Genome-wide alterations in gene methylation by the BRAF V600E mutation in papillary thyroid cancer cells. - Wikidata - wikimedia - TriplyDB

Genome-wide alterations in gene methylation by the BRAF V600E mutation in papillary thyroid cancer cells.

Genome-wide alterations in gene methylation by the BRAF V600E mutation in papillary thyroid cancer cells.

http://www.wikidata.org/entity/Q35942427

about

Genetic Testing in Differentiated Thyroid Carcinoma: Indications and Clinical Implications Integrated data analysis reveals potential drivers and pathways disrupted by DNA methylation in papillary thyroid carcinomas.c-Myc is Required for BRAFV600E-Induced Epigenetic Silencing by H3K27me3 in Tumorigenesis.MicroRNA-mediated networks underlie immune response regulation in papillary thyroid carcinoma.Molecular pathogenesis and mechanisms of thyroid cancer Methylation of DACT2 promotes papillary thyroid cancer metastasis by activating Wnt signaling RUNX3 site-specific hypermethylation predicts papillary thyroid cancer recurrence.The BRAF(V600E) causes widespread alterations in gene methylation in the genome of melanoma cells.REC8 is a novel tumor suppressor gene epigenetically robustly targeted by the PI3K pathway in thyroid cancer.Recent progress of genome study for anaplastic thyroid cancer.Loss of ARF sensitizes transgenic BRAFV600E mice to UV-induced melanoma via suppression of XPC.Association between BRAF V600E mutation and mortality in patients with papillary thyroid cancer.Genome-wide methylation patterns in papillary thyroid cancer are distinct based on histological subtype and tumor genotype.Analysis of BRAF(V600E) mutation and DNA methylation improves the diagnostics of thyroid fine needle aspiration biopsies.Epigenetically upregulated WIPF1 plays a major role in BRAF V600E-promoted papillary thyroid cancer aggressiveness.Molecular Aberrance in Papillary Thyroid Microcarcinoma Bearing High Aggressiveness: Identifying a "Tibetan Mastiff Dog" From Puppies.Minireview: Role of genetic changes of faciogenital dysplasia protein 1 in human disease.Epigenetic Alterations and Canonical Pathway Disruption in Papillary Thyroid Cancer: A Genome-wide Methylation Analysis.Expression of YY1 in Differentiated Thyroid Cancer.Meta-analysis of promoter methylation in eight tumor-suppressor genes and its association with the risk of thyroid cancer.Mutational analysis of the GNA11, MMP27, FGD1, TRRAP and GRM3 genes in thyroid cancer.Oncogenic BRAF mutation induces DNA methylation changes in a murine model for human serrated colorectal neoplasia.Regulation of mutant TERT by BRAF V600E/MAP kinase pathway through FOS/GABP in human cancer.DNA methylation analysis for the diagnosis of thyroid nodules - a pilot study with reference to BRAF(V) (600E) mutation and cytopathology results.DNA methylation profiling of well-differentiated thyroid cancer uncovers markers of recurrence free survival.Relationship of body mass index with BRAF (V600E) mutation in papillary thyroid cancer.

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Genome-wide alterations in gene methylation by the BRAF V600E mutation in papillary thyroid cancer cells.

http://www.wikidata.org/entity/Q35942427

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Dingxie Liu

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Mingzhao Xing

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Peng Hou

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P2860

CDC42 and FGD1 cause distinct signaling and transforming activities.

CpG Island microarray probe sequences derived from a physical library are representative of CpG Islands annotated on the human genome

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Mutations of the BRAF gene in human cancer

Association of aberrant methylation of tumor suppressor genes with tumor aggressiveness and BRAF mutation in papillary thyroid cancer

The epigenomics of cancer

CpG island methylator phenotype underlies sporadic microsatellite instability and is tightly associated with BRAF mutation in colorectal cancer

A National Cancer Data Base report on 53,856 cases of thyroid carcinoma treated in the U.S., 1985-1995 [see commetns]

Oncogenic BRAFV600E induces expression of neuronal differentiation marker MAP2 in melanoma cells by promoter demethylation and down-regulation of transcription repressor HES1.

High-throughput methylation profiling by MCA coupled to CpG island microarray

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