Hexokinase regulates Bax-mediated mitochondrial membrane injury following ischemic stress - Wikidata - wikimedia - TriplyDB

Hexokinase regulates Bax-mediated mitochondrial membrane injury following ischemic stress

Hexokinase regulates Bax-mediated mitochondrial membrane injury following ischemic stress

http://www.wikidata.org/entity/Q35990589

about

GSK-3β: A Bifunctional Role in Cell Death Pathways Intracellular ion channels and cancer GSK-3 as potential target for therapeutic intervention in cancer Renal adaptation during hibernation Cracking the cytotoxicity code: apoptotic induction of 10-acetylirciformonin B is mediated through ROS generation and mitochondrial dysfunction Hexokinase II integrates energy metabolism and cellular protection: Akting on mitochondria and TORCing to autophagy.Mouse model of ischemic acute kidney injury: technical notes and tricks Sirtuin-3 modulates Bak- and Bax-dependent apoptosis Nucleophosmin, a critical Bax cofactor in ischemia-induced cell death Local anesthetic bupivacaine induced ovarian and prostate cancer apoptotic cell death and underlying mechanisms in vitro.Tubular von Hippel-Lindau knockout protects against rhabdomyolysis-induced AKI.Targeting hexokinase II to mitochondria to modulate energy metabolism and reduce ischaemia-reperfusion injury in heart.Mitochondrial ion channels as oncological targets.Mitochondrial channels: ion fluxes and more.The effect of 3-bromopyruvate on human colorectal cancer cells is dependent on glucose concentration but not hexokinase II expression.Protection From Apoptotic Cell Death During Cold Storage Followed by Rewarming in 13-Lined Ground Squirrel Tubular Cells: The Role of Prosurvival Factors X-Linked Inhibitor of Apoptosis and PhosphoAkt.Ginkgolide K attenuates neuronal injury after ischemic stroke by inhibiting mitochondrial fission and GSK-3β-dependent increases in mitochondrial membrane permeability.Proteinuria causes dysfunctional autophagy in the proximal tubule.Preconditioning mice with activators of AMPK ameliorates ischemic acute kidney injury in vivo.Kidney disease models: tools to identify mechanisms and potential therapeutic targets.Succinate induces aberrant mitochondrial fission in cardiomyocytes through GPR91 signaling.Repeated exposure of epithelial cells to apoptotic cells induces the specific selection of an adaptive phenotype: Implications for tumorigenesis

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Hexokinase regulates Bax-mediated mitochondrial membrane injury following ischemic stress

http://www.wikidata.org/entity/Q35990589

description

2011 nî lūn-bûn

@nan

2011年の論文

@ja

2011年学术文章

@wuu

2011年学术文章

@zh-cn

2011年学术文章

@zh-hans

2011年学术文章

@zh-my

2011年学术文章

@zh-sg

2011年學術文章

@yue

2011年學術文章

@zh

2011年學術文章

@zh-hant

name

Hexokinase regulates Bax-mediated mitochondrial membrane injury following ischemic stress

@ast

Hexokinase regulates Bax-mediated mitochondrial membrane injury following ischemic stress

@en

type

label

Hexokinase regulates Bax-mediated mitochondrial membrane injury following ischemic stress

@ast

Hexokinase regulates Bax-mediated mitochondrial membrane injury following ischemic stress

@en

prefLabel

Hexokinase regulates Bax-mediated mitochondrial membrane injury following ischemic stress

@ast

Hexokinase regulates Bax-mediated mitochondrial membrane injury following ischemic stress

@en

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P1433

Kidney International

P1476

Hexokinase regulates Bax-mediated mitochondrial membrane injury following ischemic stress

@en

P2093

David R Pimental

http://www.w3.org/2001/XMLSchema#string

John G Pastorino

http://www.w3.org/2001/XMLSchema#string

Jonathan M Gall

http://www.w3.org/2001/XMLSchema#string

Vincent Wong

http://www.w3.org/2001/XMLSchema#string

Zhiyong Wang

http://www.w3.org/2001/XMLSchema#string

P2860

Bcl-2 family proteins regulate the release of apoptogenic cytochrome c by the mitochondrial channel VDAC

Hexokinase II detachment from mitochondria triggers apoptosis through the permeability transition pore independent of voltage-dependent anion channels

Hexokinase II: cancer's double-edged sword acting as both facilitator and gatekeeper of malignancy when bound to mitochondria

HSP72 inhibits apoptosis-inducing factor release in ATP-depleted renal epithelial cells

Essential role of voltage-dependent anion channel in various forms of apoptosis in mammalian cells

Warburg, me and Hexokinase 2: Multiple discoveries of key molecular events underlying one of cancers' most common phenotypes, the "Warburg Effect", i.e., elevated glycolysis in the presence of oxygen

Voltage-dependent anion channels are dispensable for mitochondrial-dependent cell death

Bax inserts into the mitochondrial outer membrane by different mechanisms

Hexokinase-mitochondria interaction mediated by Akt is required to inhibit apoptosis in the presence or absence of Bax and Bak

In self-defence: hexokinase promotes voltage-dependent anion channel closure and prevents mitochondria-mediated apoptotic cell death

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1207-1216

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scholarly article

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10.1038/KI.2010.532

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11

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79

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John H Schwartz

Ramon G Bonegio

Steven C Borkan

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2011-03-23T00:00:00Z

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50832015

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21430642

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3361076

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