TNF-α- and tumor-induced skeletal muscle atrophy involves sphingolipid metabolism. - Wikidata - wikimedia - TriplyDB

TNF-α- and tumor-induced skeletal muscle atrophy involves sphingolipid metabolism.

TNF-α- and tumor-induced skeletal muscle atrophy involves sphingolipid metabolism.

http://www.wikidata.org/entity/Q35935087

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Loss of muscle mass: current developments in cachexia and sarcopenia focused on biomarkers and treatment Molecular Mechanisms of Obesity-Induced Osteoporosis and Muscle Atrophy Lipid modulation of skeletal muscle mass and function Mechanisms of metabolic dysfunction in cancer-associated cachexia The effects of obesity on skeletal muscle regeneration Neutral sphingomyelinase-3 mediates TNF-stimulated oxidant activity in skeletal muscle Muscle wasting: an overview of recent developments in basic research.The Sick and the Weak: Neuropathies/Myopathies in the Critically Ill.SREBP-1 transcription factors regulate skeletal muscle cell size by controlling protein synthesis through myogenic regulatory factors.The ChIP-seq-defined networks of Bcl-3 gene binding support its required role in skeletal muscle atrophy.Sphingomyelinase promotes oxidant production and skeletal muscle contractile dysfunction through activation of NADPH oxidase.Myostatin Activates the Ubiquitin-Proteasome and Autophagy-Lysosome Systems Contributing to Muscle Wasting in Chronic Kidney Disease Diminished anabolic signaling response to insulin induced by intramuscular lipid accumulation is associated with inflammation in aging but not obesity Phospholipase D regulates the size of skeletal muscle cells through the activation of mTOR signaling.Mitofusin-2 prevents skeletal muscle wasting in cancer cachexia.Delayed bone regeneration is linked to chronic inflammation in murine muscular dystrophy.Omega-3 fatty acid EPA improves regenerative capacity of mouse skeletal muscle cells exposed to saturated fat and inflammation.Skeletal muscle mitochondrial dysfunction during chronic obstructive pulmonary disease: central actor and therapeutic target.Pathogenic role of anti-signal recognition protein and anti-3-Hydroxy-3-methylglutaryl-CoA reductase antibodies in necrotizing myopathies: Myofiber atrophy and impairment of muscle regeneration in necrotizing autoimmune myopathies.Nutraceutical properties of chestnut flours: beneficial effects on skeletal muscle atrophy.TNF- α and IFN-s-dependent muscle decay is linked to NF-κB- and STAT-1α-stimulated Atrogin1 and MuRF1 genes in C2C12 myotubes.Myriocin prevents muscle ceramide accumulation but not muscle fiber atrophy during short-term mechanical unloading.A novel in vitro model of sarcopenia using BubR1 hypomorphic C2C12 myoblasts.Syngeneic B16F10 Melanoma Causes Cachexia and Impaired Skeletal Muscle Strength and Locomotor Activity in Mice.Quercetin enhances the antitumor effect of trichostatin A and suppresses muscle wasting in tumor-bearing mice.Food Shortage Causes Differential Effects on Body Composition and Tissue-Specific Gene Expression in Salmon Modified for Increased Growth Hormone Production.Fibroblast growth factor 19 regulates skeletal muscle mass and ameliorates muscle wasting in mice.Plasma Sphingolipids are Associated With Gait Parameters in the Mayo Clinic Study of Aging.Proteolysis inhibition by hibernating bear serum leads to increased protein content in human muscle cells.Glutaminase 1 regulates the release of extracellular vesicles during neuroinflammation through key metabolic intermediate alpha-ketoglutarate.Biomarkers associated with low, moderate, and high vastus lateralis muscle hypertrophy following 12 weeks of resistance training.PGC-1α4 gene expression is suppressed by the IL-6-MEK-ERK 1/2 MAPK signalling axis and altered by resistance exercise, obesity and muscle injury.Physiological Differences Between Low Versus High Skeletal Muscle Hypertrophic Responders to Resistance Exercise Training: Current Perspectives and Future Research Directions

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TNF-α- and tumor-induced skeletal muscle atrophy involves sphingolipid metabolism.

http://www.wikidata.org/entity/Q35935087

description

2012 nî lūn-bûn

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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2012年论文

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2012年论文

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name

TNF-α- and tumor-induced skeletal muscle atrophy involves sphingolipid metabolism.

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TNF-α- and tumor-induced skeletal muscle atrophy involves sphingolipid metabolism.

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type

label

TNF-α- and tumor-induced skeletal muscle atrophy involves sphingolipid metabolism.

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TNF-α- and tumor-induced skeletal muscle atrophy involves sphingolipid metabolism.

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prefLabel

TNF-α- and tumor-induced skeletal muscle atrophy involves sphingolipid metabolism.

@ast

TNF-α- and tumor-induced skeletal muscle atrophy involves sphingolipid metabolism.

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Skeletal Muscle

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TNF-α- and tumor-induced skeletal muscle atrophy involves sphingolipid metabolism.

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Alessandra Zufferli

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David Cheillan

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Fabio Naro

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Filippo Serra

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Georges Némoz

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Joffrey De Larichaudy

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Kevin Dessalle

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Marine Desgeorges

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Monique Piraud

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The initiation factor eIF3-f is a major target for atrogin1/MAFbx function in skeletal muscle atrophy

LC3, a mammalian homologue of yeast Apg8p, is localized in autophagosome membranes after processing

Tumor necrosis factor-alpha and muscle wasting: a cellular perspective

A RAPID METHOD OF TOTAL LIPID EXTRACTION AND PURIFICATION

The sphingosine kinase 1/sphingosine-1-phosphate pathway mediates COX-2 induction and PGE2 production in response to TNF-alpha

Akt/mTOR pathway is a crucial regulator of skeletal muscle hypertrophy and can prevent muscle atrophy in vivo

Ceramide-mediated macroautophagy involves inhibition of protein kinase B and up-regulation of beclin 1

Aging up-regulates expression of inflammatory mediators in mouse adipose tissue

Serine palmitoyltransferase is the primary target of a sphingosine-like immunosuppressant, ISP-1/myriocin

Identification of ubiquitin ligases required for skeletal muscle atrophy

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10.1186/2044-5040-2-2

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1

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Andrea M. Isidori

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2012-01-18T00:00:00Z

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221759527

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