Tumor suppressor gene inactivation during cadmium-induced malignant transformation of human prostate cells correlates with overexpression of de novo DNA methyltransferase. - Wikidata - wikimedia - TriplyDB

Tumor suppressor gene inactivation during cadmium-induced malignant transformation of human prostate cells correlates with overexpression of de novo DNA methyltransferase.

Tumor suppressor gene inactivation during cadmium-induced malignant transformation of human prostate cells correlates with overexpression of de novo DNA methyltransferase.

http://www.wikidata.org/entity/Q36065002

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Association of global DNA methylation and global DNA hydroxymethylation with metals and other exposures in human blood DNA samples What causes human cancer? Approaches from the chemistry of DNA damage Environmental chemicals and DNA methylation in adults: a systematic review of the epidemiologic evidence Influence of toxicologically relevant metals on human epigenetic regulation Developmental neurotoxicants in e-waste: an emerging health concern Environmental epigenetics and its implication on disease risk and health outcomes Epigenetic effects of cadmium in cancer: focus on melanoma Endocrine-disrupting chemicals: associated disorders and mechanisms of action Incorporating epigenetic data into the risk assessment process for the toxic metals arsenic, cadmium, chromium, lead, and mercury: strategies and challenges Cadmium induces histone H3 lysine methylation by inhibiting histone demethylase activity A review of molecular events of cadmium-induced carcinogenesis Basic mechanics of DNA methylation and the unique landscape of the DNA methylome in metal-induced carcinogenesis Cadmium-Induced Pathologies: Where Is the Oxidative Balance Lost (or Not)?A functional polymorphism in the DNA methyltransferase-3A promoter modifies the susceptibility in gastric cancer but not in esophageal carcinoma Low levels of Cd induce persisting epigenetic modifications and acclimation mechanisms in the earthworm Lumbricus terrestris.Rotating night work, lifestyle factors, obesity and promoter methylation in BRCA1 and BRCA2 genes among nurses and midwives.Epigenetics meets endocrinology.Epigenetics in metal carcinogenesis: nickel, arsenic, chromium and cadmium.Methylation-associated down-regulation of RASSF1A and up-regulation of RASSF1C in pancreatic endocrine tumors.Promoter Methylation in Prostate Cancer and its Application for the Early Detection of Prostate Cancer Using Serum and Urine Samples.Metabolic profiling detects early effects of environmental and lifestyle exposure to cadmium in a human population 5-aza-2'-deoxycytidine leads to reduced embryo implantation and reduced expression of DNA methyltransferases and essential endometrial genes Short placental telomere was associated with cadmium pollution in an electronic waste recycling town in China.Role of oxidative stress in cadmium toxicity and carcinogenesis.Heavy metals and epigenetic alterations in brain tumors.Infant sex-specific placental cadmium and DNA methylation associations.Daily variation in global and local DNA methylation in mouse livers.Environmental epigenetics in metal exposure.Cadmium increases HIF-1 and VEGF expression through ROS, ERK, and AKT signaling pathways and induces malignant transformation of human bronchial epithelial cells.Epigenetic Contributions to the Relationship between Cancer and Dietary Intake of Nutrients, Bioactive Food Components, and Environmental Toxicants.Differential DNA methylation profile of key genes in malignant prostate epithelial cells transformed by inorganic arsenic or cadmium Multi-target siRNA based on DNMT3A/B homologous conserved region influences cell cycle and apoptosis of human prostate cancer cell line TSU-PR1.Peromyscus as a Mammalian epigenetic model.Low-level environmental cadmium exposure is associated with DNA hypomethylation in Argentinean women.Basic studies on epigenetic carcinogenesis of low-dose exposure to 1-trichloromethyl-1,2,3,4-tetrahydro-β-carboline (TaClo) in vitro.A Tox21 Approach to Altered Epigenetic Landscapes: Assessing Epigenetic Toxicity Pathways Leading to Altered Gene Expression and Oncogenic Transformation In Vitro Sex-specific effects of early life cadmium exposure on DNA methylation and implications for birth weight.Cadmium and its neurotoxic effects DNA methylation is differentially associated with environmental cadmium exposure based on sex and smoking status.The role of oxidative stress in carcinogenesis induced by metals and xenobiotics.

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P2860

Tumor suppressor gene inactivation during cadmium-induced malignant transformation of human prostate cells correlates with overexpression of de novo DNA methyltransferase.

http://www.wikidata.org/entity/Q36065002

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2007 nî lūn-bûn

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2007年の論文

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2007年論文

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2007年論文

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2007年論文

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Tumor suppressor gene inactiva ...... de novo DNA methyltransferase.

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Tumor suppressor gene inactiva ...... de novo DNA methyltransferase.

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Tumor suppressor gene inactiva ...... de novo DNA methyltransferase.

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Tumor suppressor gene inactiva ...... de novo DNA methyltransferase.

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Environmental Health Perspectives

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Tumor suppressor gene inactiva ...... de novo DNA methyltransferase.

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Anna L Dill

http://www.w3.org/2001/XMLSchema#string

Michael P Waalkes

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Mukta M Webber

http://www.w3.org/2001/XMLSchema#string

Robert A Waterland

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P2860

The human DNA methyltransferases (DNMTs) 1, 3a and 3b: coordinate mRNA expression in normal tissues and overexpression in tumors

DNA methyltransferases Dnmt3a and Dnmt3b are essential for de novo methylation and mammalian development

Overexpression of a splice variant of DNA methyltransferase 3b, DNMT3b4, associated with DNA hypomethylation on pericentromeric satellite regions during human hepatocarcinogenesis.

Methylation-specific PCR: a novel PCR assay for methylation status of CpG islands

DNMT1 and DNMT3b cooperate to silence genes in human cancer cells

Differential requirement for DNA methyltransferase 1 in maintaining human cancer cell gene promoter hypermethylation

Cloning and characterization of a family of novel mammalian DNA (cytosine-5) methyltransferases

CpG methylation is maintained in human cancer cells lacking DNMT1.

Epigenetic mechanisms of nickel carcinogenesis.

An essential role for DNA methyltransferase DNMT3B in cancer cell survival.

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10

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115

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