Stereological analysis of GluR2-immunoreactive hilar neurons in the pilocarpine model of temporal lobe epilepsy: correlation of cell loss with mossy fiber sprouting
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Potential implications of a monosynaptic pathway from mossy cells to adult-born granule cells of the dentate gyrusSuppression of adult neurogenesis increases the acute effects of kainic acidClassic hippocampal sclerosis and hippocampal-onset epilepsy produced by a single “cryptic” episode of focal hippocampal excitation in awake ratsInhibition of the mammalian target of rapamycin signaling pathway suppresses dentate granule cell axon sprouting in a rodent model of temporal lobe epilepsyIncreased excitatory synaptic input to granule cells from hilar and CA3 regions in a rat model of temporal lobe epilepsyHigh ratio of synaptic excitation to synaptic inhibition in hilar ectopic granule cells of pilocarpine-treated rats.Blockade of excitatory synaptogenesis with proximal dendrites of dentate granule cells following rapamycin treatment in a mouse model of temporal lobe epilepsy.Rapamycin suppresses mossy fiber sprouting but not seizure frequency in a mouse model of temporal lobe epilepsy.Systemic autoimmunity in TAM triple knockout mice causes inflammatory brain damage and cell death.Hippocampal injury, atrophy, synaptic reorganization, and epileptogenesis after perforant pathway stimulation-induced status epilepticus in the mouse.Initial loss but later excess of GABAergic synapses with dentate granule cells in a rat model of temporal lobe epilepsy.Surviving mossy cells enlarge and receive more excitatory synaptic input in a mouse model of temporal lobe epilepsy.Contributions of mature granule cells to structural plasticity in temporal lobe epilepsy.Strain differences in seizure-induced cell death following pilocarpine-induced status epilepticus.Is there a critical period for mossy fiber sprouting in a mouse model of temporal lobe epilepsy?Reorganization of supramammillary-hippocampal pathways in the rat pilocarpine model of temporal lobe epilepsy: evidence for axon terminal sprouting.Manganese-enhanced magnetic resonance imaging detects mossy fiber sprouting in the pilocarpine model of epilepsy.Hilar mossy cell degeneration causes transient dentate granule cell hyperexcitability and impaired pattern separation.Mossy cell dendritic structure quantified and compared with other hippocampal neurons labeled in rats in vivoHilar mossy cell circuitry controlling dentate granule cell excitability.Hilar mossy cells of the dentate gyrus: a historical perspective.Corruption of the dentate gyrus by "dominant" granule cells: Implications for dentate gyrus function in health and disease.Impact of rapamycin on status epilepticus induced hippocampal pathology and weight gain.Minimal latency to hippocampal epileptogenesis and clinical epilepsy after perforant pathway stimulation-induced status epilepticus in awake ratsAccumulation of abnormal adult-generated hippocampal granule cells predicts seizure frequency and severity.A reorganized GABAergic circuit in a model of epilepsy: evidence from optogenetic labeling and stimulation of somatostatin interneurons.High-dose rapamycin blocks mossy fiber sprouting but not seizures in a mouse model of temporal lobe epilepsy.Enhanced tonic GABA current in normotopic and hilar ectopic dentate granule cells after pilocarpine-induced status epilepticusAblation of Newly Generated Hippocampal Granule Cells Has Disease-Modifying Effects in EpilepsyEmbryonic stem cell-derived neural precursor grafts for treatment of temporal lobe epilepsy.Morphologic integration of hilar ectopic granule cells into dentate gyrus circuitry in the pilocarpine model of temporal lobe epilepsy.Mechanisms regulating neuronal excitability and seizure development following mTOR pathway hyperactivation.The enigmatic mossy cell of the dentate gyrusNew insights into the role of hilar ectopic granule cells in the dentate gyrus based on quantitative anatomic analysis and three-dimensional reconstruction.Suppression of epileptogenesis-associated changes in response to seizures in FGF22-deficient mice.Neurogenesis is enhanced and mossy fiber sprouting arises in FGF7-deficient mice during development.Posttraumatic epilepsy after controlled cortical impact injury in mice.Localized overexpression of FGF-2 and BDNF in hippocampus reduces mossy fiber sprouting and spontaneous seizures up to 4 weeks after pilocarpine-induced status epilepticus.Seizure frequency correlates with loss of dentate gyrus GABAergic neurons in a mouse model of temporal lobe epilepsy.Acute restraint stress decreases c-fos immunoreactivity in hilar mossy cells of the adult dentate gyrus.
P2860
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P2860
Stereological analysis of GluR2-immunoreactive hilar neurons in the pilocarpine model of temporal lobe epilepsy: correlation of cell loss with mossy fiber sprouting
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Stereological analysis of GluR ...... oss with mossy fiber sprouting
@ast
Stereological analysis of GluR ...... oss with mossy fiber sprouting
@en
type
label
Stereological analysis of GluR ...... oss with mossy fiber sprouting
@ast
Stereological analysis of GluR ...... oss with mossy fiber sprouting
@en
prefLabel
Stereological analysis of GluR ...... oss with mossy fiber sprouting
@ast
Stereological analysis of GluR ...... oss with mossy fiber sprouting
@en
P2860
P1476
Stereological analysis of GluR ...... oss with mossy fiber sprouting
@en
P2093
J Victor Nadler
Yiqun Jiao
P2860
P304
P356
10.1016/J.EXPNEUROL.2007.03.025
P407
P577
2007-03-30T00:00:00Z