Macrophage-derived Wnt opposes Notch signaling to specify hepatic progenitor cell fate in chronic liver disease. - Wikidata - wikimedia - TriplyDB

Macrophage-derived Wnt opposes Notch signaling to specify hepatic progenitor cell fate in chronic liver disease.

Macrophage-derived Wnt opposes Notch signaling to specify hepatic progenitor cell fate in chronic liver disease.

http://www.wikidata.org/entity/Q36000076

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Liver regeneration signature in hepatitis B virus (HBV)-associated acute liver failure identified by gene expression profiling Macrophages contribute to the cyclic activation of adult hair follicle stem cells The Involving Roles of Intrahepatic and Extrahepatic Stem/Progenitor Cells (SPCs) to Liver Regeneration Environmental Ligands of the Aryl Hydrocarbon Receptor and Their Effects in Models of Adult Liver Progenitor Cells Stem/Progenitor Cell Niches Involved in Hepatic and Biliary Regeneration Advances in Liver Regeneration: Revisiting Hepatic Stem/Progenitor Cells and Their Origin Stem cell-based regenerative opportunities for the liver: State of the art and beyond Advances in hepatic stem/progenitor cell biology Kupffer cells in the liver Role of liver progenitors in acute liver injury Hepatic progenitor cells in canine and feline medicine: potential for regenerative strategies Adult Hepatic Progenitor Cell Niche: How it affects the Progenitor Cell Fate Contributions of metabolic dysregulation and inflammation to nonalcoholic steatohepatitis, hepatic fibrosis, and cancer Immune modulation of stem cells and regeneration Vascular patterning sets the stage for macro and micro hepatic architecture Cellular mechanisms of tissue fibrosis. 5. Novel insights into liver fibrosis Macrophage Depletion Attenuates Extracellular Matrix Deposition and Ductular Reaction in a Mouse Model of Chronic Cholangiopathies The Immune System in Tissue Environments Regaining Homeostasis after Injury: Is "Inflammation" Always Inflammation?Immune Modulation of Cardiac Repair and Regeneration: The Art of Mending Broken Hearts YAP and TAZ: a nexus for Hippo signaling and beyond Macrophage biology in development, homeostasis and disease Adipose tissue macrophage in immune regulation of metabolism.Immunoregulatory Role of NK Cells in Tissue Inflammation and Regeneration.The spleen in liver cirrhosis: revisiting an old enemy with novel targets.Autologous bone marrow-derived cell transplantation in decompensated alcoholic liver disease: what is the impact on liver histology and gene expression patterns?Enhanced Wnt Signalling in Hepatocytes is Associated with Schistosoma japonicum Infection and Contributes to Liver Fibrosis.EpCAM+ Liver Cancer Stem-Like Cells Exhibiting Autocrine Wnt Signaling Potentially Originate in Cirrhotic Patients MicroRNA-205 signaling regulates mammary stem cell fate and tumorigenesis.Bone marrow-derived stromal cell therapy in cirrhosis: clinical evidence, cellular mechanisms, and implications for the treatment of hepatocellular carcinoma.Clonal tracing of Sox9+ liver progenitors in mouse oval cell injury.Hepatocyte nuclear factor-1beta enhances the stemness of hepatocellular carcinoma cells through activation of the Notch pathway.Divergent angiocrine signals from vascular niche balance liver regeneration and fibrosis.Liver bioengineering: from the stage of liver decellularized matrix to the multiple cellular actors and bioreactor special effects.Shotgun proteomics: identification of unique protein profiles of apoptotic bodies from biliary epithelial cells Role of liver stem cells in hepatocarcinogenesis.Fibrogenesis and Carcinogenesis in Nonalcoholic Steatohepatitis (NASH): Involvement of Matrix Metalloproteinases (MMPs) and Tissue Inhibitors of Metalloproteinase (TIMPs).Principles of liver regeneration and growth homeostasis.Cardio-miRNAs and onco-miRNAs: circulating miRNA-based diagnostics for non-cancerous and cancerous diseases Antagonistic interaction between Wnt and Notch activity modulates the regenerative capacity of a zebrafish fibrotic liver model.The promise of perfect adult tissue repair and regeneration in mammals: Learning from regenerative amphibians and fish.

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P2860

Macrophage-derived Wnt opposes Notch signaling to specify hepatic progenitor cell fate in chronic liver disease.

http://www.wikidata.org/entity/Q36000076

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P2860

NOTCH2 mutations cause Alagille syndrome, a heterogeneous disorder of the notch signaling pathway

Mutations in the human Jagged1 gene are responsible for Alagille syndrome

Alagille syndrome is caused by mutations in human Jagged1, which encodes a ligand for Notch1

A mouse model of Alagille syndrome: Notch2 as a genetic modifier of Jag1 haploinsufficiency

HES and HERP families: multiple effectors of the Notch signaling pathway

Jagged1 in the portal vein mesenchyme regulates intrahepatic bile duct development: insights into Alagille syndrome

Suppression of C/EBPalpha expression in periportal hepatoblasts may stimulate biliary cell differentiation through increased Hnf6 and Hnf1b expression

Wnt9b signaling regulates planar cell polarity and kidney tubule morphogenesis

Notch signaling controls hepatoblast differentiation by altering the expression of liver-enriched transcription factors

Redundant regulation of T cell differentiation and TCRalpha gene expression by the transcription factors LEF-1 and TCF-1

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221886178

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