Prostacyclin receptor-mediated ATP release from erythrocytes requires the voltage-dependent anion channel.
about
Erythrocyte-derived ATP and perfusion distribution: role of intracellular and intercellular communicationBlood cells: an historical account of the roles of purinergic signallingThe pannexins: past and presentExported Epoxide Hydrolases Modulate Erythrocyte Vasoactive Lipids during Plasmodium falciparum InfectionPlasma membrane coenzyme Q: evidence for a role in autism.Mitochondrial biogenesis dysfunction and metabolic dysfunction from a novel mitochondrial tRNAMet 4467 C>A mutation in a Han Chinese family with maternally inherited hypertensionBacterial RTX toxins allow acute ATP release from human erythrocytes directly through the toxin pore.Regulation of extracellular ATP in human erythrocytes infected with Plasmodium falciparumDynamic Regulation of Cell Volume and Extracellular ATP of Human Erythrocytes.Role of erythrocyte-released ATP in the regulation of microvascular oxygen supply in skeletal muscle.Red Blood Cell Homeostasis: Pharmacological Interventions to Explore Biochemical, Morphological and Mechanical Properties.Phosphodiesterase 5 inhibitors augment UT-15C-stimulated ATP release from erythrocytes of humans with pulmonary arterial hypertensionTSPO ligands stimulate ZnPPIX transport and ROS accumulation leading to the inhibition of P. falciparum growth in human blood.CR1-mediated ATP release by human red blood cells promotes CR1 clustering and modulates the immune transfer processSynergistic effects of C-peptide and insulin on low O2-induced ATP release from human erythrocytes.Regulation of blood flow distribution in skeletal muscle: role of erythrocyte-released ATP.Tissue oxygen demand in regulation of the behavior of the cells in the vasculature.The red blood cell: a new key player in cardiovascular homoeostasis? Focus on the nitric oxide pathway.Purinergic control of red blood cell metabolism: novel strategies to improve red cell storage quality.Mitochondrial tRNA mutation with high-salt stimulation on cardiac damage: underlying mechanism associated with change of Bax and VDAC.New insights on the regulation of the adenine nucleotide pool of erythrocytes in mouse modelsHemolysis is a primary ATP-release mechanism in human erythrocytes.Effects of Hypoxia on Erythrocyte Membrane Properties-Implications for Intravascular Hemolysis and Purinergic Control of Blood Flow.Erythrocyte's aging in microgravity highlights how environmental stimuli shape metabolism and morphology.Human erythrocytes release ATP by a novel pathway involving VDAC oligomerization independent of pannexin-1
P2860
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P2860
Prostacyclin receptor-mediated ATP release from erythrocytes requires the voltage-dependent anion channel.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Prostacyclin receptor-mediated ...... ltage-dependent anion channel.
@ast
Prostacyclin receptor-mediated ...... ltage-dependent anion channel.
@en
type
label
Prostacyclin receptor-mediated ...... ltage-dependent anion channel.
@ast
Prostacyclin receptor-mediated ...... ltage-dependent anion channel.
@en
prefLabel
Prostacyclin receptor-mediated ...... ltage-dependent anion channel.
@ast
Prostacyclin receptor-mediated ...... ltage-dependent anion channel.
@en
P2093
P2860
P1476
Prostacyclin receptor-mediated ...... ltage-dependent anion channel.
@en
P2093
Alan H Stephenson
Elizabeth A Bowles
Jennifer P Richards
Katie L Davis
Kristine A Dietrich
Mary L Ellsworth
Medina Krantic
Meera Sridharan
Randy S Sprague
P2860
P304
P356
10.1152/AJPHEART.00998.2011
P577
2011-12-09T00:00:00Z