Reversal of persistent fibrosis in aging by targeting Nox4-Nrf2 redox imbalance - Wikidata - wikimedia - TriplyDB

Reversal of persistent fibrosis in aging by targeting Nox4-Nrf2 redox imbalance

Reversal of persistent fibrosis in aging by targeting Nox4-Nrf2 redox imbalance

http://www.wikidata.org/entity/Q35983620

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Redox mechanisms in age-related lung fibrosis Evolving Insights on Metabolism, Autophagy, and Epigenetics in Liver Myofibroblasts Role of redoximiRs in fibrogenesis Reciprocal regulation of TGF-β and reactive oxygen species: A perverse cycle for fibrosis Redox-fibrosis: Impact of TGFβ1 on ROS generators, mediators and functional consequences The Role of Mitochondrial DNA in Mediating Alveolar Epithelial Cell Apoptosis and Pulmonary Fibrosis Reactive Oxygen-Related Diseases: Therapeutic Targets and Emerging Clinical Indications Reactive oxygen species and fibrosis: further evidence of a significant liaison Nuclear Factor-Erythroid-2-Related Factor 2 in Aging and Lung Fibrosis Time- and compartment-resolved proteome profiling of the extracellular niche in lung injury and repair The code of non-coding RNAs in lung fibrosis Differential transcriptional regulation of hypoxia-inducible factor-1α by arsenite under normoxia and hypoxia: involvement of Nrf2 Global gene profiling of aging lungs in Atp8b1 mutant mice Mitochondria in the spotlight of aging and idiopathic pulmonary fibrosis The Impacts of Cellular Senescence in Elderly Pneumonia and in Age-Related Lung Diseases That Increase the Risk of Respiratory Infections.European contribution to the study of ROS: A summary of the findings and prospects for the future from the COST action BM1203 (EU-ROS).Negative regulation of NADPH oxidase 4 by hydrogen peroxide-inducible clone 5 (Hic-5) protein Therapeutic targeting of SRC kinase in myofibroblast differentiation and pulmonary fibrosis.New therapeutic targets in idiopathic pulmonary fibrosis. Aiming to rein in runaway wound-healing responses.Transcript profile of cellular senescence-related genes in Fuchs endothelial corneal dystrophy.PINK1 deficiency impairs mitochondrial homeostasis and promotes lung fibrosis.Review: interstitial lung disease associated with systemic sclerosis and idiopathic pulmonary fibrosis: how similar and distinct?Fibrosis: ultimate and proximate causes Blue journal conference. Aging and susceptibility to lung disease.Inhibition of myocardin-related transcription factor/serum response factor signaling decreases lung fibrosis and promotes mesenchymal cell apoptosis.Plasminogen activator inhibitor 1, fibroblast apoptosis resistance, and aging-related susceptibility to lung fibrosis Upstream regulators and downstream effectors of NADPH oxidases as novel therapeutic targets for diabetic kidney disease.Targeting NADPH oxidase with a novel dual Nox1/Nox4 inhibitor attenuates renal pathology in type 1 diabetes.Total Glucosides of Danggui Buxue Tang Attenuate BLM-Induced Pulmonary Fibrosis via Regulating Oxidative Stress by Inhibiting NOX4.Alteration of Aging-Dependent MicroRNAs in Idiopathic Pulmonary Fibrosis.Disruption of microRNA-21 by TALEN leads to diminished cell transformation and increased expression of cell-environment interaction genes.Targeting of Gamma-Glutamyl-Cysteine Ligase by miR-433 Reduces Glutathione Biosynthesis and Promotes TGF-β-Dependent Fibrogenesis.NOX4 NADPH Oxidase-Dependent Mitochondrial Oxidative Stress in Aging-Associated Cardiovascular Disease miR-9-5p suppresses pro-fibrogenic transformation of fibroblasts and prevents organ fibrosis by targeting NOX4 and TGFBR2 Targeting Inhibitor of Apoptosis Proteins Protects from Bleomycin-Induced Lung Fibrosis.Mechanisms of Lung Fibrosis Resolution.The matricellular protein CCN1 enhances TGF-β1/SMAD3-dependent profibrotic signaling in fibroblasts and contributes to fibrogenic responses to lung injury.NADPH Oxidase-4 Overexpression Is Associated With Epithelial Ciliary Dysfunction in Neutrophilic Asthma Triptolide mitigates radiation-induced pulmonary fibrosis via inhibition of axis of alveolar macrophages-NOXes-ROS-myofibroblasts.An Endothelial Hsp70-TLR4 Axis Limits Nox3 Expression and Protects Against Oxidant Injury in Lungs

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Reversal of persistent fibrosis in aging by targeting Nox4-Nrf2 redox imbalance

http://www.wikidata.org/entity/Q35983620

description

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2014年の論文

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name

Reversal of persistent fibrosis in aging by targeting Nox4-Nrf2 redox imbalance

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Reversal of persistent fibrosis in aging by targeting Nox4-Nrf2 redox imbalance

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Reversal of persistent fibrosis in aging by targeting Nox4-Nrf2 redox imbalance

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Reversal of persistent fibrosis in aging by targeting Nox4-Nrf2 redox imbalance

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Reversal of persistent fibrosis in aging by targeting Nox4-Nrf2 redox imbalance

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Reversal of persistent fibrosis in aging by targeting Nox4-Nrf2 redox imbalance

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Science Translational Medicine

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Reversal of persistent fibrosis in aging by targeting Nox4-Nrf2 redox imbalance

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Ashish Kurundkar

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Deepali Kurundkar

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Eric Meldrum

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Karen Bernard

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Louise Hecker

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Naomi J Logsdon

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Thomas Hock

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Victor J Thannickal

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Yan Y Sanders

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Heart regeneration

Senescence of activated stellate cells limits liver fibrosis

The matricellular protein CCN1 induces fibroblast senescence and restricts fibrosis in cutaneous wound healing

The myofibroblast: one function, multiple origins

Host responses in tissue repair and fibrosis

Recent developments in myofibroblast biology: paradigms for connective tissue remodeling

Reactive oxygen species in cell signaling

Wound repair and regeneration

Incidence and prevalence of idiopathic pulmonary fibrosis.

Regulation of the Nrf2-Keap1 antioxidant response by the ubiquitin proteasome system: an insight into cullin-ring ubiquitin ligases.

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