Exploring joint effects of genes and the clinical efficacy of morphine for cancer pain: OPRM1 and COMT gene. - Wikidata - wikimedia - TriplyDB

Exploring joint effects of genes and the clinical efficacy of morphine for cancer pain: OPRM1 and COMT gene.

Exploring joint effects of genes and the clinical efficacy of morphine for cancer pain: OPRM1 and COMT gene.

http://www.wikidata.org/entity/Q36016764

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Progress in genetic studies of pain and analgesia The pharmacogenomics of pain management: prospects for personalized medicine Increased sensitivity to thermal pain following a single opiate dose is influenced by the COMT val(158)met polymorphism The val158met polymorphism of human catechol-O-methyltransferase (COMT) affects anterior cingulate cortex activation in response to painful laser stimulation.Pharmacogenetics of OPRM1.OPRM1 c.118A>G Polymorphism and Duration of Morphine Treatment Associated with Morphine Doses and Quality-of-Life in Palliative Cancer Pain Settings.Precise genetic mapping and integrative bioinformatics in Diversity Outbred mice reveals Hydin as a novel pain gene.Subjective health complaints in patients with lumbar radicular pain and disc herniation are associated with a sex - OPRM1 A118G polymorphism interaction: a prospective 1-year observational study Genetic Testing for Opioid Pain Management: A Primer The genetic influence on the cortical processing of experimental pain and the moderating effect of pain status OPRM1 SNP (A118G): involvement in disease development, treatment response, and animal models.Genetics of pain perception, COMT and postoperative pain management in children.The application of genomic and molecular data in the treatment of chronic cancer pain.An imaging genetics approach to understanding social influence Opioid rotation in clinical practice.Pharmacogenomic considerations in opioid analgesia.Genetic contributions to clinical pain and analgesia: avoiding pitfalls in genetic research Recent advances in the use of opioids for cancer pain Buprenorphine signalling is compromised at the N40D polymorphism of the human μ opioid receptor in vitro Individualized pain medicine Drug response profiles to experimental pain are opioid and pain modality specific Genotyping test with clinical factors: better management of acute postoperative pain?Association of OPRM1 and COMT single-nucleotide polymorphisms with hospital length of stay and treatment of neonatal abstinence syndrome.Variations in opioid receptor genes in neonatal abstinence syndrome μ-Opioid receptor gene A118G polymorphism predicts survival in patients with breast cancer MAPK1/ERK2 as novel target genes for pain in head and neck cancer patients.Molecular epidemiology, cancer-related symptoms, and cytokines pathway.The influence of tumor necrosis factor-alpha -308 G/A and IL-6 -174 G/C on pain and analgesia response in lung cancer patients receiving supportive care.Human pain and genetics: some basics Pharmacogenetics of analgesic drugs Pain and stress in a systems perspective: reciprocal neural, endocrine, and immune interactions.Combined analysis of circulating β-endorphin with gene polymorphisms in OPRM1, CACNAD2 and ABCB1 reveals correlation with pain, opioid sensitivity and opioid-related side effects.The correlation between post-operative fentanyl requirements and μ-opioid receptor gene A118G polymorphism in patients undergoing radical gastrectomy.Analgesia and central side-effects: two separate dimensions of morphine response.Consequences of the 118A>G polymorphism in the OPRM1 gene: translation from bench to bedside?Morphine-induced antinociception and reward in "humanized" mice expressing the mu opioid receptor A118G polymorphism Pharmacogenetics of chronic pain and its treatment.Life-threatening adverse events following therapeutic opioid administration in adults: is pharmacogenetic analysis useful?Ethnicity interacts with the OPRM1 gene in experimental pain sensitivity.The pharmacogenetics of analgesia.

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Exploring joint effects of genes and the clinical efficacy of morphine for cancer pain: OPRM1 and COMT gene.

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P2860

Human mu opiate receptor. cDNA and genomic clones, pharmacologic characterization and chromosomal assignment

Single-nucleotide polymorphism in the human mu opioid receptor gene alters beta-endorphin binding and activity: possible implications for opiate addiction

Pain measurement tools and methods in clinical research in palliative care: recommendations of an Expert Working Group of the European Association of Palliative Care

Pain Measurement Tools and Methods in Clinical Research in Palliative Care

A single nucleotide polymorphic mutation in the human mu-opioid receptor severely impairs receptor signaling.

The genetic mediation of individual differences in sensitivity to pain and its inhibition.

The mu opiate receptor as a candidate gene for pain: polymorphisms, variations in expression, nociception, and opiate responses.

Pain genes?: natural variation and transgenic mutants.

No evidence of association or interaction between the IL4RA, IL4, and IL13 genes in type 1 diabetes

COMT val158met genotype affects mu-opioid neurotransmitter responses to a pain stressor.

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