Release of full-length 55-kDa TNF receptor 1 in exosome-like vesicles: a mechanism for generation of soluble cytokine receptors. - Wikidata - wikimedia - TriplyDB

Release of full-length 55-kDa TNF receptor 1 in exosome-like vesicles: a mechanism for generation of soluble cytokine receptors.

Release of full-length 55-kDa TNF receptor 1 in exosome-like vesicles: a mechanism for generation of soluble cytokine receptors.

http://www.wikidata.org/entity/Q36017850

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The brefeldin A-inhibited guanine nucleotide-exchange protein, BIG2, regulates the constitutive release of TNFR1 exosome-like vesicles Identification and proteomic profiling of exosomes in human urine An association between RBMX, a heterogeneous nuclear ribonucleoprotein, and ARTS-1 regulates extracellular TNFR1 release Colocalization of endogenous TNF with a functional intracellular splice form of human TNF receptor type 2.Routes and mechanisms of extracellular vesicle uptake Glycosyl-Phosphatidyl-Inositol (GPI)-Anchors and Metalloproteases: Their Roles in the Regulation of Exosome Composition and NKG2D-Mediated Immune Recognition The Influence of Ionizing Radiation on Exosome Composition, Secretion and Intercellular Communication 14-Deoxyandrographolide desensitizes hepatocytes to tumour necrosis factor-alpha-induced apoptosis through calcium-dependent tumour necrosis factor receptor superfamily member 1A release via the NO/cGMP pathway Formation and role of exosomes in cancer.Extracellular TNFR1 release requires the calcium-dependent formation of a nucleobindin 2-ARTS-1 complex.Primary cell cultures from human renal cortex and renal-cell carcinoma evidence a differential expression of two spliced isoforms of Annexin A3.Flow cytometric analysis of circulating microparticles in plasma.Cellular phenotype switching and microvesicles.Soluble TNF receptors are associated with infarct size and ventricular dysfunction in ST-elevation myocardial infarction TNF/TNFR1 signaling mediates doxorubicin-induced diaphragm weakness.Falling into TRAPS--receptor misfolding in the TNF receptor 1-associated periodic fever syndrome Isolation of biologically-active exosomes from human plasma.Double-stranded RNA induces shedding of the 34-kDa soluble TNFR1 from human airway epithelial cells via TLR3-TRIF-RIP1-dependent signaling: roles for dual oxidase 2- and caspase-dependent pathways.Activation of TNFR1 ectodomain shedding by mitochondrial Ca2+ determines the severity of inflammation in mouse lung microvessels.Osteoprotegerin in exosome-like vesicles from human cultured tubular cells and urine Soluble tumor necrosis factor receptor 1 and 2 predict outcomes in advanced chronic kidney disease: a prospective cohort study.Elevation of circulating TNF receptors 1 and 2 increases the risk of end-stage renal disease in American Indians with type 2 diabetes.Tumour necrosis factor receptor trafficking dysfunction opens the TRAPS door to pro-inflammatory cytokine secretion.Identification and proteomic profiling of exosomes in human cerebrospinal fluid.Circulating TNF receptors 1 and 2 predict stage 3 CKD in type 1 diabetes.Abnormal disulfide-linked oligomerization results in ER retention and altered signaling by TNFR1 mutants in TNFR1-associated periodic fever syndrome (TRAPS)Extracellular membrane vesicles and immune regulation in the brain.Mechanism of evenness interrupted (Evi)-exosome release at synaptic boutons.Extracellular vesicles and their synthetic analogues in aging and age-associated brain diseases.Microparticles of human atherosclerotic plaques enhance the shedding of the tumor necrosis factor-alpha converting enzyme/ADAM17 substrates, tumor necrosis factor and tumor necrosis factor receptor-1 Localization, Shedding, Regulation and Function of Aminopeptidase N/CD13 on Fibroblast like Synoviocytes.Characterization of membrane-shed microvesicles from cytokine-stimulated β-cells using proteomics strategies.A role for cGMP in inducible nitric-oxide synthase (iNOS)-induced tumor necrosis factor (TNF) α-converting enzyme (TACE/ADAM17) activation, translocation, and TNF receptor 1 (TNFR1) shedding in hepatocytes.Circulating TNFR1 exosome-like vesicles partition with the LDL fraction of human plasma.Molecular mechanisms of soluble cytokine receptor generation.Molecular pathways: tumor-derived microvesicles and their interactions with immune cells in vivo cAMP-dependent protein kinase A (PKA) signaling induces TNFR1 exosome-like vesicle release via anchoring of PKA regulatory subunit RIIbeta to BIG2.Collective cell migration requires vesicular trafficking for chemoattractant delivery at the trailing edge.Cytomegalovirus-infected human endothelial cells can stimulate allogeneic CD4+ memory T cells by releasing antigenic exosomes The comorbidity of HIV-associated neurocognitive disorders and Alzheimer's disease: a foreseeable medical challenge in post-HAART era

P2860

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P2860

Release of full-length 55-kDa TNF receptor 1 in exosome-like vesicles: a mechanism for generation of soluble cytokine receptors.

http://www.wikidata.org/entity/Q36017850

description

2004 nî lūn-bûn

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2004年の論文

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name

Release of full-length 55-kDa ...... of soluble cytokine receptors.

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Release of full-length 55-kDa ...... of soluble cytokine receptors.

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Release of full-length 55-kDa ...... of soluble cytokine receptors.

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Release of full-length 55-kDa ...... of soluble cytokine receptors.

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Release of full-length 55-kDa ...... of soluble cytokine receptors.

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Release of full-length 55-kDa ...... of soluble cytokine receptors.

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Proceedings of the National Academy of Sciences of the United States of America

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Release of full-length 55-kDa ...... of soluble cytokine receptors.

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Caitriona Buckley

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Farshid N Rouhani

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Feras I Hawari

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Maryann Kaler

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Stewart J Levine

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Xinle Cui

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Zu-Xi Yu

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P2860

Prevention of constitutive TNF receptor 1 signaling by silencer of death domains

Molecular cloning and expression of a receptor for human tumor necrosis factor

Soluble forms of tumor necrosis factor receptors (TNF-Rs). The cDNA for the type I TNF-R, cloned using amino acid sequence data of its soluble form, encodes both the cell surface and a soluble form of the receptor

Identification of ARTS-1 as a novel TNFR1-binding protein that promotes TNFR1 ectodomain shedding

Induction of TNF receptor I-mediated apoptosis via two sequential signaling complexes

B lymphocytes secrete antigen-presenting vesicles

TNF-R1 signaling: a beautiful pathway

TNF recruits TRADD to the plasma membrane but not the trans-Golgi network, the principal subcellular location of TNF-R1

Exosomes: composition, biogenesis and function

Activated platelets release two types of membrane vesicles: microvesicles by surface shedding and exosomes derived from exocytosis of multivesicular bodies and alpha-granules

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10.1073/PNAS.0307981100

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101

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8901650

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14745008

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