Novel insights into interactions between mitochondria and xanthine oxidase in acute cardiac volume overload.
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Dynamic Myofibrillar Remodeling in Live Cardiomyocytes under Static Stretch.Pathophysiological role of oxidative stress in systolic and diastolic heart failure and its therapeutic implicationsPhysiological and structural differences in spatially distinct subpopulations of cardiac mitochondria: influence of cardiac pathologiesMitochondrial redox signaling: Interaction of mitochondrial reactive oxygen species with other sources of oxidative stressXanthine oxidase inhibition preserves left ventricular systolic but not diastolic function in cardiac volume overload.Cardiomyocyte mitochondrial oxidative stress and cytoskeletal breakdown in the heart with a primary volume overload.Overexpression of TFAM or twinkle increases mtDNA copy number and facilitates cardioprotection associated with limited mitochondrial oxidative stressReperfusion injury and reactive oxygen species: The evolution of a concept.Volume overload induces autophagic degradation of procollagen in cardiac fibroblasts.Integration of cellular bioenergetics with mitochondrial quality control and autophagy.Physiologic and molecular characterization of a murine model of right ventricular volume overload.Mitochondrial genetic background modulates bioenergetics and susceptibility to acute cardiac volume overload.The multiple mechanistic faces of a pure volume overload: implications for therapyDispelling dogma and misconceptions regarding the most pharmacologically targetable source of reactive species in inflammatory disease, xanthine oxidoreductase.Cardiovascular Mitochondrial Dysfunction Induced by Cocaine: Biomarkers and Possible Beneficial Effects of Modulators of Oxidative Stress.Atrophic cardiomyocyte signaling in hypertensive heart diseaseTesticular torsion and reperfusion: evidences for biochemical and molecular alterations.Desmin loss and mitochondrial damage precede left ventricular systolic failure in volume overload heart failure.Twinkle overexpression prevents cardiac rupture after myocardial infarction by alleviating impaired mitochondrial biogenesis.
P2860
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P2860
Novel insights into interactions between mitochondria and xanthine oxidase in acute cardiac volume overload.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Novel insights into interactio ...... acute cardiac volume overload.
@ast
Novel insights into interactio ...... acute cardiac volume overload.
@en
type
label
Novel insights into interactio ...... acute cardiac volume overload.
@ast
Novel insights into interactio ...... acute cardiac volume overload.
@en
prefLabel
Novel insights into interactio ...... acute cardiac volume overload.
@ast
Novel insights into interactio ...... acute cardiac volume overload.
@en
P2093
P2860
P1476
Novel insights into interactio ...... acute cardiac volume overload.
@en
P2093
Blake R Zelickson
Chih-Chang Wei
Elena Ulasova
James D Gladden
Junying Zheng
Louis J Dell'Italia
Marcas Bamman
Mustafa I Ahmed
Scott Ballinger
Victor Darley-Usmar
P2860
P304
P356
10.1016/J.FREERADBIOMED.2011.08.022
P577
2011-08-30T00:00:00Z