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Mitochondria in the diabetic heartHow exercise may amend metabolic disturbances in diabetic cardiomyopathyDiabetic cardiomyopathy: pathophysiology and clinical featuresCardiomyocyte contractile status is associated with differences in fibronectin and integrin interactions.Increased myocardial SERCA expression in early type 2 diabetes mellitus is insulin dependent: In vivo and in vitro data.Fibronectin increases the force production of mouse papillary muscles via α5β1 integrinAblation of phospholamban and sarcolipin results in cardiac hypertrophy and decreased cardiac contractilityGSH or palmitate preserves mitochondrial energetic/redox balance, preventing mechanical dysfunction in metabolically challenged myocytes/hearts from type 2 diabetic miceMalondialdehyde and 4-hydroxynonenal adducts are not formed on cardiac ryanodine receptor (RyR2) and sarco(endo)plasmic reticulum Ca2+-ATPase (SERCA2) in diabetesMechanisms of impaired calcium handling underlying subclinical diastolic dysfunction in diabetes.Myocardial dysfunction occurs prior to changes in ventricular geometry in mice with chronic kidney disease (CKD)Determinants of frequency-dependent contraction and relaxation of mammalian myocardium.Conditional increase in SERCA2a protein is able to reverse contractile dysfunction and abnormal calcium flux in established diabetic cardiomyopathy.High- and moderate-intensity training normalizes ventricular function and mechanoenergetics in mice with diet-induced obesity.Cysteine-674 oxidation and degradation of sarcoplasmic reticulum Ca(2+) ATPase in diabetic pig aorta.Exercise training during diabetes attenuates cardiac ryanodine receptor dysregulation.The SR-mitochondria interaction: a new player in cardiac pathophysiology.FoxO1 is crucial for sustaining cardiomyocyte metabolism and cell survival.Impairment of calcium ATPases by high glucose and potential pharmacological protection.Matrix revisited: mechanisms linking energy substrate metabolism to the function of the heart.Endothelial cell-cardiomyocyte crosstalk in diabetic cardiomyopathy.Inflammation and metabolic cardiomyopathy.The autophagic response to nutrient deprivation in the hl-1 cardiac myocyte is modulated by Bcl-2 and sarco/endoplasmic reticulum calcium stores.Dysregulation of microRNAs and renin-angiotensin system in high salt diet-induced cardiac dysfunction in uninephrectomized rats.Exercise training prevents the development of cardiac dysfunction in the low-dose streptozotocin diabetic rats fed a high-fat diet.Exendin-4 attenuates high glucose-induced cardiomyocyte apoptosis via inhibition of endoplasmic reticulum stress and activation of SERCA2a.Endothelial dysfunction in Type 2 diabetes correlates with deregulated expression of the tail-anchored membrane protein SLMAP.Metoprolol improves cardiac function and modulates cardiac metabolism in the streptozotocin-diabetic rat.New Antihyperglycemic Drugs and Heart Failure: Synopsis of Basic and Clinical Data.Hyperglycemia-induced cardiac contractile dysfunction in the diabetic heart.Proarrhythmic Remodeling of Calcium Homeostasis in Cardiac Disease; Implications for Diabetes and Obesity
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description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年学术文章
@wuu
2004年学术文章
@zh-cn
2004年学术文章
@zh-hans
2004年学术文章
@zh-my
2004年学术文章
@zh-sg
2004年學術文章
@yue
2004年學術文章
@zh
2004年學術文章
@zh-hant
name
Altered cardiac calcium handling in diabetes.
@ast
Altered cardiac calcium handling in diabetes.
@en
type
label
Altered cardiac calcium handling in diabetes.
@ast
Altered cardiac calcium handling in diabetes.
@en
prefLabel
Altered cardiac calcium handling in diabetes.
@ast
Altered cardiac calcium handling in diabetes.
@en
P1476
Altered cardiac calcium handling in diabetes.
@en
P2093
Darrell D Belke
Wolfgang H Dillmann
P2860
P2888
P304
P356
10.1007/S11906-004-0035-3
P577
2004-12-01T00:00:00Z