Post-exposure antioxidant treatment in rats decreases airway hyperplasia and hyperreactivity due to chlorine inhalation. - Wikidata - wikimedia - TriplyDB

Post-exposure antioxidant treatment in rats decreases airway hyperplasia and hyperreactivity due to chlorine inhalation.

Post-exposure antioxidant treatment in rats decreases airway hyperplasia and hyperreactivity due to chlorine inhalation.

http://www.wikidata.org/entity/Q35987148

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Characterization of defects in ion transport and tissue development in cystic fibrosis transmembrane conductance regulator (CFTR)-knockout rats Nitrite therapy improves survival postexposure to chlorine gas Abnormal epithelial structure and chronic lung inflammation after repair of chlorine-induced airway injury Administration of nitrite after chlorine gas exposure prevents lung injury: effect of administration modality Chlorine gas exposure disrupts nitric oxide homeostasis in the pulmonary vasculature Persistent effects of chlorine inhalation on respiratory health Differential susceptibility of inbred mouse strains to chlorine-induced airway fibrosis Repair of tracheal epithelium by basal cells after chlorine-induced injury Acute Lung Injury and Persistent Small Airway Disease in a Rabbit Model of Chlorine Inhalation An Official American Thoracic Society Workshop Report: Chemical Inhalational Disasters. Biology of Lung Injury, Development of Novel Therapeutics, and Medical Preparedness.Hyaluronan mediates airway hyperresponsiveness in oxidative lung injury Chlorine inhalation-induced myocardial depression and failure.Respiratory syncytial virus infection increases chlorine-induced airway hyperresponsiveness Targeted aerosolized delivery of ascorbate in the lungs of chlorine-exposed rats.Chlorine-induced cardiopulmonary injury Chlorine induces the unfolded protein response in murine lungs and skin Upregulation of autophagy decreases chlorine-induced mitochondrial injury and lung inflammation.Nitrite therapy prevents chlorine gas toxicity in rabbits.Formation of chlorinated lipids post-chlorine gas exposure.Acute chlorine gas exposure produces transient inflammation and a progressive alteration in surfactant composition with accompanying mechanical dysfunction.Postexposure aerosolized heparin reduces lung injury in chlorine-exposed mice.Guidelines for Burn Care Under Austere Conditions: Special Etiologies: Blast, Radiation, and Chemical Injuries.The role of glucose-6-phosphate dehydrogenase in adipose tissue inflammation in obesity.Halogen Inhalation-Induced Lung Injury and Acute Respiratory Distress Syndrome.

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P2860

Post-exposure antioxidant treatment in rats decreases airway hyperplasia and hyperreactivity due to chlorine inhalation.

http://www.wikidata.org/entity/Q35987148

description

2011 nî lūn-bûn

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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2011年论文

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2011年论文

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name

Post-exposure antioxidant trea ...... ty due to chlorine inhalation.

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Post-exposure antioxidant trea ...... ty due to chlorine inhalation.

@en

type

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Post-exposure antioxidant trea ...... ty due to chlorine inhalation.

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Post-exposure antioxidant trea ...... ty due to chlorine inhalation.

@en

prefLabel

Post-exposure antioxidant trea ...... ty due to chlorine inhalation.

@ast

Post-exposure antioxidant trea ...... ty due to chlorine inhalation.

@en

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RCMB.2011-0196OC

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American Journal of Respiratory Cell and Molecular Biology

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Post-exposure antioxidant trea ...... ty due to chlorine inhalation.

@en

P2093

Andreas Bracher

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Edward M Postlethwait

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Giuseppe L Squadrito

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Larry Bowen

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Michelle V Fanucchi

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Sadis Matalon

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Solana Fernandez

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Stephen F Doran

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P2860

Chlorine-induced injury to the airways in mice

Development of a research strategy for integrated technology-based toxicological and chemical evaluation of complex mixtures of drinking water disinfection byproducts

Effects of ozone and endotoxin coexposure on rat airway epithelium: potentiation of toxicant-induced alterations.

Mitigation of chlorine gas lung injury in rats by postexposure administration of sodium nitrite

Postexposure administration of a {beta}2-agonist decreases chlorine-induced airway hyperreactivity in mice

Ascorbate and deferoxamine administration after chlorine exposure decrease mortality and lung injury in mice

Chlorine gas exposure causes systemic endothelial dysfunction by inhibiting endothelial nitric oxide synthase-dependent signaling

2007 Annual Report of the American Association of Poison Control Centers' National Poison Data System (NPDS): 25th Annual Report.

Investigating the dependence of the hypoxia-inducible factor hydroxylases (factor inhibiting HIF and prolyl hydroxylase domain 2) on ascorbate and other reducing agents.

Identification of triptolide, a natural diterpenoid compound, as an inhibitor of lung inflammation.

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599-606

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10.1165/RCMB.2011-0196OC

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5

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46

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2011-12-08T00:00:00Z

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22162906

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3359900

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