Glomerular VEGF resistance induced by PKCδ/SHP-1 activation and contribution to diabetic nephropathy. - Wikidata - wikimedia - TriplyDB

Glomerular VEGF resistance induced by PKCδ/SHP-1 activation and contribution to diabetic nephropathy.

Glomerular VEGF resistance induced by PKCδ/SHP-1 activation and contribution to diabetic nephropathy.

http://www.wikidata.org/entity/Q36055520

about

Targeting inflammation in diabetes: Newer therapeutic options Inflammation and oxidative stress in diabetic nephropathy: new insights on its inhibition as new therapeutic targets MicroRNA-29a promotion of nephrin acetylation ameliorates hyperglycemia-induced podocyte dysfunction.Increased SHP-1 protein expression by high glucose levels reduces nephrin phosphorylation in podocytes Vascular complications of diabetes: mechanisms of injury and protective factors Minimal change disease onset observed after bevacizumab administration.Immunopathogenesis of idiopathic nephrotic syndrome with relapse.Pyruvate kinase M2 activation may protect against the progression of diabetic glomerular pathology and mitochondrial dysfunction.Expression of SHP-1 induced by hyperglycemia prevents insulin actions in podocytes.Regulation of Nephrin Phosphorylation in Diabetes and Chronic Kidney Injury.Effect of NADPH oxidase inhibitor-apocynin on the expression of Src homology-2 domain-containing phosphatase-1 (SHP-1) exposed renal ischemia/reperfusion injury in rats.Diabetic Microvascular Disease: An Endocrine Society Scientific Statement.SHP-1 activation inhibits vascular smooth muscle cell proliferation and intimal hyperplasia in a rodent model of insulin resistance and diabetes.Blood glucose fluctuation accelerates renal injury involved to inhibit the AKT signaling pathway in diabetic rats.Obesity-associated glomerular inflammation increases albuminuria without renal histological changes.Methotrexate induced pneumatosis intestinalis under hemodialysis patient.Oxidative Stress, Apoptosis, and Mitochondrial Function in Diabetic Nephropathy.Nephrin Signaling in the Podocyte: An Updated View of Signal Regulation at the Slit Diaphragm and Beyond.

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Glomerular VEGF resistance induced by PKCδ/SHP-1 activation and contribution to diabetic nephropathy.

http://www.wikidata.org/entity/Q36055520

description

2012 nî lūn-bûn

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name

Glomerular VEGF resistance ind ...... ution to diabetic nephropathy.

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Glomerular VEGF resistance ind ...... ution to diabetic nephropathy.

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Akira Mima

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Chenzhong Li

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Christian Rask-Madsen

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George L King

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Koji Mizutani

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Michael Leitges

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Motonobu Matsumoto

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Munehiro Kitada

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Pedro Geraldes

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Qian Li

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P2860

Activation of PKC-delta and SHP-1 by hyperglycemia causes vascular cell apoptosis and diabetic retinopathy

VEGF inhibition and renal thrombotic microangiopathy

Intensive blood-glucose control with sulphonylureas or insulin compared with conventional treatment and risk of complications in patients with type 2 diabetes (UKPDS 33). UK Prospective Diabetes Study (UKPDS) Group

Biochemistry and molecular cell biology of diabetic complications

Diminished loss of proteoglycans and lack of albuminuria in protein kinase C-alpha-deficient diabetic mice

Characterization of protein kinase C beta isoform's action on retinoblastoma protein phosphorylation, vascular endothelial growth factor-induced endothelial cell proliferation, and retinal neovascularization.

Protein kinase C-delta mediates neuronal apoptosis in the retinas of diabetic rats via the Akt signaling pathway

Glucose or diabetes activates p38 mitogen-activated protein kinase via different pathways

Protein oxidation and aging.

Mechanisms of progression and regression of renal lesions of chronic nephropathies and diabetes.

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2963-2974

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scholarly article

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10.1096/FJ.11-202994

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26

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224006078

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22499584

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3382088

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