Endometrial miR-181a and miR-98 expression is altered during transition from normal into cancerous state and target PGR, PGRMC1, CYP19A1, DDX3X, and TIMP3.
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MicroRNA regulation of progesterone receptor in breast cancerCelecoxib increases miR-222 while deterring aromatase-expressing breast tumor growth in micemiR-98 suppresses melanoma metastasis through a negative feedback loop with its target gene IL-6.Progesterone receptor membrane component 1 deficiency attenuates growth while promoting chemosensitivity of human endometrial xenograft tumorsRole of aromatase in sex-specific cerebrovascular endothelial function in miceHsa-microRNA-181a is a regulator of a number of cancer genes and a biomarker for endometrial carcinoma in patients: a bioinformatic and clinical study and the therapeutic implication.Timp3 deficient mice show resistance to developing breast cancer.MicroRNA-181a is involved in the regulation of human endometrial stromal cell decidualization by inhibiting Krüppel-like factor 12MiR-191 modulates malignant transformation of endometriosis through regulating TIMP3.Plasminogen activator inhibitor 1 RNA-binding protein interacts with progesterone receptor membrane component 1 to regulate progesterone's ability to maintain the viability of spontaneously immortalized granulosa cells and rat granulosa cellsMicroRNA-132 promotes estradiol synthesis in ovarian granulosa cells via translational repression of Nurr1.Genome-wide analysis of brain and gonad transcripts reveals changes of key sex reversal-related genes expression and signaling pathways in three stages of Monopterus albusDifferential network analysis reveals the genome-wide landscape of estrogen receptor modulation in hormonal cancersmiR-720 is a downstream target of an ADAM8-induced ERK signaling cascade that promotes the migratory and invasive phenotype of triple-negative breast cancer cells.MicroRNA miR-98 inhibits tumor angiogenesis and invasion by targeting activin receptor-like kinase-4 and matrix metalloproteinase-11Role of microRNAs in the regulation of drug metabolism and disposition genes in diabetes and liver disease.Role of microRNAs in cancers of the female reproductive tract: insights from recent clinical and experimental discovery studies.The role of microRNAs in the pathogenesis of endometrial cancer: a systematic review.TIMPs: versatile extracellular regulators in cancer.Hyaluronic acid promotes the expression of progesterone receptor membrane component 1 via epigenetic silencing of miR-139-5p in human and rat granulosa cells.Dynamic and Modularized MicroRNA Regulation and Its Implication in Human Cancers.MiR-98 is involved in rat embryo implantation by targeting Bcl-xl.Altered microRNA expression patterns during the initiation and promotion stages of neonatal diethylstilbestrol-induced dysplasia/neoplasia in the hamster (Mesocricetus auratus) uterus.miR-98-5p contributes to cisplatin resistance in epithelial ovarian cancer by suppressing miR-152 biogenesis via targeting Dicer1.Progesterone receptor membrane component 1 as a potential prognostic biomarker for hepatocellular carcinoma.
P2860
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P2860
Endometrial miR-181a and miR-98 expression is altered during transition from normal into cancerous state and target PGR, PGRMC1, CYP19A1, DDX3X, and TIMP3.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Endometrial miR-181a and miR-9 ...... C1, CYP19A1, DDX3X, and TIMP3.
@ast
Endometrial miR-181a and miR-9 ...... C1, CYP19A1, DDX3X, and TIMP3.
@en
type
label
Endometrial miR-181a and miR-9 ...... C1, CYP19A1, DDX3X, and TIMP3.
@ast
Endometrial miR-181a and miR-9 ...... C1, CYP19A1, DDX3X, and TIMP3.
@en
prefLabel
Endometrial miR-181a and miR-9 ...... C1, CYP19A1, DDX3X, and TIMP3.
@ast
Endometrial miR-181a and miR-9 ...... C1, CYP19A1, DDX3X, and TIMP3.
@en
P2093
P2860
P356
P1476
Endometrial miR-181a and miR-9 ...... C1, CYP19A1, DDX3X, and TIMP3.
@en
P2093
Harekrushna Panda
Nasser Chegini
Tsai-Der Chuang
Xiaoping Luo
P2860
P304
P356
10.1210/JC.2012-1018
P407
P577
2012-04-06T00:00:00Z