The carbon monoxide-releasing molecule CORM-2 inhibits the inflammatory response induced by cytokines in Caco-2 cells.
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The carbon monoxide releasing molecule CORM-2 attenuates Pseudomonas aeruginosa biofilm formationA Photoactive Carbon-Monoxide-Releasing Protein Cage for Dose-Regulated Delivery in Living CellsNitric oxide and carbon monoxide antagonize TGF-β through ligand-independent internalization of TβR1/ALK5Carbon monoxide blocks lipopolysaccharide-induced gene expression by interfering with proximal TLR4 to NF-kappaB signal transduction in human monocytesThe CO donor CORM-2 inhibits LPS-induced vascular cell adhesion molecule-1 expression and leukocyte adhesion in human rheumatoid synovial fibroblasts.Carbon monoxide-releasing molecule-2 suppresses thrombomodulin and endothelial protein C receptor expression of human umbilical vein endothelial cells induced by lipopolysaccharide in vitro.Heme oxygenase-1 and carbon monoxide regulate intestinal homeostasis and mucosal immune responses to the enteric microbiota.Carbon monoxide abrogates ischemic insult to neuronal cells via the soluble guanylate cyclase-cGMP pathway.Friend or foe? Carbon monoxide and the mitochondria.Protective effects of carbon monoxide-releasing molecule-2 on the barrier function of intestinal epithelial cells.Carbon monoxide down-regulates α4β1 integrin-specific ligand binding and cell adhesion: a possible mechanism for cell mobilization.Carbon Monoxide (CO) Released from Tricarbonyldichlororuthenium (II) Dimer (CORM-2) in Gastroprotection against Experimental Ethanol-Induced Gastric DamageSpontaneous CO release from Ru(II)(CO)2-protein complexes in aqueous solution, cells, and miceColonic insufflation with carbon monoxide gas inhibits the development of intestinal inflammation in rats.Suppression by CD4+CD25+ regulatory T cells is dependent on expression of heme oxygenase-1 in antigen-presenting cells.NIR-Responsive On-Demand Release of CO from Metal Carbonyl-Caged Graphene Oxide Nanomedicine.Exogenous carbon monoxide inhibits neutrophil infiltration in LPS-induced sepsis by interfering with FPR1 via p38 MAPK but not GRK2.Carbon Monoxide Inhibits Porcine Reproductive and Respiratory Syndrome Virus Replication by the Cyclic GMP/Protein Kinase G and NF-κB Signaling PathwayHeme oxygenase-1: its therapeutic roles in inflammatory diseases.Haem oxygenase-1: non-canonical roles in physiology and pathology.Carbon monoxide: an unusual drug.Emerging concepts on the anti-inflammatory actions of carbon monoxide-releasing molecules (CO-RMs).CO and CO-releasing molecules (CO-RMs) in acute gastrointestinal inflammation.Differential effects of the essential oils of Lavandula luisieri and Eryngium duriaei subsp. juresianum in cell models of two chronic inflammatory diseases.Increased metabolites of 5-lipoxygenase from hypoxic ovarian cancer cells promote tumor-associated macrophage infiltration.Cell damage following carbon monoxide releasing molecule exposure: implications for therapeutic applications.Gaseous Mediators in Gastrointestinal Mucosal Defense and Injury.Heme oxygenase 1 controls early innate immune response of macrophages to Salmonella Typhimurium infection.Nrf2 is essential for the anti-inflammatory effect of carbon monoxide in LPS-induced inflammation.Ultraviolet-A1 irradiation therapy for systemic lupus erythematosus.A metal carbonyl-protein needle composite designed for intracellular CO delivery to modulate NF-κB activity.Physiological activities of carbon monoxide-releasing molecules: Ca ira.Prevention of NSAID-Induced Small Intestinal Mucosal Injury: Prophylactic Potential of Lansoprazole.Heme oxygenase-1: a novel therapeutic target for gastrointestinal diseases.Carbon monoxide-releasing molecule attenuates allograft airway rejection.Pinocembrin Suppresses H2O2-Induced Mitochondrial Dysfunction by a Mechanism Dependent on the Nrf2/HO-1 Axis in SH-SY5Y Cells.The CO-releasing molecule CORM-2 is a novel regulator of the inflammatory process in osteoarthritic chondrocytes.Cerebroprotective effects of the CO-releasing molecule CORM-A1 against seizure-induced neonatal vascular injury.Repurposing tin mesoporphyrin as an immune checkpoint inhibitor shows therapeutic efficacy in preclinical models of cancer.The Interaction of CORM-2 with Block Copolymers Containing Poly(4-vinylpyridine): Macromolecular Scaffolds for Carbon Monoxide Delivery in Biological Systems.
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P2860
The carbon monoxide-releasing molecule CORM-2 inhibits the inflammatory response induced by cytokines in Caco-2 cells.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
The carbon monoxide-releasing ...... by cytokines in Caco-2 cells.
@ast
The carbon monoxide-releasing ...... by cytokines in Caco-2 cells.
@en
type
label
The carbon monoxide-releasing ...... by cytokines in Caco-2 cells.
@ast
The carbon monoxide-releasing ...... by cytokines in Caco-2 cells.
@en
prefLabel
The carbon monoxide-releasing ...... by cytokines in Caco-2 cells.
@ast
The carbon monoxide-releasing ...... by cytokines in Caco-2 cells.
@en
P2860
P356
P1476
The carbon monoxide-releasing ...... d by cytokines in Caco-2 cells
@en
P2093
P2860
P304
P356
10.1038/SJ.BJP.0707184
P407
P577
2007-03-05T00:00:00Z