Posttetanic potentiation critically depends on an enhanced Ca(2+) sensitivity of vesicle fusion mediated by presynaptic PKC. - Wikidata - wikimedia - TriplyDB

Posttetanic potentiation critically depends on an enhanced Ca(2+) sensitivity of vesicle fusion mediated by presynaptic PKC.

Posttetanic potentiation critically depends on an enhanced Ca(2+) sensitivity of vesicle fusion mediated by presynaptic PKC.

http://www.wikidata.org/entity/Q36023865

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Molecular Machines Regulating the Release Probability of Synaptic Vesicles at the Active Zone Extremely Low Frequency Electromagnetic Fields Facilitate Vesicle Endocytosis by Increasing Presynaptic Calcium Channel Expression at a Central Synapse Munc18-1 is a dynamically regulated PKC target during short-term enhancement of transmitter release Sustained firing of cartwheel cells in the dorsal cochlear nucleus evokes endocannabinoid release and retrograde suppression of parallel fiber synapses.Post-tetanic increase in the fast-releasing synaptic vesicle pool at the expense of the slowly releasing pool.Dynamin-1 deletion enhances post-tetanic potentiation and quantal size after tetanic stimulation at the calyx of Held.Cocaine-seeking is associated with PKC-dependent reduction of excitatory signaling in accumbens shell D2 dopamine receptor-expressing neurons.In vivo evidence for neuroplasticity in older adults.Platelet Activating Factor Enhances Synaptic Vesicle Exocytosis Via PKC, Elevated Intracellular Calcium, and Modulation of Synapsin 1 Dynamics and Phosphorylation.Altered short-term plasticity in the prefrontal cortex after early life seizures.Adaptive regulation maintains posttetanic potentiation at cerebellar granule cell synapses in the absence of calcium-dependent PKC Calcium-dependent isoforms of protein kinase C mediate glycine-induced synaptic enhancement at the calyx of Held.Selective saturation of slow endocytosis at a giant glutamatergic central synapse lacking dynamin 1 Phosphorylation of synaptotagmin-1 controls a post-priming step in PKC-dependent presynaptic plasticity Short-term forms of presynaptic plasticity.Short-term presynaptic plasticity.Theoretical models of synaptic short term plasticity.Translating neuronal activity at the synapse: presynaptic calcium sensors in short-term plasticity.Synaptic plasticity in the auditory system: a review.Molecular mechanisms governing Ca(2+) regulation of evoked and spontaneous release.The calyx of Held in the auditory system: Structure, function, and development.Calcium-dependent isoforms of protein kinase C mediate posttetanic potentiation at the calyx of Held.Calcium-dependent PKC isoforms have specialized roles in short-term synaptic plasticity Synaptic gain-of-function effects of mutant Cav2.1 channels in a mouse model of familial hemiplegic migraine are due to increased basal [Ca2+]i Additive effects on the energy barrier for synaptic vesicle fusion cause supralinear effects on the vesicle fusion rate.Calcium-Dependent Protein Kinase C Is Not Required for Post-Tetanic Potentiation at the Hippocampal CA3 to CA1 Synapse.SNAP-25 phosphorylation at Ser187 regulates synaptic facilitation and short-term plasticity in an age-dependent manner.Post-tetanic potentiation involves the presynaptic binding of calcium to calmodulin A C1-C2 Module in Munc13 Inhibits Calcium-Dependent Neurotransmitter Release.A single episode of high intensity sound inhibits long-term potentiation in the hippocampus of rats.Developmental upregulation of presynaptic NCKX underlies the decrease of mitochondria-dependent posttetanic potentiation at the rat calyx of Held synapse.Post-tetanic potentiation is caused by two signalling mechanisms affecting quantal size and quantal content.Single bursts of individual granule cells functionally rearrange feed-forward inhibition.Homeostatic plasticity induced by brief activity deprivation enhances long-term potentiation in the mature rat hippocampus.Persistent but labile synaptic plasticity at excitatory synapses.BDNF-TrkB Signaling Coupled to nPKCε and cPKCβI Modulate the Phosphorylation of the Exocytotic Protein Munc18-1 During Synaptic Activity at the Neuromuscular Junction.Molecular Mechanisms of Short-Term Plasticity: Role of Synapsin Phosphorylation in Augmentation and Potentiation of Spontaneous Glutamate Release

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P2860

Posttetanic potentiation critically depends on an enhanced Ca(2+) sensitivity of vesicle fusion mediated by presynaptic PKC.

http://www.wikidata.org/entity/Q36023865

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Posttetanic potentiation criti ...... n mediated by presynaptic PKC.

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Posttetanic potentiation criti ...... n mediated by presynaptic PKC.

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Posttetanic potentiation criti ...... n mediated by presynaptic PKC.

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Posttetanic potentiation criti ...... n mediated by presynaptic PKC.

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Posttetanic potentiation criti ...... n mediated by presynaptic PKC.

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Natalya Korogod

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Ralf Schneggenburger

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Xuelin Lou

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P2860

Genetic analysis of synaptotagmin 2 in spontaneous and Ca2+-triggered neurotransmitter release

Short-term synaptic plasticity

Phosphorylation of Munc-18/n-Sec1/rbSec1 by protein kinase C: its implication in regulating the interaction of Munc-18/n-Sec1/rbSec1 with syntaxin

Beta phorbol ester- and diacylglycerol-induced augmentation of transmitter release is mediated by Munc13s and not by PKCs

Combining deconvolution and noise analysis for the estimation of transmitter release rates at the calyx of held.

Identification of PKC-isoform-specific biological actions using pharmacological approaches.

PTP and LTP at a hippocampal mossy fiber-interneuron synapse

Action potential broadening and frequency-dependent facilitation of calcium signals in pituitary nerve terminals

Protein kinase c increases the apparent affinity of the release machinery to Ca2+ by enhancing the release machinery downstream of the Ca2+ sensor.

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15923-15928

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10.1073/PNAS.0704603104

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104

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