Chop deficiency prevents UUO-induced renal fibrosis by attenuating fibrotic signals originated from Hmgb1/TLR4/NFκB/IL-1β signaling.

Chop deficiency prevents UUO-induced renal fibrosis by attenuating fibrotic signals originated from Hmgb1/TLR4/NFκB/IL-1β signaling.

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http://www.wikidata.org/entity/Q36022252
Vascular Remodelling and Mesenchymal Transition in Systemic SclerosisNupr1/Chop signal axis is involved in mitochondrion-related endothelial cell apoptosis induced by methamphetamineHMGB1 exacerbates bronchiolitis obliterans syndrome via RAGE/NF-κB/HPSE signaling to enhance latent TGF-β release from ECM.C/EBP homologous protein (CHOP) deficiency ameliorates renal fibrosis in unilateral ureteral obstructive kidney disease.Endoplasmic reticulum stress in bone marrow-derived cells prevents acute cardiac inflammation and injury in response to angiotensin II.Endoplasmic reticulum stress, a new wrestler, in the pathogenesis of idiopathic pulmonary fibrosis.Proinflammatory effect of high-mobility group protein B1 on keratinocytes: an autocrine mechanism underlying psoriasis development.Bacteroides fragilis Enterotoxin Induces Formation of Autophagosomes in Endothelial Cells but Interferes with Fusion with Lysosomes for Complete Autophagic Flux through a Mitogen-Activated Protein Kinase-, AP-1-, and C/EBP Homologous Protein-DependeHMGB1 induces lung fibroblast to myofibroblast differentiation through NF‑κB‑mediated TGF‑β1 release.Endoplasmic reticulum stress inhibition attenuates hypertensive chronic kidney disease through reduction in proteinuria.Both conditional ablation and overexpression of E2 SUMO-conjugating enzyme (UBC9) in mouse pancreatic beta cells result in impaired beta cell function.Transcription Factor C/EBP Homologous Protein in Health and Diseases.Endogenous ligands of TLR4 promote unresolving tissue fibrosis: Implications for systemic sclerosis and its targeted therapy.High Uric Acid-Induced Epithelial-Mesenchymal Transition of Renal Tubular Epithelial Cells via the TLR4/NF-kB Signaling Pathway.
P2860

Chop deficiency prevents UUO-induced renal fibrosis by attenuating fibrotic signals originated from Hmgb1/TLR4/NFκB/IL-1β signaling.

Item
http://www.wikidata.org/entity/Q36022252
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
Chop deficiency prevents UUO-i ...... gb1/TLR4/NFκB/IL-1β signaling.
@ast
Chop deficiency prevents UUO-i ...... gb1/TLR4/NFκB/IL-1β signaling.
@en
type
label
Chop deficiency prevents UUO-i ...... gb1/TLR4/NFκB/IL-1β signaling.
@ast
Chop deficiency prevents UUO-i ...... gb1/TLR4/NFκB/IL-1β signaling.
@en
prefLabel
Chop deficiency prevents UUO-i ...... gb1/TLR4/NFκB/IL-1β signaling.
@ast
Chop deficiency prevents UUO-i ...... gb1/TLR4/NFκB/IL-1β signaling.
@en
P1433
P1476
P2093
P2860
P2888
P304
P31
P356
P478
P577
P5875
P6179
P698
P921
P932
P356
P1433
P1476
Chop deficiency prevents UUO-i ...... gb1/TLR4/NFκB/IL-1β signaling.
@en
P2093
P2860
P2888
P304
P31
P356
P478
P577
P5875
P6179
P698
P921
P932