Redox signaling and splicing dependent change in myosin phosphatase underlie early versus late changes in NO vasodilator reserve in a mouse LPS model of sepsis.
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Mechanisms of Vascular Smooth Muscle Contraction and the Basis for Pharmacologic Treatment of Smooth Muscle DisordersThe stress of maternal separation causes misprogramming in the postnatal maturation of rat resistance arteries.Enzymatic changes in myosin regulatory proteins may explain vasoplegia in terminally ill patients with sepsis.A splice variant of the myosin phosphatase regulatory subunit tunes arterial reactivity and suppresses response to salt loading
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Redox signaling and splicing dependent change in myosin phosphatase underlie early versus late changes in NO vasodilator reserve in a mouse LPS model of sepsis.
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2015 nî lūn-bûn
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Redox signaling and splicing d ...... n a mouse LPS model of sepsis.
@ast
Redox signaling and splicing d ...... n a mouse LPS model of sepsis.
@en
type
label
Redox signaling and splicing d ...... n a mouse LPS model of sepsis.
@ast
Redox signaling and splicing d ...... n a mouse LPS model of sepsis.
@en
prefLabel
Redox signaling and splicing d ...... n a mouse LPS model of sepsis.
@ast
Redox signaling and splicing d ...... n a mouse LPS model of sepsis.
@en
P2093
P2860
P1476
Redox signaling and splicing d ...... in a mouse LPS model of sepsis
@en
P2093
Laureano D Asico
Steven A Fisher
Xiaoxu Zheng
P2860
P304
P356
10.1152/AJPHEART.00912.2014
P50
P577
2015-02-27T00:00:00Z