Early recurrences in histologically benign/grade I meningiomas are associated with large tumors and coexistence of monosomy 14 and del(1p36) in the ancestral tumor cell clone - Wikidata - wikimedia - TriplyDB

Early recurrences in histologically benign/grade I meningiomas are associated with large tumors and coexistence of monosomy 14 and del(1p36) in the ancestral tumor cell clone

Early recurrences in histologically benign/grade I meningiomas are associated with large tumors and coexistence of monosomy 14 and del(1p36) in the ancestral tumor cell clone

http://www.wikidata.org/entity/Q36013346

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Differential expression profiling analyses identifies downregulation of 1p, 6q, and 14q genes and overexpression of 6p histone cluster 1 genes as markers of recurrence in meningiomas.Association between inflammatory infiltrates and isolated monosomy 22/del(22q) in meningiomas Genetic/molecular alterations of meningiomas and the signaling pathways targeted.Proposal for a new risk stratification classification for meningioma based on patient age, WHO tumor grade, size, localization, and karyotype.Could serum measurements of S100 proteins be reliable markers to predict recurrence in meningiomas?Diagnostic Challenges in Meningioma.Tissue Engineering Strategies as Tools for Personalized Meningioma Treatment.Correlation of volumetric growth and histological grade in 50 meningiomas.Global epigenetic profiling identifies methylation subgroups associated with recurrence-free survival in meningioma.Clinicopathological and molecular characteristics of pediatric meningiomas.Genetic alterations associated with progression and recurrence in meningiomas.Immunocytochemical analysis of glucose transporter protein-1 (GLUT-1) in typical, brain invasive, atypical and anaplastic meningioma.Prognostic factors for olfactory groove meningioma with nasal cavity extension.The protein expression profile of meningioma cells is associated with distinct cytogenetic tumour subgroups.Tumor grade-related NDRG2 gene expression in primary and recurrent intracranial meningiomas.Meningioma recurrence.Radiation effects on human leptomeningeal cell response to cerebrospinal fluid and PDGF-BB.Identification of key genes and pathways in meningioma by bioinformatics analysis.

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P2860

Early recurrences in histologically benign/grade I meningiomas are associated with large tumors and coexistence of monosomy 14 and del(1p36) in the ancestral tumor cell clone

http://www.wikidata.org/entity/Q36013346

description

2007 nî lūn-bûn

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2007年の論文

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Early recurrences in histologi ...... the ancestral tumor cell clone

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Early recurrences in histologi ...... the ancestral tumor cell clone

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Early recurrences in histologi ...... the ancestral tumor cell clone

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Alberto Orfao

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Ana B Espinosa

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Angel Maillo

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José María Sayagués

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Marta Merino

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María Dolores Tabernero

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Monica Lara

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Pablo Sousa

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P2860

Implications of a RAD54L polymorphism (2290C/T) in human meningiomas as a risk factor and/or a genetic marker

Integrative genomic analysis identifies NDRG2 as a candidate tumor suppressor gene frequently inactivated in clinically aggressive meningioma

Loss of chromosomes 22 and 14 in the malignant progression of meningiomas. A comparative study of fluorescence in situ hybridization (FISH) and standard cytogenetic analysis.

The WHO classification of tumors of the nervous system.

The recurrence of intracranial meningiomas after surgical treatment

The prognostic value of progesterone receptor status in meningiomas.

Alterations of the tumor suppressor genes CDKN2A (p16(INK4a)), p14(ARF), CDKN2B (p15(INK4b)), and CDKN2C (p18(INK4c)) in atypical and anaplastic meningiomas

Intratumoral patterns of clonal evolution in meningiomas as defined by multicolor interphase fluorescence in situ hybridization (FISH): is there a relationship between histopathologically benign and atypical/anaplastic lesions?

The molecular genetics of meningiomas.

Analysis of genomic alterations in benign, atypical, and anaplastic meningiomas: toward a genetic model of meningioma progression.

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