Severe acute intermittent hypoxia elicits phrenic long-term facilitation by a novel adenosine-dependent mechanism.
about
Hypoxia-induced phrenic long-term facilitation: emergent propertiesAdenosine 2A receptor inhibition enhances intermittent hypoxia-induced diaphragm but not intercostal long-term facilitation.Therapeutic potential of intermittent hypoxia: a matter of dose.Acute intermittent optogenetic stimulation of nucleus tractus solitarius neurons induces sympathetic long-term facilitation.Repetitive acute intermittent hypoxia increases expression of proteins associated with plasticity in the phrenic motor nucleusRespiratory function after selective respiratory motor neuron death from intrapleural CTB-saporin injectionsMammalian target of rapamycin is required for phrenic long-term facilitation following severe but not moderate acute intermittent hypoxia.Spinal 5-HT7 receptors induce phrenic motor facilitation via EPAC-mTORC1 signaling.Acute intermittent hypoxia induced phrenic long-term facilitation despite increased SOD1 expression in a rat model of ALS.Peripheral-central chemoreceptor interaction and the significance of a critical period in the development of respiratory control.Phrenic long-term facilitation after acute intermittent hypoxia requires spinal ERK activation but not TrkB synthesisIntermittent hypoxia and neurorehabilitation.Daily acute intermittent hypoxia elicits functional recovery of diaphragm and inspiratory intercostal muscle activity after acute cervical spinal injurySpinal atypical protein kinase C activity is necessary to stabilize inactivity-induced phrenic motor facilitation.The impact of arousal state, sex, and sleep apnea on the magnitude of progressive augmentation and ventilatory long-term facilitation.Activation of 5-hyrdoxytryptamine 7 receptors within the rat nucleus tractus solitarii modulates synaptic properties.Inactivity-induced phrenic and hypoglossal motor facilitation are differentially expressed following intermittent vs. sustained neural apnea.Ventilatory long-term facilitation is evident after initial and repeated exposure to intermittent hypoxia in mice genetically depleted of brain serotoninQuantitative assessment of integrated phrenic nerve activity.Time Domains of the Hypoxic Ventilatory Response and Their Molecular Basis.Sustained Hypoxia Elicits Competing Spinal Mechanisms of Phrenic Motor Facilitation.Intermittent hypoxia, respiratory plasticity and sleep apnea in humans: present knowledge and future investigations.Spinal 5-HT7 receptors and protein kinase A constrain intermittent hypoxia-induced phrenic long-term facilitation.Inactivity-induced respiratory plasticity: protecting the drive to breathe in disorders that reduce respiratory neural activityEnhanced recovery of breathing capacity from combined adenosine 2A receptor inhibition and daily acute intermittent hypoxia after chronic cervical spinal injury.Acute intermittent hypoxia induced neural plasticity in respiratory motor control.Spinal metaplasticity in respiratory motor controlDaily acute intermittent hypoxia improves breathing function with acute and chronic spinal injury via distinct mechanisms.Adenosine-dependent phrenic motor facilitation is inflammation resistant.Chronic Intermittent Hypoxia Differentially Impacts Different States of Inspiratory Activity at the Level of the preBötzinger Complex.Signalling mechanisms of long term facilitation of breathing with intermittent hypoxia.Exposure to intermittent hypoxia and sustained hypercapnia reduces therapeutic CPAP in participants with obstructive sleep apnea.Mechanisms of Enhanced Phrenic Long-Term Facilitation in SOD1G93A Rats.Pharmacological modulation of hypoxia-induced respiratory neuroplasticity.Phrenic motor neuron adenosine 2A receptors elicit phrenic motor facilitation.Intermittent hypercapnic hypoxia during sleep does not induce ventilatory long-term facilitation in healthy males.Enhancement of phrenic long-term facilitation following repetitive acute intermittent hypoxia is blocked by the glycolytic inhibitor 2-deoxyglucose.Spinal BDNF-induced phrenic motor facilitation requires PKCθ activity.Phrenic long-term facilitation following intrapleural CTB-SAP-induced respiratory motor neuron death.Intermittent hypoxia promotes recovery of respiratory motor function in spinal cord-injured mice depleted of serotonin in the central nervous system.
P2860
Q26864573-38E7A8EC-4A45-4CB7-A191-C08356122F21Q34579320-6D53388D-456E-4CA1-8FB3-87E181C20389Q35043452-F0A7CE16-C329-4857-80C6-EAF5DBE836C6Q35086373-78278A1B-6B1C-4A13-83D5-2648C0DCC63EQ35220339-E9244FB4-5813-4602-B9D4-2926D8214583Q35561748-FB92A1EB-6701-4A08-AAF8-F76EBBB77009Q36064784-F2A82B1E-7240-4484-A7C8-56797489C82DQ36094185-F54C48B1-4FE5-44BE-AAEA-18395891B211Q36278903-B2BAE463-C7E9-433C-9A44-FC0FEDD21F09Q36306261-96BFD5C6-AD7E-454B-BB71-6D9275371B19Q36320919-8480B288-6E91-4175-91A4-47E915CC6986Q36382232-76AB2181-F1A0-420D-9324-2973B3E9770AQ36473351-B82B6FE1-B6C2-4138-A19B-16CDEF54D109Q36488893-7B5C8BC6-B1FF-4BAE-94DE-40156105E340Q36531006-F79E3797-2D13-48EF-BBC0-4D91B5A2C677Q36654900-B21750B4-05B0-4734-BBCA-AA90C2433259Q36850754-1068468A-2B08-44A5-9D25-0304CDCCC0CEQ36942242-B0D07501-8876-433D-A29E-348497B7F749Q37009007-6C7CC932-07B4-4153-AE1A-C9F7FE4529CCQ37069679-FBF21165-2181-454B-B890-E65C4F30742BQ37127837-9A7F20B0-897D-4C67-8765-5E6D69BDF88DQ37179232-E10F246A-B3F8-466F-98B6-487E241EC8C8Q37323774-6E968AA8-17C1-419D-95A2-2BE40C8AEB15Q37504342-36807682-7F4A-41FE-8933-5E374E5DA823Q37540272-6EC8E74A-A57A-47B9-8C3E-042792FE2009Q38115569-75206439-52B1-4FBA-833F-803439300DD5Q38365377-1D56380E-1BF9-4834-88C9-D32BAC8EEF66Q38760867-AC93F3DD-6C23-4A22-B2D4-C5AF8461282EQ40419760-98B906CC-2374-453D-A814-4BCA299CDAC9Q41705938-78AB671C-1D13-4684-A123-323A6CB6FA73Q42144708-6A872070-D38B-4353-BD2B-BBED4470039CQ46442994-497373E2-D5D5-41C1-AB8F-E5648CC8DDB3Q47134004-3245A993-0633-43CF-95B5-687BA0D86161Q47379993-DBBE771D-B34F-47BC-86BE-110D355EB78EQ47641350-F5F7F1AC-74F1-4069-8AA4-DD9C8244702BQ47719982-D39343B3-E98B-4A4F-AB9B-BCF78920CBF6Q47747292-3139558B-7CF4-4FAF-8FB6-1FBF387701C0Q47788441-D652D81D-2480-4920-8DEE-9F4E36A84BE9Q47797776-C01CEB71-9196-476A-85FF-FD0D15B4E350Q47838102-B060E724-A371-4986-A787-5442833B1F9F
P2860
Severe acute intermittent hypoxia elicits phrenic long-term facilitation by a novel adenosine-dependent mechanism.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Severe acute intermittent hypo ...... adenosine-dependent mechanism.
@ast
Severe acute intermittent hypo ...... adenosine-dependent mechanism.
@en
type
label
Severe acute intermittent hypo ...... adenosine-dependent mechanism.
@ast
Severe acute intermittent hypo ...... adenosine-dependent mechanism.
@en
prefLabel
Severe acute intermittent hypo ...... adenosine-dependent mechanism.
@ast
Severe acute intermittent hypo ...... adenosine-dependent mechanism.
@en
P2093
P2860
P1476
Severe acute intermittent hypo ...... adenosine-dependent mechanism.
@en
P2093
Erica A Dale
Gordon S Mitchell
Nicole L Nichols
P2860
P304
P356
10.1152/JAPPLPHYSIOL.00060.2012
P407
P577
2012-03-08T00:00:00Z