Resistance to anticancer drugs in NIH3T3 cells transfected with c-myc and/or c-H-ras genes
about
Mechanisms of resistance to alkylating agentsIntegrity of the N-terminal transcription domain of p53 is required for mutant p53 interference with drug-induced apoptosisThe multixenobiotic resistance mechanism in aquatic organisms.Tumour cells surviving in vivo cisplatin chemotherapy display elevated c-myc expression.Are RAS mutations predictive markers of resistance to standard chemotherapy?Glutamine deprivation plus BPTES alters etoposide- and cisplatin-induced apoptosis in triple negative breast cancer cellsGlutathione depletion induced by c-Myc downregulation triggers apoptosis on treatment with alkylating agents.Oncogenic H-Ras enhances DNA repair through the Ras/phosphatidylinositol 3-kinase/Rac1 pathway in NIH3T3 cells. Evidence for association with reactive oxygen species.MycN sensitizes neuroblastoma cells for drug-triggered apoptosis.MycN sensitizes neuroblastoma cells for drug-induced apoptosis.Effects of an inducible anti-sense c-myc gene transfer in a drug-resistant human small-cell-lung-carcinoma cell line.Establishment, characterization and drug sensitivity of four new human soft tissue sarcoma cell lines.Hypersensitivity of NIH3T3 cells transformed by H-ras gene to DNA-topoisomerase-I inhibitors.Lack of elevated drug efflux in adriamycin-resistant immunoblastic B lymphoma cells with mdr1 overexpression.Enhancement of sensitivity of urinary bladder tumor cells to cisplatin by c-myc antisense oligonucleotide.The erbB 2 oncogene and chemotherapy: a mini-review.Induction of multidrug resistance (MDR) by transfection of MCF-10A cell line with c-Ha-ras and c-erbB-2 oncogenes.Topographic genotyping of colorectal carcinoma: from a molecular carcinogenesis model to clinical relevance.Transformation of mouse fibroblasts with the oncogenes H-ras OR trk is associated with pronounced changes in drug sensitivity and metabolism.Inauhzin(c) inactivates c-Myc independently of p53.Expression of resistance factors (P-glycoprotein, glutathione S-transferase-pi, and topoisomerase II) and their interrelationship to proto-oncogene products in renal cell carcinomas.
P2860
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P2860
Resistance to anticancer drugs in NIH3T3 cells transfected with c-myc and/or c-H-ras genes
description
1991 nî lūn-bûn
@nan
1991年の論文
@ja
1991年論文
@yue
1991年論文
@zh-hant
1991年論文
@zh-hk
1991年論文
@zh-mo
1991年論文
@zh-tw
1991年论文
@wuu
1991年论文
@zh
1991年论文
@zh-cn
name
Resistance to anticancer drugs ...... ith c-myc and/or c-H-ras genes
@ast
Resistance to anticancer drugs ...... ith c-myc and/or c-H-ras genes
@en
type
label
Resistance to anticancer drugs ...... ith c-myc and/or c-H-ras genes
@ast
Resistance to anticancer drugs ...... ith c-myc and/or c-H-ras genes
@en
prefLabel
Resistance to anticancer drugs ...... ith c-myc and/or c-H-ras genes
@ast
Resistance to anticancer drugs ...... ith c-myc and/or c-H-ras genes
@en
P2093
P2860
P356
P1476
Resistance to anticancer drugs ...... ith c-myc and/or c-H-ras genes
@en
P2093
Nakagawa K
Terashima Y
Tsunokawa Y
P2860
P2888
P304
P356
10.1038/BJC.1991.56
P407
P577
1991-02-01T00:00:00Z