When saliva meets acid: chemical warfare at the oesophagogastric junction.
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Role of nitric oxide in the pathogenesis of Barrett's-associated carcinogenesisNitric oxide-modulating agents for gastrointestinal disordersReview article: from gastrin to gastro-oesophageal reflux disease--a century of acid suppressionModelling a population with Barrett's oesophagus from oesophageal adenocarcinoma incidence data.Nornicotine nitrosation in saliva and its relation to endogenous synthesis of N'-nitrosonornicotine in humans.Selenium, selenoenzymes, oxidative stress and risk of neoplastic progression from Barrett's esophagus: results from biomarkers and genetic variants.Systematic review: the chemoprevention of oesophageal adenocarcinoma.The frequency of histological features mimicking reflux esophagitis: a study in non-human primates.Trends in the incidence of adenocarcinoma of the oesophagus and cardia in the Netherlands 1989-2003.The frequency of lymphocytic and reflux esophagitis in non-human primates.Genetic variants of the XRCC1 gene and susceptibility to esophageal cancer: a meta-analysis.Involvement of luminal nitric oxide in the pathogenesis of the gastroesophageal reflux disease spectrum.Long-term risk of gastric cancer by subsite in operated and unoperated patients hospitalized for peptic ulcer.The role of nitric oxide in the induction of caudal-type homeobox 2 through epidermal growth factor receptor in the development of Barrett's esophagus.Inducible nitric oxide synthase (iNOS) and nitric oxide (NO) are important mediators of reflux-induced cell signalling in esophageal cells.Combined treatment with green tea catechins and sodium nitrite selectively promotes rat forestomach carcinogenesis after initiation with N-methyl-N'- nitro-N-nitrosoguanidine.Enhancement of esophageal carcinogenesis in acid reflux model rats treated with ascorbic acid and sodium nitrite in combination with or without initiation.Gastric carditis: Is it a histological response to high concentrations of luminal nitric oxide?Dietary Nitrate: Ward et al. Respond.Exogenous luminal nitric oxide exacerbates esophagus tissue damage in a reflux esophagitis model of rats.Effects of pH on nitrite-induced formation of reactive nitrogen oxide species and their scavenging by phenolic antioxidants in human oral cavity.Saliva transport to the distal esophagus.Dual-function of thiocyanate on nitrite-induced formation of reactive nitrogen oxide species in human oral cavity: inhibition under neutral and enhancement under acidic conditions.Gastroesophageal reflux patients' defective antioxidative capacity in the proximal esophageal mucosa before antireflux surgery and also after 4-year follow-up.Estrogen Enhances Esophageal Barrier Function by Potentiating Occludin Expression.Oral nitrate reductase activity and erosive gastro-esophageal reflux disease: a nitrate hypothesis for GERD pathogenesis.Reactive nitrogen oxide species induce dilatation of the intercellular space of rat esophagus.Esophageal Cancer: Priorities for Prevention
P2860
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P2860
When saliva meets acid: chemical warfare at the oesophagogastric junction.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
2005年论文
@zh
2005年论文
@zh-cn
name
When saliva meets acid: chemical warfare at the oesophagogastric junction.
@ast
When saliva meets acid: chemical warfare at the oesophagogastric junction.
@en
type
label
When saliva meets acid: chemical warfare at the oesophagogastric junction.
@ast
When saliva meets acid: chemical warfare at the oesophagogastric junction.
@en
prefLabel
When saliva meets acid: chemical warfare at the oesophagogastric junction.
@ast
When saliva meets acid: chemical warfare at the oesophagogastric junction.
@en
P2860
P356
P1433
P1476
When saliva meets acid: chemical warfare at the oesophagogastric junction.
@en
P2093
K E L McColl
P2860
P356
10.1136/GUT.2004.047126
P407
P577
2005-01-01T00:00:00Z