Retinoblastoma-repression of E2F-dependent transcription depends on the ability of the retinoblastoma protein to interact with E2F and is abrogated by the adenovirus E1A oncoprotein.
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DDB, a putative DNA repair protein, can function as a transcriptional partner of E2F1.Jumonji regulates cardiomyocyte proliferation via interaction with retinoblastoma proteinDual mechanisms of repression of E2F1 activity by the retinoblastoma gene productp53-dependent G1 arrest involves pRB-related proteins and is disrupted by the human papillomavirus 16 E7 oncoprotein.Deregulated transcription factor E2F-1 expression leads to S-phase entry and p53-mediated apoptosis.Computational modeling of signaling pathways mediating cell cycle checkpoint control and apoptotic responses to ionizing radiation-induced DNA damage.Functional interaction between human topoisomerase IIalpha and retinoblastoma protein.E2F-1-mediated transactivation is inhibited by complex formation with the retinoblastoma susceptibility gene product.The accumulation of an E2F-p130 transcriptional repressor distinguishes a G0 cell state from a G1 cell state.Transcription of the E2F-1 gene is rendered cell cycle dependent by E2F DNA-binding sites within its promoter.A bipartite nuclear localization signal in the retinoblastoma gene product and its importance for biological activityAssociation of the human papillomavirus type 16 E7 protein with the S-phase-specific E2F-cyclin A complex.Positive regulation of minichromosome maintenance gene expression, DNA replication, and cell transformation by a plant retinoblastoma gene.Ginsenoside Rh2-mediated G1 phase cell cycle arrest in human breast cancer cells is caused by p15 Ink4B and p27 Kip1-dependent inhibition of cyclin-dependent kinases.Interferons and interleukin-6 suppress the DNA-binding activity of E2F in growth-sensitive hematopoietic cells.trans-Activation by the hnRNP K protein involves an increase in RNA synthesis from the reporter genes.Involvement of E2F1 transcriptional activity in cadmium-induced cell-cycle arrest at G1 in human lung fibroblasts.The human papillomavirus type 16 E7 protein complements adenovirus type 5 E1A amino-terminus-dependent transactivation of adenovirus type 5 early genes and increases ATF and Oct-1 DNA binding activity.p21 Disrupts the interaction between cdk2 and the E2F-p130 complex.Negative regulation of transcription in eukaryotes.Functional characterization of the transcription silencer element located within the human Pi class glutathione S-transferase promoter.Activation of a retinoblastoma-protein-dependent pathway by sphingosineOncolytic adenoviruses as a therapeutic approach for osteosarcoma: A new hope.Cell-cycle Checkpoints and Aneuploidy on the Path to Cancer.The Cell Cycle: A Review
P2860
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P2860
Retinoblastoma-repression of E2F-dependent transcription depends on the ability of the retinoblastoma protein to interact with E2F and is abrogated by the adenovirus E1A oncoprotein.
description
1992 nî lūn-bûn
@nan
1992年の論文
@ja
1992年論文
@yue
1992年論文
@zh-hant
1992年論文
@zh-hk
1992年論文
@zh-mo
1992年論文
@zh-tw
1992年论文
@wuu
1992年论文
@zh
1992年论文
@zh-cn
name
Retinoblastoma-repression of E ...... he adenovirus E1A oncoprotein.
@ast
Retinoblastoma-repression of E ...... he adenovirus E1A oncoprotein.
@en
type
label
Retinoblastoma-repression of E ...... he adenovirus E1A oncoprotein.
@ast
Retinoblastoma-repression of E ...... he adenovirus E1A oncoprotein.
@en
prefLabel
Retinoblastoma-repression of E ...... he adenovirus E1A oncoprotein.
@ast
Retinoblastoma-repression of E ...... he adenovirus E1A oncoprotein.
@en
P2860
P356
P1476
Retinoblastoma-repression of E ...... he adenovirus E1A oncoprotein.
@en
P2093
P Raychaudhuri
P2860
P304
P356
10.1093/NAR/20.22.5947
P407
P577
1992-11-01T00:00:00Z