Nerve growth factor enhances the excitability of rat sensory neurons through activation of the atypical protein kinase C isoform, PKMζ.
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Neurotrophins regulate cholinergic synaptic transmission in cultured rat sympathetic neurons through a p75-dependent mechanismMemory maintenance by PKMζ--an evolutionary perspectiveSphingosine 1-phosphate enhances the excitability of rat sensory neurons through activation of sphingosine 1-phosphate receptors 1 and/or 3Long-term potentiation decay and memory loss are mediated by AMPAR endocytosis.Sensory nerve terminal mitochondrial dysfunction induces hyperexcitability in airway nociceptors via protein kinase CN-type calcium current, Cav2.2, is enhanced in small-diameter sensory neurons isolated from Nf1+/- mice.Epac activation sensitizes rat sensory neurons through activation of Ras.Spinal atypical protein kinase C activity is necessary to stabilize inactivity-induced phrenic motor facilitation.Aromatase inhibitors augment nociceptive behaviors in rats and enhance the excitability of sensory neurons.Nerve growth factor/p75 neurotrophin receptor-mediated sensitization of rat sensory neurons depends on membrane cholesterol.Angiotensin II receptor type 2 activation is required for cutaneous sensory hyperinnervation and hypersensitivity in a rat hind paw model of inflammatory painDeconstructing the neuropathic pain phenotype to reveal neural mechanisms.Nerve growth factor and the physiology of pain: lessons from congenital insensitivity to pain with anhidrosis.ZIPping to pain relief: the role (or not) of PKMζ in chronic pain.Matching biochemical and functional efficacies confirm ZIP as a potent competitive inhibitor of PKMζ in neurons.BDNF regulates atypical PKC at spinal synapses to initiate and maintain a centralized chronic pain state.The p75NTR signaling cascade mediates mechanical hyperalgesia induced by nerve growth factor injected into the rat hind paw.Nociceptors as chronic drivers of pain and hyperreflexia after spinal cord injury: an adaptive-maladaptive hyperfunctional state hypothesis.The TrkA receptor mediates experimental thermal hyperalgesia produced by nerve growth factor: Modulation by the p75 neurotrophin receptor.Peripheral Synthesis of an Atypical Protein Kinase C Mediates the Enhancement of Excitability and the Development of Mechanical Hyperalgesia Produced by Nerve Growth Factor.Role of Protein Kinase C in Bipolar Disorder: A Review of the Current Literature.
P2860
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P2860
Nerve growth factor enhances the excitability of rat sensory neurons through activation of the atypical protein kinase C isoform, PKMζ.
description
2011 nî lūn-bûn
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2011年の論文
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2011年学术文章
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2011年学术文章
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2011年学术文章
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2011年学术文章
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2011年学术文章
@zh-sg
2011年學術文章
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2011年學術文章
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2011年學術文章
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name
Nerve growth factor enhances t ...... rotein kinase C isoform, PKMζ.
@ast
Nerve growth factor enhances t ...... rotein kinase C isoform, PKMζ.
@en
type
label
Nerve growth factor enhances t ...... rotein kinase C isoform, PKMζ.
@ast
Nerve growth factor enhances t ...... rotein kinase C isoform, PKMζ.
@en
prefLabel
Nerve growth factor enhances t ...... rotein kinase C isoform, PKMζ.
@ast
Nerve growth factor enhances t ...... rotein kinase C isoform, PKMζ.
@en
P2093
P2860
P356
P1476
Nerve growth factor enhances t ...... rotein kinase C isoform, PKMζ.
@en
P2093
P2860
P304
P356
10.1152/JN.00030.2011
P407
P577
2011-10-05T00:00:00Z