Effects of N-acetylaspartylglutamate (NAAG) peptidase inhibition on release of glutamate and dopamine in prefrontal cortex and nucleus accumbens in phencyclidine model of schizophrenia
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In vivo assessment of neurotransmitters and modulators with magnetic resonance spectroscopy: application to schizophreniaN-Acetylaspartate reductions in brain injury: impact on post-injury neuroenergetics, lipid synthesis, and protein acetylationNAAG peptidase inhibition in the periaqueductal gray and rostral ventromedial medulla reduces flinching in the formalin model of inflammationPharmacokinetics and pharmacodynamics of the glutamate carboxypeptidase II inhibitor 2-MPPA show prolonged alleviation of neuropathic pain through an indirect mechanism.Glutamate carboxypeptidase II gene knockout attenuates oxidative stress and cortical apoptosis after traumatic brain injury.ATP-binding Cassette Subfamily C Member 5 (ABCC5) Functions as an Efflux Transporter of Glutamate Conjugates and Analogs.NAAG peptidase inhibitors and deletion of NAAG peptidase gene enhance memory in novel object recognition test.The prostate-specific membrane antigen: lessons and current clinical implications from 20 years of research.A role for the locus coeruleus in the analgesic efficacy of N-acetylaspartylglutamate peptidase (GCPII) inhibitors ZJ43 and 2-PMPA.NAAG Peptidase Inhibitors Act via mGluR3: Animal Models of Memory, Alzheimer's, and Ethanol Intoxication.Mice lacking glutamate carboxypeptidase II develop normally, but are less susceptible to traumatic brain injury.Maternal inflammation leads to impaired glutamate homeostasis and up-regulation of glutamate carboxypeptidase II in activated microglia in the fetal/newborn rabbit brain.Striatal N-acetylaspartate synthetase Shati/Nat8l regulates depression-like behaviors via mGluR3-mediated serotonergic suppression in mice.Do reductions in brain N-acetylaspartate levels contribute to the etiology of some neuropsychiatric disorders?Report on the Symposium "Molecular Mechanisms Involved in Neurodegeneration".Neonatal exposure to MK-801 reduces mRNA expression of mGlu3 receptors in the medial prefrontal cortex of adolescent rats.
P2860
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P2860
Effects of N-acetylaspartylglutamate (NAAG) peptidase inhibition on release of glutamate and dopamine in prefrontal cortex and nucleus accumbens in phencyclidine model of schizophrenia
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Effects of N-acetylaspartylglu ...... clidine model of schizophrenia
@ast
Effects of N-acetylaspartylglu ...... clidine model of schizophrenia
@en
type
label
Effects of N-acetylaspartylglu ...... clidine model of schizophrenia
@ast
Effects of N-acetylaspartylglu ...... clidine model of schizophrenia
@en
prefLabel
Effects of N-acetylaspartylglu ...... clidine model of schizophrenia
@ast
Effects of N-acetylaspartylglu ...... clidine model of schizophrenia
@en
P2093
P2860
P356
P1476
Effects of N-acetylaspartylglu ...... clidine model of schizophrenia
@en
P2093
Daiying Zuo
John R Moffett
Joseph H Neale
Rafal T Olszewski
Tomasz Bzdega
P2860
P304
21773-21782
P356
10.1074/JBC.M112.363226
P407
P577
2012-05-08T00:00:00Z