Mutant SOD1 forms ion channel: implications for ALS pathophysiology.
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Mechanisms of mutant SOD1 induced mitochondrial toxicity in amyotrophic lateral sclerosisPyrimethamine decreases levels of SOD1 in leukocytes and cerebrospinal fluid of ALS patients: a phase I pilot studyComparative modeling of hypothetical amyloid pores based on cylindrin.The effect of amyotrophic lateral sclerosis-linked exogenous SOD1-G93A on electrophysiological properties and intracellular calcium in cultured rat astrocytes.Role of Neuroinflammation in Amyotrophic Lateral Sclerosis: Cellular Mechanisms and Therapeutic Implications.Abnormal SDS-PAGE migration of cytosolic proteins can identify domains and mechanisms that control surfactant binding.Poloxamer 188 decreases membrane toxicity of mutant SOD1 and ameliorates pathology observed in SOD1 mouse model for ALS.The voltage-gated calcium channel blocker lomerizine is neuroprotective in motor neurons expressing mutant SOD1, but not TDP-43.
P2860
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P2860
Mutant SOD1 forms ion channel: implications for ALS pathophysiology.
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2011 nî lūn-bûn
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2011年の論文
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2011年学术文章
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2011年学术文章
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2011年学术文章
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2011年学术文章
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Mutant SOD1 forms ion channel: implications for ALS pathophysiology.
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Mutant SOD1 forms ion channel: implications for ALS pathophysiology.
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Mutant SOD1 forms ion channel: implications for ALS pathophysiology.
@ast
Mutant SOD1 forms ion channel: implications for ALS pathophysiology.
@en
prefLabel
Mutant SOD1 forms ion channel: implications for ALS pathophysiology.
@ast
Mutant SOD1 forms ion channel: implications for ALS pathophysiology.
@en
P2093
P2860
P1476
Mutant SOD1 forms ion channel: implications for ALS pathophysiology.
@en
P2093
Ghanashyam D Ghadge
Jenny L Whitlock
Jérome J Lacroix
Michael J Allen
Morton F Arnsdorf
Ratnesh Lal
Raymond P Roos
P2860
P304
P356
10.1016/J.NBD.2011.08.031
P577
2011-09-10T00:00:00Z