Cellular mechanisms of insulin resistance, lipodystrophy and atherosclerosis induced by HIV protease inhibitors.
about
The role of protease inhibitors in the pathogenesis of HIV-associated lipodystrophy: cellular mechanisms and clinical implicationsOxidative Stress Mediates the Antiproliferative Effects of Nelfinavir in Breast Cancer CellsAtherosclerotic Cardiovascular Disease and Anti-Retroviral Therapy.Effect of ritonavir and atazanavir on human subcutaneous preadipocyte proliferation and differentiation.Novel macromolecular inhibitors of human immunodeficiency virus-1 protease.Effects of the HIV protease inhibitor ritonavir on GLUT4 knock-out mice.HIV protease inhibitors elicit volume-sensitive Cl- current in cardiac myocytes via mitochondrial ROS.Cardio-metabolic effectsof HIV protease inhibitors (lopinavir/ritonavir)Nordihydroguaiaretic acid (NDGA) inhibits ritonavir-induced endothelial dysfunction in porcine pulmonary arteries.Cytochrome P-450 CYP2E1 knockout mice are protected against high-fat diet-induced obesity and insulin resistanceHuman immunodeficiency virus (HIV)-1 viral protein R suppresses transcriptional activity of peroxisome proliferator-activated receptor {gamma} and inhibits adipocyte differentiation: implications for HIV-associated lipodystrophy.Antiretroviral-related adipocyte dysfunction and lipodystrophy in HIV-infected patients: Alteration of the PPARγ-dependent pathwaysEffects of ritonavir on adipocyte gene expression: evidence for a stress-related responseRoles and mechanisms of human immunodeficiency virus protease inhibitor ritonavir and other anti-human immunodeficiency virus drugs in endothelial dysfunction of porcine pulmonary arteries and human pulmonary artery endothelial cells.Human Immunodeficiency Virus and Risk of Type 2 Diabetes in a Large Adult Cohort in Jos, Nigeria.Postreceptoral adipocyte insulin resistance induced by nelfinavir is caused by insensitivity of PKB/Akt to phosphatidylinositol-3,4,5-trisphosphate.Nutrition and the HIV-associated lipodystrophy syndrome.Cardiovascular involvements in HIV-infected patients.Adipose Tissue in HIV Infection.The induction of a nucleoplasmic reticulum by prelamin A accumulation requires CTP:phosphocholine cytidylyltransferase-α.Acipimox, an inhibitor of lipolysis, attenuates atherogenesis in LDLR-null mice treated with HIV protease inhibitor ritonavir.Regulation of adipocyte lipolysis by degradation of the perilipin protein: nelfinavir enhances lysosome-mediated perilipin proteolysis.Indinavir and nelfinavir inhibit proximal insulin receptor signaling and salicylate abrogates inhibition: potential role of the NFkappa B pathway.Adipose Tissue is Enriched for Activated and Late-Differentiated CD8+ T Cells and Shows Distinct CD8+ Receptor Usage, Compared With Blood in HIV-Infected Persons.
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P2860
Cellular mechanisms of insulin resistance, lipodystrophy and atherosclerosis induced by HIV protease inhibitors.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
2005年论文
@zh
2005年论文
@zh-cn
name
Cellular mechanisms of insulin ...... ed by HIV protease inhibitors.
@ast
Cellular mechanisms of insulin ...... ed by HIV protease inhibitors.
@en
type
label
Cellular mechanisms of insulin ...... ed by HIV protease inhibitors.
@ast
Cellular mechanisms of insulin ...... ed by HIV protease inhibitors.
@en
prefLabel
Cellular mechanisms of insulin ...... ed by HIV protease inhibitors.
@ast
Cellular mechanisms of insulin ...... ed by HIV protease inhibitors.
@en
P2093
P2860
P1476
Cellular mechanisms of insulin ...... ced by HIV protease inhibitors
@en
P2093
P2860
P356
10.1111/J.1365-201X.2004.01383.X
P577
2005-01-01T00:00:00Z