about
Evidence against either a premature stop codon or the absence of obese gene mRNA in human obesityThe adipocyte specific transcription factor C/EBPalpha modulates human ob gene expressionCentral role of ceramide biosynthesis in body weight regulation, energy metabolism, and the metabolic syndromeObese gene expression at in vivo levels by fat pads derived from s.c. implanted 3T3-F442A preadipocytesGenetic dissection of complex genetic factor involved in NIDDM of OLETF ratLeptin-dependent platelet aggregation and arterial thrombosis suggests a mechanism for atherothrombotic disease in obesityCircadian system, sleep and endocrinologyA new paradigm for the understanding of obesity: the role of stem cells.TNF-alpha is required for the attraction of mesenchymal precursors to white adipose tissue in Ob/ob mice.Transcriptional activation of the mouse obese (ob) gene by CCAAT/enhancer binding protein alphaRegulated expression of the obese gene product (leptin) in white adipose tissue and 3T3-L1 adipocytes.A genetic map of quantitative trait loci for body weight in the mouse.NAG-1/GDF-15 prevents obesity by increasing thermogenesis, lipolysis and oxidative metabolism.Weighing in on the lean genes.Overexpression of the obese (ob) gene in adipose tissue of human obese subjects.Increased obese mRNA expression in omental fat cells from massively obese humans.Strategies for the molecular genetic analysis of obesity in humans.Leptin is related to body fat content in male distance runners.A molecular model for the genetic and phenotypic characteristics of the mouse lethal yellow (Ay) mutation.How can drug addiction help us understand obesity?OB protein binds specifically to the choroid plexus of mice and rats.Sphingolipid metabolism and obesity-induced inflammation.Leptin as an important link between obesity and cardiovascular risk factors in men with acute myocardial infarctionCould there be a fine-tuning role for brain-derived adipokines in the regulation of bodyweight and prevention of obesity?Endotoxin and cytokines induce expression of leptin, the ob gene product, in hamsters.Induction of ob gene expression by corticosteroids is accompanied by body weight loss and reduced food intake.Thiazolidinediones repress ob gene expression in rodents via activation of peroxisome proliferator-activated receptor gamma.Long-term and rapid regulation of ob mRNA levels in adipose tissue from normal (Sprague Dawley rats) and obese (db/db mice, fa/fa rats) rodents.Reduction of food intake and weight gain by the ob protein requires a specific secondary structure and is reversible.Transgenic mouse models of disease: altering adipose tissue function in vivo.Genetic aspects of obesity.The rat corpulent (cp) mutation maps to the same interval on (Pgm1-Glut1) rat chromosome 5 as the fatty (fa) mutation.Expression studies of key adipogenic transcriptional factors reveal that the anti-adipogenic properties of retinol in primary cultured human preadipocytes are due to retinol per se.CRISPR-Cas9-mediated generation of obese and diabetic mouse models.
P2860
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P2860
description
1992 nî lūn-bûn
@nan
1992年の論文
@ja
1992年論文
@yue
1992年論文
@zh-hant
1992年論文
@zh-hk
1992年論文
@zh-mo
1992年論文
@zh-tw
1992年论文
@wuu
1992年论文
@zh
1992年论文
@zh-cn
name
Tackling a weighty problem.
@ast
Tackling a weighty problem.
@en
type
label
Tackling a weighty problem.
@ast
Tackling a weighty problem.
@en
prefLabel
Tackling a weighty problem.
@ast
Tackling a weighty problem.
@en
P1433
P1476
Tackling a weighty problem.
@en
P2093
Friedman JM
P304
P356
10.1016/0092-8674(92)90402-X
P407
P577
1992-04-01T00:00:00Z