Paneth cell ablation in the presence of Klebsiella pneumoniae induces necrotizing enterocolitis (NEC)-like injury in the small intestine of immature mice.
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Necrotizing enterocolitis: new insights into pathogenesis and mechanismsImmunologic and Hematological Abnormalities in Necrotizing Enterocolitis.Commensal and probiotic bacteria may prevent NEC by maturing intestinal host defenses.Postnatal development of the small intestinal mucosa drives age-dependent, regio-selective susceptibility to Escherichia coli K1 infection.Paneth-cell-disruption-induced necrotizing enterocolitis in mice requires live bacteria and occurs independently of TLR4 signalingUpdate in pathogenesis and prospective in treatment of necrotizing enterocolitis.The role of intestinal epithelial barrier function in the development of NEC.Intrauterine Growth Restriction Alters Mouse Intestinal Architecture during Development.Preterm infant gut microbiota affects intestinal epithelial development in a humanized microbiome gnotobiotic mouse model.Paneth cells and necrotizing enterocolitis.The ErbB4 ligand neuregulin-4 protects against experimental necrotizing enterocolitis.Paneth cells and necrotizing enterocolitis: a novel hypothesis for disease pathogenesis.Evaluation of hematologic variables in newborn C57/BL6 mice up to day 35.Mapping the New World of Necrotizing Enterocolitis (NEC): Review and Opinion.Innate immune signaling in the pathogenesis of necrotizing enterocolitis.Heme oxygenase-1 deficiency promotes the development of necrotizing enterocolitis-like intestinal injury in a newborn mouse model.The immunological landscape in necrotising enterocolitisZnT2-Mediated Zinc Import Into Paneth Cell Granules Is Necessary for Coordinated Secretion and Paneth Cell Function in Mice.The Role of Mucosal Immunity in the Pathogenesis of Necrotizing Enterocolitis.ErbB receptors and their growth factor ligands in pediatric intestinal inflammation.Progress in the field of necrotising enterocolitis--year 2012.Pathogenesis of necrotizing enterocolitis: modeling the innate immune response.FLLL32, a curcumin analog, ameliorates intestinal injury in necrotizing enterocolitis.Tumor necrosis factor induces developmental stage-dependent structural changes in the immature small intestine.Heme oxygenase-1 confers protection and alters T-cell populations in a mouse model of neonatal intestinal inflammation.Paneth cells in intestinal physiology and pathophysiology.
P2860
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P2860
Paneth cell ablation in the presence of Klebsiella pneumoniae induces necrotizing enterocolitis (NEC)-like injury in the small intestine of immature mice.
description
2012 nî lūn-bûn
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2012年の論文
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2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
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2012年论文
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name
Paneth cell ablation in the pr ...... ll intestine of immature mice.
@ast
Paneth cell ablation in the pr ...... ll intestine of immature mice.
@en
type
label
Paneth cell ablation in the pr ...... ll intestine of immature mice.
@ast
Paneth cell ablation in the pr ...... ll intestine of immature mice.
@en
prefLabel
Paneth cell ablation in the pr ...... ll intestine of immature mice.
@ast
Paneth cell ablation in the pr ...... ll intestine of immature mice.
@en
P2093
P2860
P356
P1476
Paneth cell ablation in the pr ...... all intestine of immature mice
@en
P2093
Chunxian Zhang
David Bader
Jörn-Hendrik Weitkamp
Lawrence S Prince
Michael P Sherman
Steven J McElroy
P2860
P304
P356
10.1242/DMM.009001
P577
2012-02-10T00:00:00Z