Prevalence of prostate cancer metastases after intravenous inoculation provides clues into the molecular basis of dormancy in the bone marrow microenvironment.
about
Growth Arrest-Specific 6 (GAS6) Promotes Prostate Cancer Survival by G1 Arrest/S Phase Delay and Inhibition of Apoptosis During Chemotherapy in Bone MarrowOsteoblasts Are the Centerpiece of the Metastatic Bone MicroenvironmentDissecting the role of bone marrow stromal cells on bone metastases.Stem cells and bone diseases: new tools, new perspectiveGAS6 receptor status is associated with dormancy and bone metastatic tumor formationBone marrow as a metastatic niche for disseminated tumor cells from solid tumors.Modeling selective elimination of quiescent cancer cells from bone marrow.Cancer stem cells and their role in metastasisEndogenous GAS6 and Mer receptor signaling regulate prostate cancer stem cells in bone marrow.Parathyroid hormone-related protein drives a CD11b+Gr1+ cell-mediated positive feedback loop to support prostate cancer growth.Axl is required for TGF-β2-induced dormancy of prostate cancer cells in the bone marrow.Cancer subclonal genetic architecture as a key to personalized medicine.The marrow niche controls the cancer stem cell phenotype of disseminated prostate cancer.Molecular insights of Gas6/TAM in cancer development and therapy.Synergistic action of image-guided radiotherapy and androgen deprivation therapy.The Bone Microenvironment: a Fertile Soil for Tumor Growth.Enhanced regulation of cell cycle and suppression of osteoblast differentiation molecular signatures by prostate cancer stem-like holoclones.Endocytic adaptor protein epsin is elevated in prostate cancer and required for cancer progression.Overcoming intratumor heterogeneity of polygenic cancer drug resistance with improved biomarker integrationMer Tyrosine Kinase Regulates Disseminated Prostate Cancer Cellular Dormancy.Cancer cell dormancy: mechanisms and implications of cancer recurrence and metastasis.Apoptosis-induced CXCL5 accelerates inflammation and growth of prostate tumor metastases in bone.Targeting Gas6/TAM in cancer cells and tumor microenvironment.Bone Marrow Microenvironment as a Regulator and Therapeutic Target for Prostate Cancer Bone Metastasis.Parallels between hematopoietic stem cell and prostate cancer disseminated tumor cell regulation.Sympathetic Signaling Reactivates Quiescent Disseminated Prostate Cancer Cells in the Bone Marrow.
P2860
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P2860
Prevalence of prostate cancer metastases after intravenous inoculation provides clues into the molecular basis of dormancy in the bone marrow microenvironment.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
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2012年论文
@zh-cn
name
Prevalence of prostate cancer ...... bone marrow microenvironment.
@ast
Prevalence of prostate cancer ...... bone marrow microenvironment.
@en
type
label
Prevalence of prostate cancer ...... bone marrow microenvironment.
@ast
Prevalence of prostate cancer ...... bone marrow microenvironment.
@en
prefLabel
Prevalence of prostate cancer ...... bone marrow microenvironment.
@ast
Prevalence of prostate cancer ...... bone marrow microenvironment.
@en
P2093
P2860
P50
P356
P1433
P1476
Prevalence of prostate cancer ...... e bone marrow microenvironment
@en
P2093
Aaron M Havens
Evan T Keller
Janice E Berry
Jeena Joseph
Jin Koo Kim
Jingcheng Wang
Laurie K McCauley
Natalie McGregor
Peter Carmeliet
P2860
P304
P356
10.1596/NEO.111740
P577
2012-05-01T00:00:00Z