Inhibition of high-mobility group box 1 protein (HMGB1) enhances bacterial clearance and protects against Pseudomonas Aeruginosa pneumonia in cystic fibrosis.
about
Innate Immunity and Biomaterials at the Nexus: Friends or FoesEmerging role of HMGB1 in fibrotic diseasesAsbestos and erionite prime and activate the NLRP3 inflammasome that stimulates autocrine cytokine release in human mesothelial cellsEmerging role of HMGB1 in lung diseases: friend or foe.2-O, 3-O-desulfated heparin inhibits neutrophil elastase-induced HMGB-1 secretion and airway inflammation.Testosterone suppresses uropathogenic Escherichia coli invasion and colonization within prostate cells and inhibits inflammatory responses through JAK/STAT-1 signaling pathway.HMGB1 in health and disease.High-mobility group box 1: a novel target for treatment of Pseudomonas aeruginosa keratitisThe compromise of macrophage functions by hyperoxia is attenuated by ethacrynic acid via inhibition of NF-κB-mediated release of high-mobility group box-1.Elevated HMGB1-related interleukin-6 is associated with dynamic responses of monocytes in patients with active pulmonary tuberculosis.Absence of the cystic fibrosis transmembrane regulator (Cftr) from myeloid-derived cells slows resolution of inflammation and infection.HMGB1 promotes neutrophil extracellular trap formation through interactions with Toll-like receptor 4High Mobility Group Box-1 mediates hyperoxia-induced impairment of Pseudomonas aeruginosa clearance and inflammatory lung injury in mice.Hyperoxemia as a risk factor for ventilator-associated pneumonia.Helicobacter pylori Activates HMGB1 Expression and Recruits RAGE into Lipid Rafts to Promote Inflammation in Gastric Epithelial Cells.Glycyrrhizin Reduces HMGB1 and Bacterial Load in Pseudomonas aeruginosa KeratitisJAK/STAT1 signaling promotes HMGB1 hyperacetylation and nuclear translocation.Transplantation of umbilical cord mesenchymal stem cells alleviates pneumonitis of MRL/lpr mice.Targeting HMGB1 in the treatment of sepsisAutophagy and transporter-based multi-drug resistance.Clinically Promising Biomarkers in Cystic Fibrosis Pulmonary Exacerbations.Regulation of Posttranslational Modifications of HMGB1 During Immune ResponsesAdvanced Role of Neutrophils in Common Respiratory Diseases2-O, 3-O Desulfated Heparin Blocks High Mobility Group Box 1 Release by Inhibition of p300 Acetyltransferase Activity.Ascorbic Acid Attenuates Hyperoxia-Compromised Host Defense against Pulmonary Bacterial Infection.The α7 nicotinic acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function.Frontline Science: HMGB1 induces neutrophil dysfunction in experimental sepsis and in patients who survive septic shock.Unravelling the networks dictating host resistance versus tolerance during pulmonary infections.Aged Human Stored Red Blood Cell Supernatant Inhibits Macrophage Phagocytosis in an HMGB1 Dependent Manner After Trauma in a Murine Model.High-mobility group box 1 and the receptor for advanced glycation end products contribute to lung injury during Staphylococcus aureus pneumonia.Cyclic stretch induced IL-33 production through HMGB1/TLR-4 signaling pathway in murine respiratory epithelial cells.Reduced HMGB 1-Mediated Pathway and Oxidative Stress in Resveratrol-Treated Diabetic Mice: A Possible Mechanism of Cardioprotection of Resveratrol in Diabetes MellitusHMGB1 induces lung fibroblast to myofibroblast differentiation through NF‑κB‑mediated TGF‑β1 release."With a little help from my friends": efferocytosis as an antimicrobial mechanism.Hyperoxia, HMGB1, and ventilator-associated pneumonia: reducing risk by practicing what we teach.Targeting the "Rising DAMP" during a Francisella tularensis InfectionHypoxia promotes danger-mediated inflammation via receptor for advanced glycation end products in cystic fibrosis.Alarmins in Frozen Shoulder: A Molecular Association Between Inflammation and Pain.High-Mobility Group Box 1 Upregulates MUC5AC and MUC5B Expression in Primary Airway Epithelial Cells.Carnosic Acid Alleviates BDL-Induced Liver Fibrosis through miR-29b-3p-Mediated Inhibition of the High-Mobility Group Box 1/Toll-Like Receptor 4 Signaling Pathway in Rats.
P2860
Q26801604-60269168-73CF-4CE1-960C-54479DC1200BQ26828596-23242F25-24EB-4984-8166-076B8B87849CQ28387699-50E33A7D-EC59-4BD9-8FCD-F220EFDBFF7EQ33684145-1D1D94F0-33BA-4BD6-A609-07999AFC9140Q33799602-5AD8EA22-964A-475D-9F05-13B3A47E5BC5Q33860198-3CCF4196-E604-4037-89D5-DA05B2D86A59Q34622835-B61295BF-3D56-41F7-BCD5-FD22F55F2E1AQ35070914-4B52E02A-3755-45E9-966E-ADFDAF96D656Q35207661-593A0BED-CEF6-4809-945E-62BDAB6B16AFQ35406168-E458D75C-AC7D-4DBB-9FC0-5CA872BB4C1BQ36332718-3A623B2A-1906-483B-8140-0A6691B77FADQ36699968-180C2E5F-7F86-4085-8DED-F7FADC34D27AQ36703983-73DEFE36-08C3-43FD-A543-CC818FBB6764Q37031079-3ECC252F-A3CC-45E5-B43F-8689A94B8B97Q37242901-E513FC85-4966-4C33-9DDD-0D965B4C7C99Q37384368-F6AC34EB-CD56-487D-B325-B54D8CA7C9CCQ37612678-9841888B-97E3-4471-9782-811CEAE3D488Q37620545-D3AF32B4-5D68-42FE-BADC-87A56DFA0568Q37622744-B363E955-4531-4969-AAE7-4AE49A8D1CFDQ38202874-826235FD-5C2D-4515-90B9-5C86DEA33B69Q38669996-B660F341-75E9-4CE7-9170-31C0F023A012Q38682450-CC4F0A99-22E6-454F-AE8B-92806EDC1367Q38739370-8856B683-A228-4A4A-AD6C-40BDA5A9D076Q38748673-752D0CB9-EF5B-4DAA-9010-3EE56DA7909CQ38775073-0DA77E4C-37BE-45DC-8E13-A9788B849AA0Q39012184-A2C5100B-56B6-4497-B9BB-9FFF4C5070BDQ39103864-49D6E7F6-551E-496A-BCF9-3DA57F835DA6Q39123548-1CC26E3A-495B-41F5-ADE7-E45BAA1A5E28Q40589554-C965E0D4-50DF-4343-85DB-EE5B062C3EA2Q40691707-C0EF59B4-2104-46E8-B8EB-89EA1C7F5802Q41227461-A1B59DA1-CEAB-4F82-8EA0-0384011C5893Q41643658-ACC76D08-6F62-4E55-AC73-4F6CCF2FA965Q41848319-930AEA74-805D-4630-9AC8-E5188D8EF75BQ42325962-C7D287C6-1CD2-4569-A9F9-1C1C02F6CDCEQ42554510-1F41D2E8-F1FB-4ED4-9030-41F0BB7142AAQ42572961-5C379F20-A467-432E-98B1-969E71582DBBQ44909879-AC7A55BE-DC17-4628-993B-CD8B80382786Q47347447-142D9D71-82C4-46BA-BBC5-BCCB7A9FC72CQ48331221-D728E6FA-2F46-40E2-B3AC-9D3D60E5B8FDQ49540955-D28AD7FC-1CD9-4929-BFE3-2628D22CD6AD
P2860
Inhibition of high-mobility group box 1 protein (HMGB1) enhances bacterial clearance and protects against Pseudomonas Aeruginosa pneumonia in cystic fibrosis.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Inhibition of high-mobility gr ...... pneumonia in cystic fibrosis.
@ast
Inhibition of high-mobility gr ...... pneumonia in cystic fibrosis.
@en
type
label
Inhibition of high-mobility gr ...... pneumonia in cystic fibrosis.
@ast
Inhibition of high-mobility gr ...... pneumonia in cystic fibrosis.
@en
prefLabel
Inhibition of high-mobility gr ...... pneumonia in cystic fibrosis.
@ast
Inhibition of high-mobility gr ...... pneumonia in cystic fibrosis.
@en
P2093
P2860
P1433
P1476
Inhibition of high-mobility gr ...... a pneumonia in cystic fibrosis
@en
P2093
Binh D Phan
Daniel J Weiss
Edmund J Miller
Haichao Wang
JianHua Li
Kevin J Tracey
Laurie Whittaker
Lin L Mantell
Maria Entezari
P2860
P2888
P304
P356
10.2119/MOLMED.2012.00024
P407
P577
2012-05-09T00:00:00Z
P5875
P6179
1068965513