Quantifying crosstalk among interferon-γ, interleukin-12, and tumor necrosis factor signaling pathways within a TH1 cell model
about
In silico model-based inference: a contemporary approach for hypothesis testing in network biologyEnhancing the discovery and development of immunotherapies for cancer using quantitative and systems pharmacology: Interleukin-12 as a case studyInduction of Wnt-inducible signaling protein-1 correlates with invasive breast cancer oncogenesis and reduced type 1 cell-mediated cytotoxic immunity: a retrospective studyA quantitative systems approach to identify paracrine mechanisms that locally suppress immune response to Interleukin-12 in the B16 melanoma model.Systems modeling of molecular mechanisms controlling cytokine-driven CD4+ T cell differentiation and phenotype plasticity.In silico model-based inference: an emerging approach for inverse problems in engineering better medicinesUnderstanding immunology via engineering design: the role of mathematical prototyping.Downregulation of Hlx closely related to the decreased expressions of T-bet and Runx3 in patients with gastric cancer may be associated with a pathological event leading to the imbalance of Th1/Th2.Interlocked positive and negative feedback network motifs regulate β-catenin activity in the adherens junction pathway.An evolutionary perspective on anti-tumor immunityExploring higher-order EGFR oligomerisation and phosphorylation--a combined experimental and theoretical approach.Collapse of the tumor stroma is triggered by IL-12 induction of Fas.Synergistic Communication between CD4+ T Cells and Monocytes Impacts the Cytokine Environment.Inferring the Impact of Regulatory Mechanisms that Underpin CD8+ T Cell Control of B16 Tumor Growth In vivo Using Mechanistic Models and Simulation.Melanoma exosomes deliver a complex biological payload that upregulates PTPN11 to suppress T lymphocyte function.Negative interactions and feedback regulations are required for transient cellular response.Analytical solution of steady-state equations for chemical reaction networks with bilinear rate laws.Identifying local mechanisms for tumor-derived immunosuppression: an integrated phenotypic screening approach.
P2860
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P2860
Quantifying crosstalk among interferon-γ, interleukin-12, and tumor necrosis factor signaling pathways within a TH1 cell model
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2012 nî lūn-bûn
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2012年の論文
@ja
2012年学术文章
@wuu
2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
@yue
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@zh-hant
name
Quantifying crosstalk among in ...... thways within a TH1 cell model
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Quantifying crosstalk among in ...... thways within a TH1 cell model
@en
type
label
Quantifying crosstalk among in ...... thways within a TH1 cell model
@ast
Quantifying crosstalk among in ...... thways within a TH1 cell model
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prefLabel
Quantifying crosstalk among in ...... thways within a TH1 cell model
@ast
Quantifying crosstalk among in ...... thways within a TH1 cell model
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P2860
P1433
P1476
Quantifying crosstalk among in ...... thways within a TH1 cell model
@en
P2093
Emily Chambers
Ning Cheng
P2860
P356
10.1126/SCISIGNAL.2002657
P577
2012-04-17T00:00:00Z