Severe inflammatory arthritis and lymphadenopathy in the absence of TNF. - Wikidata - wikimedia - TriplyDB

Severe inflammatory arthritis and lymphadenopathy in the absence of TNF.

Severe inflammatory arthritis and lymphadenopathy in the absence of TNF.

http://www.wikidata.org/entity/Q36028645

about

Infliximab therapy in rheumatoid arthritis and ankylosing spondylitis-induced specific antinuclear and antiphospholipid autoantibodies without autoimmune clinical manifestations: a two-year prospective study Accelerated development of arthritis in mice lacking endothelial selectins.Perforin deficiency attenuates collagen-induced arthritis.Therapeutic strategies for the clinical blockade of IL-6/gp130 signaling Utilizing regulatory T cells against rheumatoid arthritis Interleukin-1β and interleukin-6 in arthritis animal models: roles in the early phase of transition from acute to chronic inflammation and relevance for human rheumatoid arthritis SOCS-3 negatively regulates innate and adaptive immune mechanisms in acute IL-1-dependent inflammatory arthritis.Suppressor of cytokine signaling-1 regulates acute inflammatory arthritis and T cell activation.Loss of tumor necrosis factor alpha potentiates transforming growth factor beta-mediated pathogenic tissue response during wound healing.Molecular targets in immune-mediated diseases: the case of tumour necrosis factor and rheumatoid arthritis.Decreasing TNF-alpha results in less fibrosis and earlier resolution of granulomatous experimental autoimmune thyroiditis.Anti-CD4 monoclonal antibody treatment in acute and early chronic antigen induced arthritis: influence on macrophage activation.Regulation of haematopoiesis by growth factors - emerging insights and therapies.CD69 downregulates autoimmune reactivity through active transforming growth factor-beta production in collagen-induced arthritis.TNF and receptors in organ-specific autoimmune disease: multi-layered functioning mirrored in animal models IKK{beta} as a target for treatment of inflammation induced bone loss.Critical roles for interleukin 1 and tumor necrosis factor alpha in antibody-induced arthritis Anti-Inflammatory Activity of a Polymeric Proanthocyanidin from Serjania schiedeana.Tumor necrosis factor polymorphisms associated with tumor necrosis factor production influence the risk of idiopathic intermediate uveitis.Potential new targets in arthritis therapy: interleukin (IL)-17 and its relation to tumour necrosis factor and IL-1 in experimental arthritis.Regulation of autoimmune arthritis by the pro-inflammatory cytokine interferon-gamma.Regulation of autoimmune inflammation by pro-inflammatory cytokines Consumption of hydrolyzable tannins-rich pomegranate extract suppresses inflammation and joint damage in rheumatoid arthritis.Critical role for granulocyte colony-stimulating factor in inflammatory arthritis.Lessons from animal models of arthritis over the past decade.Classical and Paradoxical Effects of TNF-α on Bone Homeostasis.Bone-targeting endogenous secretory receptor for advanced glycation end products rescues rheumatoid arthritis Therapeutic Targeting of the G-CSF Receptor Reduces Neutrophil Trafficking and Joint Inflammation in Antibody-Mediated Inflammatory Arthritis.TNF-alpha is a critical negative regulator of type 1 immune activation during intracellular bacterial infection Negative regulation of lung inflammation and immunopathology by TNF-α during acute influenza infection If the treatment works, do we need to know why?: the promise of immunotherapy for experimental medicine.Multiple interleukin-1beta-converting enzymes contribute to inflammatory arthritis.Blockade of tumor necrosis factor in collagen-induced arthritis reveals a novel immunoregulatory pathway for Th1 and Th17 cells.Anti-IL-17A therapy protects against bone erosion in experimental models of rheumatoid arthritis.IL-6 signal blockade ameliorates the enhanced osteoclastogenesis and the associated joint destruction in a novel FcγRIIB-deficient rheumatoid arthritis mouse model.Interleukin-1 and tumor necrosis factor α blockade treatment of experimental polymyositis in mice.

P2860

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P2860

Severe inflammatory arthritis and lymphadenopathy in the absence of TNF.

http://www.wikidata.org/entity/Q36028645

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2001 nî lūn-bûn

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2001年の論文

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2001年論文

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2001年論文

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2001年論文

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2001年論文

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2001年論文

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2001年论文

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2001年论文

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2001年论文

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name

Severe inflammatory arthritis and lymphadenopathy in the absence of TNF.

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Severe inflammatory arthritis and lymphadenopathy in the absence of TNF.

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Severe inflammatory arthritis and lymphadenopathy in the absence of TNF.

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Severe inflammatory arthritis and lymphadenopathy in the absence of TNF.

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Severe inflammatory arthritis and lymphadenopathy in the absence of TNF.

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Severe inflammatory arthritis and lymphadenopathy in the absence of TNF.

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Journal of Clinical Investigation

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Severe inflammatory arthritis and lymphadenopathy in the absence of TNF.

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Campbell IK

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Lawlor KE

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O'Donnell K

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Wicks IP

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P2860

Transgenic mice expressing human tumour necrosis factor: a predictive genetic model of arthritis

Characterization of tumor necrosis factor-deficient mice

Distinct roles for lymphotoxin-alpha and tumor necrosis factor in organogenesis and spatial organization of lymphoid tissue

Immune and inflammatory responses in TNF alpha-deficient mice: a critical requirement for TNF alpha in the formation of primary B cell follicles, follicular dendritic cell networks and germinal centers, and in the maturation of the humoral immune re

Role of cytokines in rheumatoid arthritis

Mature T lymphocyte apoptosis--immune regulation in a dynamic and unpredictable antigenic environment.

Tumor necrosis factor: a master-regulator of leukocyte movement.

Anti-TNF alpha therapy of rheumatoid arthritis: what have we learned?

Current evidence for the induction of autoimmune rheumatic manifestations by cytokine therapy.

Increased peripheral T cell reactivity to microbial antigens and collagen type II in rheumatoid arthritis after treatment with soluble TNFalpha receptors.

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