated with chronic kidney disease: the Baltimore longitudinal study of aging.SOCS3 overexpression inhibits advanced glycation end product-induced EMT in proximal tubule epithelial cells Role of transcription factor acetylation in diabetic kidney disease.Autocrine and paracrine mechanisms in the early stages of diabetic nephropathy.Alterations in T2 relaxation magnetic resonance imaging of the ovine intervertebral disc due to nonenzymatic glycation.Deletion of the receptor for advanced glycation end products reduces glomerulosclerosis and preserves renal function in the diabetic OVE26 mouse.Neurotoxicity of advanced glycation endproducts during focal stroke and neuroprotective effects of aminoguanidine Therapeutic potential of anti-oxidants and diabetic retinopathy.Therapeutic potential of AGE inhibitors and breakers of AGE protein cross-links.Advanced glycation and the immune system: stimulation, inhibition or both?Endothelial dysfunction in children with type 1 diabetes mellitus.-374 T/A RAGE polymorphism is associated with chronic kidney disease progression in subjects affected by nephrocardiovascular disease.Effects of pyridoxamine in combined phase 2 studies of patients with type 1 and type 2 diabetes and overt nephropathy.Caloric restriction mimetics: metabolic interventions.Advanced glycation end products and their circulating receptors predict cardiovascular disease mortality in older community-dwelling women.Preventing microvascular diabetic complications in children and adolescents: looking beyond glycaemic control.Advanced glycation end-products induce connective tissue growth factor-mediated renal fibrosis predominantly through transforming growth factor beta-independent pathway.Advanced glycation end products (AGEs) co-localize with AGE receptors in the retinal vasculature of diabetic and of AGE-infused rats.Aminoguanidine prevents age-related arterial stiffening and cardiac hypertrophy Improved diabetes control in the elderly delays global cognitive decline Glycation, oxidation, and lipoxidation in the development of the complications of diabetes: a carbonyl stress hypothesis Chronic Running Exercise Alleviates Early Progression of Nephropathy with Upregulation of Nitric Oxide Synthases and Suppression of Glycation in Zucker Diabetic Rats.

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P2860

Advanced glycation end products induce glomerular sclerosis and albuminuria in normal rats

http://www.wikidata.org/entity/Q35931294

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1994年论文

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1994年论文

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Advanced glycation end product ...... and albuminuria in normal rats

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Advanced glycation end product ...... and albuminuria in normal rats

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P2860

Human and rat mesangial cell receptors for glucose-modified proteins: potential role in kidney tissue remodelling and diabetic nephropathy.

Aging of proteins: immunological detection of a glucose-derived pyrrole formed during maillard reaction in vivo.

Advanced glycation end products up-regulate gene expression found in diabetic glomerular disease.

Modification of low density lipoprotein by advanced glycation end products contributes to the dyslipidemia of diabetes and renal insufficiency.

Exogenous advanced glycosylation end products induce complex vascular dysfunction in normal animals: a model for diabetic and aging complications.

Evidence for the central role of glomerular growth promoters in the development of sclerosis.

Effects of aging on the renal glomerulus.

Pathogenic effects of advanced glycosylation: biochemical, biologic, and clinical implications for diabetes and aging.

The kidney disease of diabetes mellitus (KDDM): a cell and molecular biology approach.

Aminoguanidine, a novel inhibitor of nitric oxide formation, prevents diabetic vascular dysfunction.

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