Early growth response-1 transcription factor is essential for ethanol-induced fatty liver injury in mice.
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Identifying Novel Targets for Treatment of Liver Fibrosis: What Can We Learn from Injured Tissues which Heal Without a Scar?Kupffer cells in the liverEthanol-induced oxidant stress modulates hepatic autophagy and proteasome activityCellular and molecular mechanisms in the pathogenesis of liver fibrosis: An updateAlcoholic liver disease: pathogenesis and new therapeutic targets.Early growth response-1 transcription factor promotes hepatic fibrosis and steatosis in long-term ethanol-fed Long-Evans ratsLive in vivo imaging of Egr-1 promoter activity during neonatal development, liver regeneration and wound healingImpaired methylation as a novel mechanism for proteasome suppression in liver cellsHepatic fibrosis is enhanced and accompanied by robust oval cell activation after chronic carbon tetrachloride administration to Egr-1-deficient miceEarly growth response-1 attenuates liver injury and promotes hepatoprotection after carbon tetrachloride exposure in miceToll-like receptors in the pathogenesis of alcoholic liver disease.Tributyrin supplementation protects mice from acute ethanol-induced gut injury.Data mining reveals a network of early-response genes as a consensus signature of drug-induced in vitro and in vivo toxicityBile acids induce inflammatory genes in hepatocytes: a novel mechanism of inflammation during obstructive cholestasis.Alternative complement pathway component Factor D contributes to efficient clearance of tissue debris following acute CCl₄-induced injury.Adenosine 2A receptor antagonist prevented and reversed liver fibrosis in a mouse model of ethanol-exacerbated liver fibrosisRedox signaling and the innate immune system in alcoholic liver disease.Early growth response (EGR)-1 is required for timely cell-cycle entry and progression in hepatocytes after acute carbon tetrachloride exposure in mice.Early growth response 1 (Egr1) regulates cholesterol biosynthetic gene expression.Measurement of serum, liver, and brain cytokine induction, thiamine levels, and hepatopathology in rats exposed to a 4-day alcohol binge protocol.Ethanol-induced oxidative stress via the CYP2E1 pathway disrupts adiponectin secretion from adipocytesDifferential contributions of C3, C5, and decay-accelerating factor to ethanol-induced fatty liver in mice.Early growth response factor-1 limits biliary fibrosis in a model of xenobiotic-induced cholestasis in mice.Early growth response-1 contributes to steatosis development after acute ethanol administration.Chronic ethanol feeding increases activation of NADPH oxidase by lipopolysaccharide in rat Kupffer cells: role of increased reactive oxygen in LPS-stimulated ERK1/2 activation and TNF-alpha production.Adiponectin normalizes LPS-stimulated TNF-alpha production by rat Kupffer cells after chronic ethanol feeding.New Approaches for Studying Alcoholic Liver DiseaseEthanol-induced liver injury: potential roles for egr-1.Cellular steatosis in ethanol oxidizing-HepG2 cells is partially controlled by the transcription factor, early growth response-1.The pathogenesis of ethanol versus methionine and choline deficient diet-induced liver injury.Regulation of macrophage activation in alcoholic liver disease.Alcohol-induced steatosis in liver cellsSignaling mechanisms in alcoholic liver injury: role of transcription factors, kinases and heat shock proteinsNew concepts in the pathogenesis of alcoholic liver disease.Signalling pathways in alcohol-induced liver inflammation.Elk-3 Contributes to the Progression of Liver Fibrosis by Regulating the Epithelial-Mesenchymal Transition.Synergistic interaction between C5a and NOD2 signaling in the regulation of chemokine expression in RAW 264.7 macrophages.Mesothelin/mucin 16 signaling in activated portal fibroblasts regulates cholestatic liver fibrosis.Alcohol, liver, systemic inflammation and skin: a focus on patients with psoriasis.Genome-wide investigation of in vivo EGR-1 binding sites in monocytic differentiation.
P2860
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P2860
Early growth response-1 transcription factor is essential for ethanol-induced fatty liver injury in mice.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
2005年论文
@zh
2005年论文
@zh-cn
name
Early growth response-1 transc ...... ed fatty liver injury in mice.
@ast
Early growth response-1 transc ...... ed fatty liver injury in mice.
@en
type
label
Early growth response-1 transc ...... ed fatty liver injury in mice.
@ast
Early growth response-1 transc ...... ed fatty liver injury in mice.
@en
prefLabel
Early growth response-1 transc ...... ed fatty liver injury in mice.
@ast
Early growth response-1 transc ...... ed fatty liver injury in mice.
@en
P2093
P2860
P1433
P1476
Early growth response-1 transc ...... ced fatty liver injury in mice
@en
P2093
Carrie A Millward
Colleen M Croniger
Laura E Nagy
Megan R McMullen
Qifang Wang
P2860
P304
P356
10.1053/J.GASTRO.2005.02.065
P407
P577
2005-06-01T00:00:00Z