Role of macrophage chemoattractant protein-1 in acute inflammation after lung contusion - Wikidata - wikimedia - TriplyDB

Role of macrophage chemoattractant protein-1 in acute inflammation after lung contusion

Role of macrophage chemoattractant protein-1 in acute inflammation after lung contusion

http://www.wikidata.org/entity/Q36049758

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Interleukin 10 overexpression alters survival in the setting of gram-negative pneumonia following lung contusion Activation of hypoxia-inducible factor-1α in type 2 alveolar epithelial cell is a major driver of acute inflammation following lung contusion Low-dose TNF augments fracture healing in normal and osteoporotic bone by up-regulating the innate immune response.Nogo-B protects mice against lipopolysaccharide-induced acute lung injury Electroporation-mediated delivery of genes in rodent models of lung contusion.Alveolar macrophage depletion increases the severity of acute inflammation following nonlethal unilateral lung contusion in mice Molecular Characterization of Hypoxic Alveolar Epithelial Cells After Lung Contusion Indicates an Important Role for HIF-1α.Role of pulmonary artery reactivity and nitric oxide in injury and inflammation following lung contusion.The protective role of MnTBAP in oxidant-mediated injury and inflammation in a rat model of lung contusion.Electroporation-mediated delivery of the FER gene in the resolution of trauma-related fatal pneumonia.Phase-directed therapy: TSG-6 targeted to early inflammation improves bleomycin-injured lungs.The innate immune response to lower respiratory tract E. Coli infection and the role of the CCL2-CCR2 axis in neonatal mice.Gram-Negative Pneumonia Alters Large-Vein Cell-Adhesion Molecule Profile and Potentiates Experimental Stasis Venous Thrombosis.Double-Stranded RNA Interacts With Toll-Like Receptor 3 in Driving the Acute Inflammatory Response Following Lung Contusion.Toll-Like Receptor-9 (TLR9) is Requisite for Acute Inflammatory Response and Injury Following Lung Contusion.Development of a murine model of blunt hepatic trauma.Surgical stimulation of angiogenesis.The heme oxygenase system selectively suppresses the proinflammatory macrophage m1 phenotype and potentiates insulin signaling in spontaneously hypertensive rats.

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Role of macrophage chemoattractant protein-1 in acute inflammation after lung contusion

http://www.wikidata.org/entity/Q36049758

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Role of macrophage chemoattractant protein-1 in acute inflammation after lung contusion

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Role of macrophage chemoattractant protein-1 in acute inflammation after lung contusion

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Role of macrophage chemoattractant protein-1 in acute inflammation after lung contusion

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Role of macrophage chemoattractant protein-1 in acute inflammation after lung contusion

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Role of macrophage chemoattractant protein-1 in acute inflammation after lung contusion

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Role of macrophage chemoattractant protein-1 in acute inflammation after lung contusion

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RCMB.2011-0358OC

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American Journal of Respiratory Cell and Molecular Biology

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Role of macrophage chemoattractant protein-1 in acute inflammation after lung contusion

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Bi Yu

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Bruce A Davidson

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Cory M Hogaboam

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Jadwiga D Helinski

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Krishnan Raghavendran

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Laura Ochoa-Frongia

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Madathilparambil V Suresh

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Matthew D Bender

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Paul R Knight

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P2860

Organization and differential expression of the human monocyte chemoattractant protein 1 receptor gene. Evidence for the role of the carboxyl-terminal tail in receptor trafficking

Platelet-activating factor receptor plays a role in lung injury and death caused by Influenza A in mice

Monocyte chemoattractant protein-1 released from alveolar macrophages mediates the systemic inflammation of acute alveolar hypoxia

Enhanced pulmonary allergic responses to Aspergillus in CCR2-/- mice

Significant involvement of CCL2 (MCP-1) in inflammatory disorders of the lung.

Alveolar epithelial type II cells activate alveolar macrophages and mitigate P. Aeruginosa infection.

Neutrophils and acute lung injury.

Enhanced expression of Duffy antigen in the lungs during suppurative pneumonia.

Pulmonary contusion: review of the clinical entity.

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797-806

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10.1165/RCMB.2011-0358OC

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6

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46

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Bethany B Moore

David Machado-Aranda

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2012-01-26T00:00:00Z

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22281985

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